Professor of Pathology, Brigham and Women's Hospital

Pathology

Dr. Andrew H. Lichtman is an expert in the field of Pathology. He studied medicine at University of Rochester School of Medicine and Dentistry. Andrew Lichtman is also published. He has 41 publications published. The lastest publication: "Blockade of Tim-1 and Tim-4 Enhances Atherosclerosis in Low-Density Lipoprotein Receptor-Deficient Mice.'

Publications

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• Blockade of Tim-1 and Tim-4 Enhances Atherosclerosis in Low-Density Lipoprotein Receptor-Deficient Mice.
• Atheroprotective immunity and cardiovascular disease: therapeutic opportunities and challenges.
• IRF5 deficiency ameliorates lupus but promotes atherosclerosis and metabolic dysfunction in a mouse model of lupus-associated atherosclerosis.
• Treating Atherosclerosis With Regulatory T Cells.
• NF-κB Directs Dynamic Super Enhancer Formation in Inflammation and Atherogenesis.
• Lysine-specific demethylase-1 modifies the age effect on blood pressure sensitivity to dietary salt intake.
• The Influence of Innate and Adaptive Immune Responses on Atherosclerosis.
• Immune effector mechanisms implicated in atherosclerosis: from mice to humans.
• The heart of the matter: Protection of the myocardium from T cells.
• Adaptive immunity in atherogenesis: new insights and therapeutic approaches.
• Adaptive Immunity and Atherosclerosis: Mouse Tales in the AJP.
• Difference in Th1 and th17 lymphocyte adhesion to endothelium.
• Loss of myeloid related protein-8/14 exacerbates cardiac allograft rejection.
• Foxp3+-Inducible Regulatory T Cells Suppress Endothelial Activation and Leukocyte Recruitment.
• Dynamic changes in regulatory T cells are linked to levels of diet-induced hypercholesterolemia.
• Impairment of the programmed cell death-1 pathway increases atherosclerotic lesion development and inflammation.
• T cells and blood vessels: costimulation turns up the pressure.
• Statin-induced Krüppel-like factor 2 expression in human and mouse T cells reduces inflammatory and pathogenic responses.
• T-cell costimulation and coinhibition in atherosclerosis.
• CD11c(+) dendritic cells maintain antigen processing, presentation capabilities, and CD4(+) T-cell priming efficacy under hypercholesterolemic conditions associated wi...
• The influence of the regulatory T lymphocytes on atherosclerosis.
• Endothelial programmed death-1 ligand 1 (PD-L1) regulates CD8+ T-cell mediated injury in the heart.
• Proatherogenic immune responses are regulated by the PD-1/PD-L pathway in mice.
• Targeting interferon-gamma to treat atherosclerosis.
• Impaired regulatory T-cell response and enhanced atherosclerosis in the absence of inducible costimulatory molecule.
• T-bet controls pathogenicity of CTLs in the heart by separable effects on migration and effector activity.
• Clonal deletion of thymocytes by circulating dendritic cells homing to the thymus.
• Abrogation of functional selectin-ligand expression reduces migration of pathogenic CD8+ T cells into heart.
• Protecting the pump: controlling myocardial inflammatory responses.
• T-bet is required for optimal proinflammatory CD4+ T-cell trafficking.
• BLT1-mediated T cell trafficking is critical for rejection and obliterative bronchiolitis after lung transplantation.
• T-bet deficiency reduces atherosclerosis and alters plaque antigen-specific immune responses.
• The link between IFN-gamma and allograft arteriopathy: is the answer NO?
• Co-stimulation and plaque-antigen-specific T-cell responses in atherosclerosis.
• B7-1/B7-2 costimulation regulates plaque antigen-specific T-cell responses and atherogenesis in low-density lipoprotein receptor-deficient mice.
• Neutrophils sustain pathogenic CD8+ T cell responses in the heart.
• In vivo high resolution three-dimensional imaging of antigen-specific cytotoxic T-lymphocyte trafficking to tumors.
• Influence of interferon-gamma on the extent and phenotype of diet-induced atherosclerosis in the LDLR-deficient mouse.
• IL-12 is required for differentiation of pathogenic CD8+ T cell effectors that cause myocarditis.
• Influence of C3 deficiency on atherosclerosis.

Schools

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• University Of Rochester

• University Rochester

• Brigham Womens Hospital

Doctors Specialties

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• Anatomic Pathology
• Pathology

Accepted Insurances

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Education

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• Brigham and Women's Hospital
• University of Rochester School of Medicine and Dentistry

Hospital

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• Brigham and Women's Hospital

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