Physician

General Surgery

Dr. Benjamin J. Wilkins, MD

Doctor Benjamin J. Wilkins, MD practices General Surgery. He researched medicine at University of Cincinnati College of Medicine. Benjamin Wilkins is a published doctor and has 17 publications published. The lastest was: 'Non-functional parathyroid carcinoma: a review of the literature and report of a case requiring extensive surgery.'

Publications

Non-functional parathyroid carcinoma: a review of the literature and report of a case requiring extensive surgery.
Genetic inhibition of cardiac ERK1/2 promotes stress-induced apoptosis and heart failure but has no effect on hypertrophy in vivo.
Direct interaction and reciprocal regulation between ASK1 and calcineurin-NFAT control cardiomyocyte death and growth.
Direct and indirect interactions between calcineurin-NFAT and MEK1-extracellular signal-regulated kinase 1/2 signaling pathways regulate cardiac gene expression and ce...
Calcium-calcineurin signaling in the regulation of cardiac hypertrophy.
Genetic loss of calcineurin blocks mechanical overload-induced skeletal muscle fiber type switching but not hypertrophy.
MEK1-ERK2 signaling pathway protects myocardium from ischemic injury in vivo.
Calcineurin Abeta gene targeting predisposes the myocardium to acute ischemia-induced apoptosis and dysfunction.
Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy.
Altered skeletal muscle phenotypes in calcineurin Aalpha and Abeta gene-targeted mice.
Targeted inhibition of p38 MAPK promotes hypertrophic cardiomyopathy through upregulation of calcineurin-NFAT signaling.
Targeted disruption of NFATc3, but not NFATc4, reveals an intrinsic defect in calcineurin-mediated cardiac hypertrophic growth.
Impaired cardiac hypertrophic response in Calcineurin Abeta -deficient mice.
c-Jun N-terminal kinases (JNK) antagonize cardiac growth through cross-talk with calcineurin-NFAT signaling.
Calcineurin and cardiac hypertrophy: where have we been? Where are we going?
The DNA damage checkpoint and PKA pathways converge on APC substrates and Cdc20 to regulate mitotic progression.

Schools

University of Cincinnati Main Campus

Doctors Specialties

General Surgery

Accepted Insurances

Education

Washington University
University of Cincinnati College of Medicine

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