Assistant Professor, Medicine, Tufts Medical Center

Cardiology

Dr. Chester H. Conrad, MD

Chester H. Conrad works in the field of Cardiology. He went to Harvard Medical School. Harvard Medical School is one of the top medical schools in the US. Harvard Medical School is ranked 1/12 in Research/PrimaryCare. Chester Conrad is also published. He has 48 publications published. The lastest publication: "Role of microtubules in the contractile dysfunction of hypertrophied myocardium.'

Publications

Implementation of the Department of Veterans Affairs' first point-of-care clinical trial.
Transition from compensated hypertrophy to systolic Heart failure in the spontaneously hypertensive rat: Structure, function, and transcript analysis.
Isoproterenol-induced Myocardial injury and diastolic dysfunction in mice: structural and functional correlates.
Thyroid state and tolerance of mammalian myocardium to hypoxia.
L-arginine fails to prevent ventricular remodeling and Heart failure in the spontaneously hypertensive rat.
Direct Myocardial effects of levosimendan in humans with left ventricular dysfunction: alteration of force-frequency and relaxation-frequency relationships.
Heart failure after long-term supravalvular Aortic constriction in rats.
Thoracic massage permits use of echocardiography in unanesthetized rats.
Potentiation of Atrial contractility by paired pacing augments ventricular preload and systolic performance.
Comparison of contractile function of diaphragm and cardiac muscle in response to paired electrical stimulation.
Constitutive properties of hypertrophied myocardium: cellular contribution to changes in Myocardial stiffness.
Studies of prevention, treatment and mechanisms of Heart failure in the aging spontaneously hypertensive rat.
Rationale and design of the third vasodilator-Heart failure trial (V-HeFT III): felodipine as adjunctive therapy to enalapril and loop diuretics with or without digoxi...
Incomplete, delayed functional recovery late after reperfusion following Acute myocardial infarction: "maimed myocardium".
Outward current and repolarization in hypoxic rat myocardium.
Reperfusion induced Arrhythmias following ischaemia in intact rat heart: role of intracellular calcium.
Phase-plane analysis of left ventricular chamber filling and midwall fiber lengthening in patients with left ventricular hypertrophy.
Alterations in cardiac gene expression during the transition from stable hypertrophy to Heart failure. Marked upregulation of genes encoding extracellular matrix compo...
Protective effects of diltiazem on the mechanical performance of the hypoxic myocardium.
Force pattern of hypoxic myocardium applied to oxygenated muscle preparations: comparison with effects of regional ischemia on the contraction of non-ischemic myocardium.
Fluorocarbon simulation of Myocardial ischaemia and reperfusion: studies of relationships between force and intracellular calcium.
Inhibition of hypoxic Myocardial contracture by Cobalt in the rat.
Tolerance to hypoxia of myocardium from adult and aged spontaneously Hypertensive rats.
[Role of myocardial contractile status and relaxation in ventricular dysfunction during the transition of Heart hypertrophy to failure].
Myocardial injury in the mouse induced by transthoracic cauterization.
Effect of chronic colchicine administration on the myocardium of the aging spontaneously Hypertensive rat.
Thyroid hormone effects on intracellular calcium and inotropic responses of rat ventricular myocardium.
Myocardial osteopontin expression coincides with the development of Heart failure.
Myocardial fibrosis in transforming growth factor beta(1)heterozygous mice.
Direct effects of colchicine on Myocardial function: studies in hypertrophied and failing spontaneously hypertensive rats.
Effects of treppe and calcium on intracellular calcium and function in the failing Heart from the spontaneously hypertensive rat.
Preload does not affect relaxation rate in normal, hypoxic, or hypertrophic myocardium.
Myocardial function structure and collagen in the spontaneously hypertensive rat: progression from compensated hypertrophy to haemodynamic impairment.
Role of microtubules in the contractile dysfunction of hypertrophied myocardium.
Captopril modifies gene expression in hypertrophied and failing Hearts of aged spontaneously hypertensive rats.
Altered inotropic responsiveness and gene expression of hypertrophied myocardium with captopril.
Oxygen cost of stress development in hypertrophied and failing Hearts from the spontaneously hypertensive rat.
Differences between mouse and rat Myocardial contractile responsiveness to calcium.
Modulation of left and right ventricular beta-adrenergic receptors from spontaneously hypertensive rats with left ventricular hypertrophy and Failure.
Intracellular calcium transients in myocardium from spontaneously hypertensive rats during the transition to Heart failure.
Impaired myocardial function in spontaneously hypertensive rats with Heart failure.
Effect of angiotensin-converting enzyme inhibition on Myocardial fibrosis and function in hypertrophied and failing myocardium from the spontaneously hypertensive rat.
Localization of alpha1(I) collagen mRNA in myocardium from the spontaneously hypertensive rat during the transition from compensated hypertrophy to Failure.
Myocardial fibrosis and stiffness with hypertrophy and Heart failure in the spontaneously hypertensive rat.
The spontaneously hypertensive rat as a model of the transition from compensated left ventricular hypertrophy to Failure.
A point-of-care clinical trial comparing insulin administered using a sliding scale versus a weight-based regimen.
Transcriptional changes associated with recovery from Heart failure in the SHR.

Schools

Harvard Medical School
PresbyU Hospital
Va Med Center

Conditions Treated

Angina and Acute Coronary Syndrome
Aortic Valve Disease
Arrhythmias (incl. Atrial Fibrillation)
Cardiomyopathy
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Doctors Specialties

Cardiology

Accepted Insurances

Education

Tufts Medical Center
Beth Israel Deaconess Medical Center
University of Pittsburgh Medical Center
Harvard Medical School

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