EVP Medical Affairs, Humetrix
Nephrology
Dr. Christopher R. Burrow, MD
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Christopher Burrow works in the field of Nephrology. He graduated from Baylor College of Medicine. Christopher R. Burrow, MD is a published doctor. He published 36 publications, including: 'WT1 expression induces features of renal epithelial differentiation in mesenchymal fibroblasts.'
Publications
- Lack of association between adrenergic receptor genotypes and survival in heart failure patients treated with carvedilol or metoprolol.
- Protein kinase X activates ureteric bud branching morphogenesis in developing mouse metanephric kidney.
- Disruption of polycystin-1 function interferes with branching morphogenesis of the ureteric bud in developing mouse kidneys.
- Spectrum and prevalence of cardiac sodium channel variants among black, white, Asian, and Hispanic individuals: implications for arrhythmogenic susceptibility and Brug...
- Midkine promotes selective expansion of the nephrogenic mesenchyme during kidney organogenesis.
- The exocyst localizes to the primary cilium in MDCK cells.
- Ethnic differences in cardiac potassium channel variants: implications for genetic susceptibility to sudden cardiac death and genetic testing for congenital long QT sy...
- Na transport in autosomal recessive polycystic kidney disease (ARPKD) cyst lining epithelial cells.
- PRKX, a phylogenetically and functionally distinct cAMP-dependent protein kinase, activates renal epithelial cell migration and morphogenesis.
- Polarity of alpha-galactosidase A uptake by renal tubule cells.
- Identification of two distinct regions within the adenovirus minimal origin of replication that are required for adenovirus type 4 DNA replication in vitro.
- Expression of rat fibroblast growth factor receptor 1 as three splicing variants during kidney development.
- Regulatory molecules in kidney development.
- Retinoids and renal development.
- Renal progenitor cells: problems of definition, isolation and characterization.
- Developmental regulation of CFTR expression during human nephrogenesis.
- STK-1, the human homolog of Flk-2/Flt-3, is selectively expressed in CD34+ human bone marrow cells and is involved in the proliferation of early progenitor/stem cells.
- A putative Wilms tumor-secreted growth factor activity required for primary culture of human nephroblasts.
- Renal proximal tubular epithelium from patients with nephropathic cystinosis: immortalized cell lines as in vitro model systems.
- Autosomal dominant polycystic kidney disease: cellular and molecular mechanisms of cyst formation.
- Monomeric midkine induces tumor cell proliferation in the absence of cell-surface proteoglycan binding.
- Towards understanding the polycystins.
- Expression of aquaporins-1 and -2 during nephrogenesis and in autosomal dominant polycystic kidney disease.
- Abnormalities in extracellular matrix regulation in autosomal dominant polycystic kidney disease.
- Cystic diseases of the kidney: role of adhesion molecules in normal and abnormal tubulogenesis.
- WT1 expression induces features of renal epithelial differentiation in mesenchymal fibroblasts.
- Mouse embryonic stem cell-derived embryoid bodies generate progenitors that integrate long term into renal proximal tubules in vivo.
- Herpes simplex virus as a model vector system for gene therapy in renal disease.
- Apical plasma membrane mispolarization of NaK-ATPase in polycystic kidney disease epithelia is associated with aberrant expression of the beta2 isoform.
- The PKD1 gene product, "polycystin-1," is a tyrosine-phosphorylated protein that colocalizes with alpha2beta1-integrin in focal clusters in adherent renal epithelia.
- Expression of the beta2-subunit and apical localization of Na+-K+-ATPase in metanephric kidney.
- Modification of the composition of polycystin-1 multiprotein complexes by calcium and tyrosine phosphorylation.
- Identification of phosphorylation sites in the PKD1-encoded protein C-terminal domain.
- Inhibition of HER-2(neu/ErbB2) restores normal function and structure to polycystic kidney disease (PKD) epithelia.
- Protein kinase X (PRKX) can rescue the effects of polycystic kidney disease-1 gene (PKD1) deficiency.
Schools
Baylor College Of Medicine
Baylor Affil Hosps
Johns Hopkins Hospital
Doctors Specialties
- Nephrology
Accepted Insurances
Education
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Johns Hopkins University -
Baylor College of Medicine
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