Associate Professor, Neurology/Ophthalmology, MI State Univ, Coll of Human Med
Neurology
Dr. Cynthia H. Hingtgen, MD
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Doctor Cynthia H. Hingtgen, MD practices Neurology. She attended Indiana University School of Medicine. Indiana University School of Medicine is ranked 45 in research and 47 in primary care medicine She received 1 awards: "CMS Meaningful Use Stage 1 Certification". Doctor Cynthia H. Hingtgen, MD is a published physician. She published 31 publications, including: 'The treatment of Parkinson's disease: current concepts and rationale.' She accepts Medicare.
Publications
- N-type calcium current, Cav2.2, is enhanced in small-diameter sensory neurons isolated from Nf1+/- mice.
- Neurofibromin Deficient Myeloid Cells are Critical Mediators of Aneurysm Formation In Vivo.
- Schwannomatosis: the overlooked neurofibromatosis?
- Development of the Adult PedsQL™ Neurofibromatosis Type 1 Module: Initial Feasibility, Reliability and Validity.
- Suppression of Inflammatory and neuropathic pain by uncoupling CRMP-2 from the presynaptic Ca²âº channel complex.
- Reduced expression of SynGAP, a neuronal GTPase-activating protein, enhances capsaicin-induced Peripheral sensitization.
- Ras signaling pathways mediate NGF-induced enhancement of excitability of small-diameter capsaicin-sensitive sensory neurons from wildtype but not Nf1+/- mice.
- Ret-dependent and Ret-independent mechanisms of Gfl-induced sensitization.
- Signaling pathways that mediate Nerve growth factor-induced increase in expression and release of calcitonin gene-related peptide from sensory neurons.
- Augmented sodium currents contribute to the enhanced excitability of small diameter capsaicin-sensitive sensory neurons isolated from Nf1+/? mice.
- Genetic and Cellular evidence of vascular inflammation in neurofibromin-deficient mice and humans.
- Regulation of N-type voltage-gated calcium channels (Cav2.2) and transmitter release by collapsin response mediator protein-2 (CRMP-2) in sensory neurons.
- Glial Cell line-derived neurotrophic factor family ligands enhance capsaicin-stimulated release of calcitonin gene-related peptide from sensory neurons.
- Neurofibromatosis: The role of guanosine triphosphatase activating proteins in sensory neuron function.
- Involvement of platelet-activating factor in ultraviolet B-induced hyperalgesia.
- Injection of autologous muscle stem Cells (myoblasts) for the treatment of vocal fold paralysis: a pilot study.
- Neurofibromin-deficient Schwann Cells have increased lysophosphatidic acid dependent survival and migration-implications for increased neurofibroma formation during pr...
- A new "kine" of pain: MCP-1 and sensory neuron excitability. Focus on "MCP-1 enhances excitability of nociceptive neurons in Chronically compressed dorsal root ganglia".
- Stimulus-evoked release of neuropeptides is enhanced in sensory neurons from mice with a heterozygous mutation of the Nf1 gene.
- Sensory neurons from Nf1 haploinsufficient mice exhibit increased excitability.
- Isolation and culture of sensory neurons from the dorsal-root ganglia of embryonic or adult rats.
- Neurofibromin-deficient Schwann Cells secrete a potent migratory stimulus for Nf1+/- mast Cells.
- Uptake of HIV-1 tat protein mediated by low-density lipoprotein receptor-related protein disrupts the neuronal metabolic balance of the receptor ligands.
- The tragedy of Sergei Prokofiev.
- The painful perils of a pair of pianists: the Chronic pain of Clara Schumann and Sergei Rachmaninov.
- The treatment of Parkinson's Disease: current concepts and rationale.
- Carba prostacyclin enhances the capsaicin-induced cobalt-loading of rat sensory neurons grown in culture.
- Prostaglandins facilitate peptide release from rat sensory neurons by activating the adenosine 3',5'-cyclic monophosphate transduction cascade.
- The phosphatase inhibitor, okadaic acid, increases peptide release from rat sensory neurons in culture.
- Prostacyclin enhances the evoked-release of substance P and calcitonin gene-related peptide from rat sensory neurons.
Schools
Indiana University School Of Medicine
St Vincents Hospital
In University School Med
Procedures Preformed
- EEG (Electroencephalogram)
- EMG (Electromyography)
Conditions Treated
- Alzheimer's Disease
- Ataxia
- Carpal Tunnel Syndrome
- Cerebral Palsy
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Doctors Specialties
- Neurology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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Indiana University School of Medicine
Hospital
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Spectrum Health - Butterworth Hospital
Drug Facts
| NPI NUMBER | 1598789034 | |
| NPPES Provider LastName | HINGTGEN | |
| NPPES Provider FirstName | CYNTHIA | |
| NPPES Provider ZIPCode | 495259480 | |
| NPPES Provider State | MI | |
| Specialty Description | Neurology | |
| Total Claim Count | 1258.0 | |
| Distinct Opioid Count | 1.0 | |
| Opioid Claim Count | 12.0 | |
| Percent Opioid Claims | 0.95 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1598789034 |
| Last Name Of The Provider | HINGTGEN |
| First Name Of The Provider | CYNTHIA |
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