Physician
Obstetrics & Gynecology
Dr. Dale C. Leitman, MD
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Doctor Dale C. Leitman, MD works in the field of Obstetrics & Gynecology. He went to medical school at University of California Davis School of Medicine. University of California Davis School of Medicine is ranked 43/19 in Research/PrimaryCare. Dale Leitman is also a published doctor. He has 38 paper published. The most recent paper is: Thyroid hormone activation of transcription is potentiated by activators of cAMP-dependent protein kinase.
Publications
- MF101 : a multi-component botanical selective estrogen receptor beta modulator for the treatment of menopausal vasomotor symptoms.
- Estrogenic plant foods of red colobus monkeys and mountain gorillas in uganda.
- Cross talk between glucocorticoid and estrogen receptors occurs at a subset of proinflammatory genes.
- Estrogenic Plant Extracts Reverse Weight Gain and Fat Accumulation without Causing Mammary Gland or Uterine Proliferation.
- Estrogen receptor β causes a G2 cell cycle arrest by inhibiting CDK1 activity through the regulation of cyclin B1, GADD45A, and BTG2.
- Estrogen receptor beta binds to and regulates three distinct classes of target genes.
- Selective activation of estrogen receptor-beta target genes by 3,3'-diindolylmethane.
- Estrogen receptor beta-selective agonists stimulate calcium oscillations in human and mouse embryonic stem cell-derived neurons.
- Drug and cell type-specific regulation of genes with different classes of estrogen receptor beta-selective agonists.
- Differential regulation of native estrogen receptor-regulatory elements by estradiol, tamoxifen, and raloxifene.
- Selective estrogen receptor-beta agonists repress transcription of proinflammatory genes.
- Multiple transcription factor elements collaborate with estrogen receptor alpha to activate an inducible estrogen response element in the NKG2E gene.
- Selective activation of estrogen receptor-beta transcriptional pathways by an herbal extract.
- Tumor necrosis factor-alpha down-regulates human Cu/Zn superoxide dismutase 1 promoter via JNK/AP-1 signaling pathway.
- Distinct roles of unliganded and liganded estrogen receptors in transcriptional repression.
- Differential response of estrogen receptor subtypes to 1,3-diarylindene and 2,3-diarylindene ligands.
- Estradiol and selective estrogen receptor modulators differentially regulate target genes with estrogen receptors alpha and beta.
- Estrogen receptor beta inhibits human breast cancer cell proliferation and tumor formation by causing a G2 cell cycle arrest.
- Estrogens activate bone morphogenetic protein-2 gene transcription in mouse mesenchymal stem cells.
- Alternate surfaces of transcriptional coregulator GRIP1 function in different glucocorticoid receptor activation and repression contexts.
- Estradiol represses human T-cell leukemia virus type 1 Tax activation of tumor necrosis factor-alpha gene transcription.
- Regulation of specific target genes and biological responses by estrogen receptor subtype agonists.
- Unliganded estrogen receptor-beta regulation of genes is inhibited by tamoxifen.
- Selective concomitant inhibition of mTORC1 and mTORC2 activity in estrogen receptor negative breast cancer cells by BN107 and oleanolic acid.
- Liquiritigenin is a plant-derived highly selective estrogen receptor beta agonist.
- Cell type- and estrogen receptor-subtype specific regulation of selective estrogen receptor modulator regulatory elements.
- The Endocrine Society Ethics Advisory Committee: ethical aspects of conflicts of interests, October 2003.
- Estradiol repression of tumor necrosis factor-alpha transcription requires estrogen receptor activation function-2 and is enhanced by coactivators.
- Identification of a tumor necrosis factor-responsive element in the tumor necrosis factor alpha gene.
- Estrogen receptor beta-selective transcriptional activity and recruitment of coregulators by phytoestrogens.
- Regulation of vascular endothelial growth factor (VEGF) gene transcription by estrogen receptors alpha and beta.
- The core promoter region of the tumor necrosis factor alpha gene confers phorbol ester responsiveness to upstream transcriptional activators.
- Thyroid hormone activation of transcription is potentiated by activators of cAMP-dependent protein kinase.
- Selective estrogen receptor modulators in reproductive medicine and biology.
- Regulation of particulate guanylate cyclase by natriuretic peptides and Escherichia coli heat-stable enterotoxin.
- Comparison of particulate guanylate cyclase in cells with and without atrial natriuretic peptide receptor binding activity.
- Regulation of particulate guanylate cyclase by atriopeptins: relation between peptide structure, receptor binding, and enzyme kinetics.
Schools
Univ Of Ca
Doctors Specialties
- Obstetrics
- Gynecology
Accepted Insurances
Education
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UCSF -
University of California Davis School of Medicine
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