Associate Professor, Internal Medicine, Washington University School of Medicine
Pathology
Dr. Deborah V. Novack, MD
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Dr. Deborah V. Novack is an expert in the field of Pathology. She went to medical school at Washington University School of Medicine in St. Louis. She received 1 awards: "Elected Member". Deborah Novack is a published doctor and has 59 publications published. The lastest was: 'Bcl-2 protein expression during murine development.' Dr. Deborah V. Novack is registered with Medicare and accepts Medicare payments.
Publications
- Congenital insensitivity to pain: Fracturing without apparent skeletal pathobiology caused by an autosomal dominant, second mutation in SCN11A encoding voltage-gated s...
- Inhibition of CaMKK2 reverses age-associated decline in bone mass.
- Reply to "Rapid Skeletal Turnover in Radiographic Mimic of Osteopetrosis Might Be Secondary to Systemic Mastocytosis"
- Juvenile Paget's disease with heterozygous duplication within TNFRSF11A encoding RANK.
- Thrombospondin-1 Regulates Bone Homeostasis Through Effects on Bone Matrix Integrity and Nitric Oxide Signaling In Osteoclasts.
- Rapid Skeletal Turnover In A Radiographic Mimic Of Osteopetrosis.
- p38MAPK plays a crucial role in stromal mediated tumorigenesis.
- Down-regulation of PLCγ2-β-catenin pathway promotes activation and expansion of myeloid-derived suppressor cells in cancer.
- Osteoclast-induced Foxp3+ CD8 T-cells limit bone loss in mice.
- Anti-cancer IAP antagonists promote bone metastasis: a cautionary tale.
- Acute Severe Hypercalcemia After Traumatic Fractures and Immobilization in Hypophosphatasia Complicated by Chronic Renal Failure.
- Hypermineralized Whale Rostrum as the Exemplar for Bone Mineral.
- Antagonism of Inhibitor of Apoptosis Proteins Increases Bone Metastasis via Unexpected Osteoclast Activation.
- Creation and preliminary characterization of a leptin knockout rat.
- The cyclin dependent kinase inhibitor p21 is essential for resolution of murine inflammatory arthritis via its c-terminal domain.
- CD8+ T Cells Regulate Bone Tumor Burden Independent of Osteoclast Resorption.
- Camurati-Engelmann disease: unique variant featuring a novel mutation in TGFβ1 encoding transforming growth factor beta 1 and a missense change in TNFSF11 encoding RAN...
- Unique personalities within the NF-κB family: distinct functions for p65 and RelB in the osteoclast.
- Camurati-engelmann disease: Unique variant featuring a novel mutation in TGFβ1 encoding transforming growth factor beta 1 and a missense change in TNFSF11 ...
- Dysosteosclerosis presents as an "osteoclast-poor" form of osteopetrosis: comprehensive investigation of a 3-year-old girl and literature review.
- The FOX(O1) blasts off.
- NIK stabilization in osteoclasts results in osteoporosis and enhanced inflammatory osteolysis.
- Bisphosphonate-induced osteopetrosis: novel bone modeling defects, metaphyseal osteopenia, and osteosclerosis fractures after drug exposure ceases.
- Vav/Phospholipase Cgamma2-mediated control of a neutrophil-dependent murine model of rheumatoid arthritis.
- RelA/p65 promotes osteoclast differentiation by blocking a RANKL-induced apoptotic JNK pathway in mice.
- Age-related changes in bone morphology are accelerated in group VIA phospholipase A2 (iPLA2beta)-null mice.
- RelB is the NF-kappaB subunit downstream of NIK responsible for osteoclast differentiation.
- The osteoclast: friend or foe?
- Parathyroid hormone stimulates osteoblastic expression of MCP-1 to recruit and increase the fusion of pre/osteoclasts.
- Estrogen and bone: osteoclasts take center stage.
- Damaging fatigue loading stimulates increases in periosteal vascularity at sites of bone formation in the rat ulna.
- Jawing about TNF: new hope for cherubism.
- PLCgamma2 regulates osteoclastogenesis via its interaction with ITAM proteins and GAB2.
- Critical role of beta3 integrin in experimental postmenopausal osteoporosis.
- M-CSF mediates TNF-induced inflammatory osteolysis.
- FHL2 inhibits the activated osteoclast in a TRAF6-dependent manner.
- NF-(kappa)B-inducing kinase controls lymphocyte and osteoclast activities in inflammatory arthritis.
- Marrow stromal cells and osteoclast precursors differentially contribute to TNF-alpha-induced osteoclastogenesis in vivo.
- Beta3-integrin mediates smooth muscle cell accumulation in neointima after carotid ligation in mice.
- TSH, the bone suppressing hormone.
- The IkappaB function of NF-kappaB2 p100 controls stimulated osteoclastogenesis.
- Dynamic changes in the osteoclast cytoskeleton in response to growth factors and cell attachment are controlled by beta3 integrin.
- Beta3 integrin deficiency promotes atherosclerosis and pulmonary inflammation in high-fat-fed, hyperlipidemic mice.
- Immunohistochemistry of skeletal tissues.
- Germinal center B-cells resist transformation by kras independently of tumor suppressor arf.
- MedEthEx Online: a computer-based learning program in medical ethics and communication skills.
- Suppressed bone turnover during alendronate therapy for high-turnover osteoporosis.
- Bisphosphonate-associated femoral fracture: implications for management in patients with malignancies.
- Are we overtreating papillomas diagnosed on core needle biopsy?
- Bone turnover in bone biopsies of patients with low-energy cortical fractures receiving bisphosphonates: a case series.
- Estrogen deficiency induces bone loss by enhancing T-cell production of TNF-alpha.
- Manifestations in a family with autosomal dominant bone fragility and limb-girdle myopathy.
- Mice lacking beta3 integrins are osteosclerotic because of dysfunctional osteoclasts.
- A Glanzmann's mutation in beta 3 integrin specifically impairs osteoclast function.
- Bcl-2 protein expression during murine development.
- Mapping autosomal dominant progressive limb-girdle myopathy with bone fragility to chromosome 9p21-p22: a novel locus for a musculoskeletal syndrome.
- Osteoclast motility: putting the brakes on bone resorption.
- Role of NF-κB in the skeleton.
Schools
Washington Center School of Medicine
Barnes Jewish Hospital
Doctors Specialties
- Anatomic Pathology
- Pathology
Accepted Insurances
Awards
- Elected Member
Education
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Washington University -
Washington University School of Medicine in St. Louis
Hospital
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Barnes-Jewish Hospital -
St. Louis Children's Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1790701001 |
| Last Name Of The Provider | NOVACK |
| First Name Of The Provider | DEBORAH |
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