Professor and Chair
Neurology
Dr. Dennis W. Choi, MD
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Dennis W. Choi is an expert in the field of Neurology. He attended Harvard Medical School. Harvard Medical School is a top medical university. Harvard Medical School is ranked 1 in research and 12 in primary care medicine He has 2 awards "Elected Member" and "Whos Who in Medicine and Healthcare". Doctor Dennis W. Choi, MD is a published doctor and has 20 publications published. The lastest was: 'Medicines for the Mind: Policy-Based "Pull" Incentives for Creating Breakthrough CNS Drugs.'
Publications
- Medicines for the Mind: Policy-Based "Pull" Incentives for Creating Breakthrough CNS Drugs.
- Transplantation of embryonic stem cells overexpressing Bcl-2 promotes functional recovery after transient cerebral ischemia.
- Neurotrophin and GDNF family ligands promote survival and alter excitoToxic vulnerability of neurons derived from murine embryonic stem cells.
- Cu2+ Toxicity inhibition of mitochondrial dehydrogenases in vitro and in vivo.
- Potassium attenuates zinc-induced death of cultured cortical astrocytes.
- Raising intracellular calcium attenuates neuronal apoptosis triggered by staurosporine or oxygen-glucose deprivation in the presence of glutamate receptor blockade.
- Glutamate, cell death, and hats off to Carl Cotman.
- TrkB mediates BDNF-induced potentiation of neuronal necrosis in cortical culture.
- Potassium channel blockers attenuate hypoxia- and ischemia-induced neuronal death in vitro and in vivo.
- Exploratory clinical testing of neuroscience drugs.
- Cofactors of mitochondrial enzymes attenuate copper-induced death in vitro and in vivo.
- Metabotropic glutamate receptor 1-induced upregulation of NMDA receptor current: mediation through the Pyk2/Src-family kinase pathway in cortical neurons.
- Depolarization-induced 65zinc influx into cultured cortical neurons.
- Membrane-permeant chelators can attenuate Zn2+-induced cortical neuronal death.
- Apoptotic insults impair Na+, K+-ATPase activity as a mechanism of neuronal death mediated by concurrent ATP deficiency and oxidant stress.
- Neurodegeneration: cellular defences destroyed.
- NMDA antagonists exacerbate neuronal death caused by proteasome inhibition in cultured cortical and striatal neurons.
- Involvement of poly ADP ribosyl polymerase-1 in acute but not chronic zinc Toxicity.
- Transient decrease in F-actin may be necessary for translocation of proteins into dendritic spines.
Schools
Harvard Medical School
Procedures Preformed
- EMG (Electromyography)
Conditions Treated
- Inflammatory and Toxic Neuropathy
- Multiple Sclerosis (MS)
- Spinal Stenosis
Doctors Specialties
- Neurology
Accepted Insurances
Awards
- Elected Member
- Whos Who in Medicine and Healthcare
Education
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Massachusetts General Hospital -
Boston Children's Hospital -
Brigham and Women's Hospital -
Harvard Medical School
Hospital
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Stony Brook University Medical Center
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