Associate Professor, Surgery, University of Maryland School of Medicine
General Surgery
Dr. Douglas J. Turner, MD
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Douglas Turner practices General Surgery. He graduated from Johns Hopkins University School of Medicine. Johns Hopkins University School of Medicine is a top medical college. Johns Hopkins University School of Medicine is ranked 3/29 in Research/PrimaryCare. He has 2 awards "CMS Meaningful Use Stage 1 Certification" and "Fellow (FACS)". Douglas J. Turner is a published doctor and has 35 publications published. The lastest was: 'Overexpression of miR-214-3p in esophageal squamous cancer cells enhances sensitivity to cisplatin by targeting survivin directly and indirectly through CUG-BP1.' He accepts Medicare.
Publications
- Overexpression of miR-214-3p in esophageal squamous cancer cells enhances sensitivity to cisplatin by targeting survivin directly and indirectly through CUG-BP1.
- Modulation by miR-29b of intestinal epithelium homoeostasis through the repression of menin translation.
- Caveolin-1 enhances rapid mucosal restitution by activating TRPC1-mediated Ca2+ signaling.
- Jnk2 Deletion Disrupts Intestinal Mucosal Homeostasis and Maturation by Differentially Modulating RNA-binding Proteins HuR and CUGBP1.
- Inhibition of Smurf2 translation by miR-322/503 modulates TGF-β/Smad2 signaling and intestinal epithelial homeostasis.
- Src-mediated Caveolin-1 Phosphorylation Regulates Intestinal Epithelial Restitution by Altering Ca2+ Influx after Wounding.
- CANDIDATE GENES FOR LIMITING CHOLESTATIC INTESTINAL INJURY IDENTIFIED BY GENE EXPRESSION PROFILING.
- Pneumatosis and portal venous gas: do CT findings reassure?
- Sphingosine Kinase 1 overexpression stimulates intestinal epithelial cell proliferation through increased c-Myc translation.
- The RNA-binding protein CUG-BP1 increases survivin expression in oesophageal cancer cells through enhanced mRNA stability.
- Polyamines regulate intestinal epithelial restitution through TRPC1-mediated Ca2+ signaling by differentially modulating STIM1 and STIM2.
- Induced Expression of STIM1 Sensitizes Intestinal Epithelial Cells to Apoptosis by Modulating Store-Operated Ca(2+) Influx.
- miR-503 Represses CUGBP1 Translation by Recruiting CUGBP1 mRNA to P-Bodies.
- Activation of Wnt3a Signaling Stimulates Intestinal Epithelial Repair by Promoting c-Myc-Regulated Gene Expression.
- Regulation of Cyclin-Dependent Kinase 4 Translation through CUG-Binding Protein 1 and microRNA-222 by Polyamines.
- Sphingosine-1-phosphate regulates the expression of adherens junction protein e-cadherin and enhances intestinal epithelial cell barrier function.
- Polyamines regulate c-Myc translation through Chk2-dependent HuR phosphorylation.
- Polyamines regulate E-cadherin transcription through c-Myc modulating intestinal epithelial barrier function.
- Sphingosine-1-phosphate protects intestinal epithelial cells from apoptosis through the Akt signaling pathway.
- Management of adrenal lesions.
- Surgical site infection prevention: the importance of operative duration and blood transfusion--results of the first American College of Surgeons-National Surgical Qua...
- Induced TRPC1 expression increases protein phosphatase 2A sensitizing intestinal epithelial cells to apoptosis through inhibition of NF-kappaB activation.
- Polyamines regulate the stability of activating transcription factor-2 mRNA through RNA-binding protein HuR in intestinal epithelial cells.
- Polyamines are required for expression of Toll-like receptor 2 modulating intestinal epithelial barrier integrity.
- p53-dependent NDRG1 expression induces inhibition of intestinal epithelial cell proliferation but not apoptosis after polyamine depletion.
- Substance P regulates migration in rat intestinal epithelial cells.
- Bile salts induce resistance to apoptosis through NF-kappaB-mediated XIAP expression.
- Polyamines are required for phospholipase C-gamma1 expression promoting intestinal epithelial restitution after wounding.
- TRPC1 functions as a store-operated Ca2+ channel in intestinal epithelial cells and regulates early mucosal restitution after wounding.
- Taurodeoxycholate stimulates intestinal cell proliferation and protects against apoptotic cell death through activation of NF-kappaB.
- Akt kinase activation blocks apoptosis in intestinal epithelial cells by inhibiting caspase-3 after polyamine depletion.
- Regulation of adherens junctions and epithelial paracellular permeability: a novel function for polyamines.
- Activation of TGF-beta-Smad signaling pathway following polyamine depletion in intestinal epithelial cells.
- Polyamines regulate beta-catenin tyrosine phosphorylation via Ca(2+) during intestinal epithelial cell migration.
Schools
Johns Hopkins University School of Medicine
University Mi
Doctors Specialties
- Critical Care Surgery
- General Surgery
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Select
- Amerigroup (Wellpoint)
- Assurant Health
- Blue Card PPO
- Blue Preferred PPO
- CHAMPVA
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Commercial Insurance Company
- Southern Health PPO
- Coventry Health Care of Delaware PPO
- Emblem Health
- First Health (Coventry Health Care) PPO
- Geisinger Health Plan
- Golden Rule
- GEHA
- Choice Care PPO
- Mail Handlers Benefit Plan (MHBP)
- Maryland Physicians Care
- Medicaid
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- Mutual of Omaha
- State Farm
- Tricare
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
- Wells Fargo Insurance
- Wisconsin Physicians Service
Awards
- CMS Meaningful Use Stage 1 Certification
- Fellow (FACS)
Education
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University of Michigan Hospitals and Health Centers -
Johns Hopkins University School of Medicine
Hospital
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University of Maryland Medical Center -
University of Maryland Baltimore Washington Medical Center
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1457374647 |
| Last Name Of The Provider | TURNER |
| First Name Of The Provider | DOUGLAS |
| View All |
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