Professor, Medicine, University of Colorado School of Medicine
Pulmonology
Dr. Edward C. Dempsey, MD
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Edward Dempsey practices Pulmonology. He went to Harvard Medical School. Harvard Medical School is a one of the best medicals schools. Harvard Medical School has a rank of 1 in research and a 12 in primary care He has 2 awards "Regional Top Doctor" and "CMS Meaningful Use Stage 1 Certification". Doctor Edward C. Dempsey, MD has written publications. He has 50 publications published with PubMed. The lastest publication is: 'High frequency of K-ras codon 12 mutations in bronchoalveolar lavage fluid of patients at high risk for second primary lung cancer.' Edward Dempsey is registered with Medicare.
Publications
- Neprilysin regulates Pulmonary artery smooth muscle cell phenotype through a platelet-derived growth factor receptor-dependent mechanism.
- Exocrine pancreatic insufficiency syndrome in CBA/J mice. II. Biochemical studies.
- Detection of K-ras oncogene mutations by polymerase chain reaction-based ligase chain reaction.
- Involvement of protein kinase C in macrophage activation by poly(I.C).
- Platelet-induced vascular smooth muscle cell proliferation is modulated by the growth amplification factors serotonin and adenosine diphosphate.
- High frequency of K-ras codon 12 mutations in bronchoalveolar lavage fluid of patients at high risk for second primary Lung cancer.
- Peptide amidating activity in human bronchoalveolar lavage fluid.
- Fluorescence versus white-light bronchoscopy for detection of preneoplastic lesions: a randomized study.
- Pregnancy stimulation of DNA synthesis and uterine blood flow in the guinea pig.
- Selected isozymes of PKC contribute to augmented growth of fetal and neonatal bovine PA adventitial fibroblasts.
- Expansion of bronchial epithelial cell populations by in vitro culture of explants from dysplastic and histologically normal sites.
- Angiogenic squamous dysplasia in bronchi of individuals at high risk for Lung cancer.
- Unique growth properties of neonatal Pulmonary vascular cells: importance of time- and site-specific responses, cell-cell interaction, and synergy.
- Smooth muscle cell heterogeneity in Pulmonary and systemic vessels. Importance in vascular disease.
- Hypoxia-induced Pulmonary vascular remodeling: contribution of the adventitial fibroblasts.
- Hypoxia induces cell-specific changes in gene expression in vascular wall cells: implications for Pulmonary hypertension.
- Hypoxia stimulates proliferation of a unique cell population isolated from the bovine vascular media.
- Protein kinase C activation allows Pulmonary artery smooth muscle cells to proliferate to hypoxia.
- Enhanced growth of fetal and neonatal Pulmonary artery adventitial fibroblasts is dependent on protein kinase C.
- Smooth muscle cells isolated from discrete compartments of the mature vascular media exhibit unique phenotypes and distinct growth capabilities.
- Neonatal Lung side population cells demonstrate endothelial potential and are altered in response to hyperoxia-induced Lung simplification.
- Insulin-like growth factor I and protein kinase C activation stimulate Pulmonary artery smooth muscle cell proliferation through separate but synergistic pathways.
- Enhanced growth capacity of neonatal Pulmonary artery smooth muscle cells in vitro: dependence on cell size, time from birth, insulin-like growth factor I, and auto-ac...
- Overexpression of endothelin-1 and enhanced growth of Pulmonary artery smooth muscle cells from fawn-hooded rats.
- Chronic hypoxia induces exaggerated growth responses in pulmonary artery adventitial fibroblasts: potential contribution of specific protein kinase c isozymes.
- Heterogeneity in the proliferative response of bovine Pulmonary artery smooth muscle cells to mitogens and hypoxia: importance of protein kinase C.
- cAMP response element-binding protein content is a molecular determinant of smooth muscle cell proliferation and migration.
- Protein kinase C isozymes and the regulation of diverse cell responses.
- Pulmonary artery smooth muscle cells from Chronically hypoxic neonatal calves retain fetal-like and acquire new growth properties.
- Pregnancy-stimulated growth of vascular smooth muscle cells: importance of protein kinase C-dependent synergy between estrogen and platelet-derived growth factor.
- BQ123, an ETA receptor antagonist, inhibits endothelin-1-mediated proliferation of human Pulmonary artery smooth muscle cells.
- The Ca2+-dependent isozymes of protein kinase C are uniquely important in the enhanced growth of immature adventitial fibroblasts isolated from the bovine Pulmonary ar...
- Hypoxic growth of bovine Pulmonary artery smooth muscle cells: dependence on synergy, heterogeneity, and injury-induced phenotypic change.
- Divergent contractile and structural responses of the murine PKC-epsilon null pulmonary circulation to Chronic hypoxia.
- Divergent contractile and structural responses of the murine protein kinase C-epsilon null pulmonary circulation to Chronic hypoxia.
- Mice deficient in galectin-1 exhibit attenuated physiological responses to Chronic hypoxia-induced pulmonary hypertension.
- Selective expansion of fibroblast subpopulations from Pulmonary artery adventitia in response to hypoxia.
- Decreased neprilysin and pulmonary vascular remodeling in Chronic obstructive pulmonary disease.
- Vascular endothelial growth factor receptor 2-targeted chemoprevention of murine Lung tumors.
- A randomized phase II chemoprevention trial of 13-CIS retinoic acid with or without alpha tocopherol or observation in subjects at high risk for Lung cancer.
- Neprilysin null mice develop exaggerated pulmonary vascular remodeling in response to Chronic hypoxia.
- Lung Disease and PKCs.
- Rosiglitazone attenuates hypoxia-induced Pulmonary arterial remodeling.
- Chronic hypoxia diminishes the proliferative response of Guinea pig uterine artery vascular smooth muscle cells in vitro.
- Impact of customized videotape education on quality of life in patients with Chronic obstructive pulmonary disease.
- Role of rho in the increased migration of Pulmonary artery smooth muscle cells observed in the neprilysin knockout mouse.
- High prevalence of occult endobronchial malignancy in high risk patients with moderate sputum atypia.
- High prevalence of endobronchial malignancy in high-risk patients with moderate dysplasia in sputum.
- Protein kinase C-epsilon-null mice have decreased hypoxic Pulmonary vasoconstriction.
Schools
Harvard Medical School
YaleNew Haven Medical Center Program
University Of Colorado University Hospital Program
Procedures Preformed
- Thoracentesis
Conditions Treated
- Asthma
- Bronchiectasis
- Chronic Pulmonary Heart Diseases (incl. Pulmonary Hypertension)
- Classic Polyarteritis Nodosa
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Pathway X Bronze Direct CACA IN HIX
- Anthem Blue Cross Blue Shield PPO
- Blue Card PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- LocalPlus
- Open Access Plus
- Cofinity PPO
- Commercial Insurance Company
- GEHA
- Humana
- Mail Handlers Benefit Plan (MHBP)
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- Mutual of Omaha
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Awards
- Regional Top Doctor
- CMS Meaningful Use Stage 1 Certification
Education
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University of Colorado -
Yale-New Haven Hospital -
Harvard Medical School
Hospital
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University of Colorado Hospital
Drug Facts
| NPI NUMBER | 1992895676 | |
| NPPES Provider LastName | DEMPSEY | |
| NPPES Provider FirstName | EDWARD | |
| NPPES Provider ZIPCode | 800452545 | |
| NPPES Provider State | CO | |
| Specialty Description | Pulmonary Disease | |
| Total Claim Count | 63.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1992895676 |
| Last Name Of The Provider | DEMPSEY |
| First Name Of The Provider | EDWARD |
| View All |
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