Physician
Neurology
Dr. Edward L. Hogan, MD
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Dr. Edward L. Hogan practices Neurology. He went to Tufts University School of Medicine. Tufts University School of Medicine is ranked 49/52 in Research/PrimaryCare. Edward Hogan is also a published MD. He has 101 paper published. The most recent paper is: Increased calpain expression in activated glial and inflammatory cells in experimental allergic encephalomyelitis.
Publications
- Signaling and Regulatory Functions of Bioactive Sphingolipids as Therapeutic Targets in Multiple Sclerosis.
- Novel myelin penta- and hexa-acetyl-galactosyl-ceramides: structural characterization and immunoreactivity in cerebrospinal fluid.
- Cell-specific expression of neutral glycosphingolipids in vertebrate brain: immunochemical localization of 3-O-acetyl-sphingosine-series glycolipid(s) in myelin and ol...
- Higher calpastatin levels correlate with resistance to calpain-mediated proteolysis and neuronal apoptosis in juvenile rats after spinal cord injury.
- Early induction of secondary injury factors causing activation of calpain and mitochondria-mediated neuronal apoptosis following spinal cord injury in rats.
- Molecular evidence of apoptotic death in malignant brain tumors including glioblastoma multiforme: upregulation of calpain and caspase-3.
- 3-O-acetyl-sphingosine-series myelin glycolipids: characterization of novel 3-O-acetyl-sphingosine galactosylceramide.
- Lead encephalopathy in the developing rat: effect on cortical ontogenesis.
- Fatty acid composition of cerebrosides, sulphatides and ceramides in murine leucodystrophy: the quaking mutant.
- Transient improvement in Guillain-Barré syndrome after megadose intravenous methylprednisolone.
- Incorporation of lipids into subcellular fractions of brain of quaking mutant.
- Proteolipid protein: synthesis and assembly into quaking mouse myelin.
- Composition of myelin proteins in murine genetic myelin dysgenesis: the quaking mutant.
- Confirmation of locus heterogeneity in the pure form of familial spastic paraplegia.
- Immunoglobulin G in multiple sclerosis brain.
- Abnormal T cell subpopulations and circulating immune complexes in the Guillain-Barré syndrome and multiple sclerosis.
- Fatty acid composition of cerebrosides, sulphatides and ceramides in murine sudanophilic leucodystrophy: the Jimpy mutant.
- Progression of hypertensive intracerebral hemorrhage.
- Moyamoya disease in Taiwan.
- Lead encephalopathy in the developing rat: effect upon myelination.
- Metabolism in vivo of brain galactolipids: the Jimpy mutant.
- Effect of lead intoxication on the postnatal growth of the rat nervous system.
- Undernutrition in the developing rat: effect upon myelination.
- Effects of fish oil supplementation on acute ischemic brain injury in the rat.
- Establishing brain death in South Carolina: a clinician's guide.
- Rapid isolation of genomic DNA from animal tissues.
- Polyethylene glycol-conjugated superoxide dismutase and catalase reduce ischemic brain injury.
- Effect of plasma glucose on infarct size in focal cerebral ischemia-reperfusion.
- Composition of cerebral lipids in murine leucodystrophy: the quaking mutant.
- Cerebral lipid composition during myelination in murine genetic leukodystrophies.
- Composition of cerebral lipids in murine sudanophilic leucodystrophy: the Jimpy mutant.
- Abnormal glycosylation of myelin-associated glycoprotein in quaking mouse brain.
- Acylation of myelin proteolipid protein in subcellular fractions of rat brainstem.
- Glycosphingolipids of chicken skeletal muscle in early development and genetic dystrophy.
- Lipid content of swine influenza and other vaccines.
- Receptor-linked hydrolysis of phosphoinositides and production of prostacyclin in cerebral endothelial cells.
- Arachidonic acid and its metabolites in cerebral ischemia.
- Increased turnover of myelin proteolipid protein in quaking mouse brain.
- Pentoxifylline in acute nonhemorrhagic stroke. A randomized, placebo-controlled double-blind trial.
- In situ visualization of glycosphingolipids on thin-layer chromatograms using lectin-peroxidase conjugates.
- Serum gangliosides in cerebral astrocytoma.
- Tissue calcium levels in CaCl2-induced myelopathy.
- Brain calcium content in ischemic infarction.
- Myelin basic protein gene expression in quaking, jimpy, and myelin synthesis-deficient mice.
- Schilder''s diffuse sclerosis: a biochemical and ultrastructural study of myelinoclastic demyelinaton.
- Characterization of the fraction obtained from the CNS of Jimpy mice by a procedure for myelin isolation.
- Basic proteins of rodent peripheral nerve myelin: immunochemical identification of the 21.5K, 18.5K, 17K, 14K, and P2 proteins.
- Increased thromboxane level in experimental spinal cord injury.
- Quantitation of the in vitro neuroblastoma response to exogenous, purified gangliosides.
- Therapeutic model for experimental spinal cord injury in the rat: I. Mortality and motor deficit.
- A simplified procedure for the preparation of tritiated GM1 ganglioside and other glycosphingolipids.
- Sialylation of lacto-N-neohexaosylceramide by sialyltransferase from embryonic chicken muscle.
- Synthesis and acylation of myelin proteolipid protein in quaking mouse brain.
- Leukotriene B4 release and polymorphonuclear cell infiltration in spinal cord injury.
- Glycosphingolipid biosynthesis during myogenesis of rat L6 cells in vitro.
- Thrombin activates mitogen-activated protein kinase in primary astrocyte cultures.
- Activity of 2'',3''-cyclic nucleotide 3''-phosphohydrolase in human cerebrospinal fluid.
- Biosynthesis of GM1b and similar neolactoseries gangliosides by a partially purified chicken skeletal muscle sialyltransferase. Effect of sphingomyelin and acetylcholine.
- Purification of a calcium-activated neutral proteinase from bovine brain.
- A revised structure for the disialosyl globo-series gangliosides of human erythrocytes and chicken skeletal muscle.
- Ca2+-accumulation in experimental spinal cord trauma.
- A role for calcium and activation of neutral proteinase in myelinolysis?
- Alteration of thromboxane and prostacyclin levels in experimental spinal cord injury.
- Combined TUNEL and double immunofluorescent labeling for detection of apoptotic mononuclear phagocytes in autoimmune demyelinating disease.
- The regional and subcellular distribution of calcium activated neutral proteinase (CANP) in the bovine central nervous system.
- Vascular permeability in experimental spinal cord injury.
- Kininogen and kinin in experimental spinal cord injury.
- Problematic issues in spinal cord injury.
- Xanthine oxidase in experimental spinal cord injury.
- A sensitive fluorometric method for measurement of vascular permeability in spinal cord injury.
- Protective effect of methylprednisolone on vascular injury in rat spinal cord injury.
- Expression of myelin protein genes in quaking mouse brain.
- Temporal profile of thromboxane-prostacyclin imbalance in experimental spinal cord injury.
- Effect of U-50488h, a selective opioid kappa receptor agonist, on vascular injury after spinal cord trauma.
- Activation of JNK/SAPK in primary glial cultures: II. Differential activation of kinase isoforms corresponds to their differential expression.
- A disialoganglioside of the globo-series from chicken skeletal muscle.
- Sialylation of lacto-N-neotetraosyl ceramide by a solubilized sialyltransferase(s) from chicken skeletal muscle: effect of phosphatidylcholine and sphingomyelin.
- The effects of spinal cord trauma on myelin.
- Maternal alcohol consumption increases sphingosine levels in the brains of progeny mice.
- Changes in myelin and axonal proteins in CaCl2-induced myelopathy in rat spinal cord.
- Molecular and anatomical correlates of spinal cord injury.
- Proteolysis in quaking mouse brain and spinal cord.
- Degradation of cytoskeletal proteins in experimental spinal cord injury.
- Calcium-stimulated proteolysis in myelin: evidence for a Ca2+-activated neutral proteinase associated with purified myelin of rat CNS.
- Molecular characterization of gangliotetraosylceramide (GA1) in normal human brain and its developmental change.
- Proteolytic enzymes in experimental spinal cord injury.
- Distribution of calcium activated neutral proteinase (mM CANP) in myelin and cytosolic fractions in bovine brain white matter.
- Calcium-activated mediators of secondary injury in the spinal cord.
- Calpain expression in lymphoid cells. Increased mRNA and protein levels after cell activation.
- Differential regulation of myelin gene expression in SV40 T antigen-transfected rat glioma C6 cells.
- Effects of detergents on Ca(2+)-activated neural proteinase activity (calpain) in neural and non-neural tissue: a comparative study.
- A new mechanism of methylprednisolone and other corticosteroids action demonstrated in vitro: inhibition of a proteinase (calpain) prevents myelin and cytoskeletal pro...
- The multimolecular cascade of spinal cord injury. Studies on prostanoids, calcium, and proteinases.
- Ganglioside-modulated proteolysis by Ca2(+)-activated neutral proteinase (CANP): a role of glycoconjugates in CANP regulation.
- Ca2+-mediated degradation of central nervous system (CNS) proteins: topographic and species variation.
- Inhibition of proteolysis by a cyclooxygenase inhibitor, indomethacin.
- Chromatographic resolution and quantitative assay of CNS tissue sphingoids and sphingolipids.
- Increased calpain expression in activated glial and inflammatory cells in experimental allergic encephalomyelitis.
- E-64-d prevents both calpain upregulation and apoptosis in the lesion and penumbra following spinal cord injury in rats.
- Inhibition of calpain-mediated apoptosis by E-64 d-reduced immediate early gene (IEG) expression and reactive astrogliosis in the lesion and penumbra following spinal ...
Schools
Tufts University
Barnes Hospital Wash
Boston City Hospital
Pb Brigham Hospital
Doctors Specialties
- Neurology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Direct Access w/HSA
- Choice Care PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
Education
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Boston University Medical Center -
Brigham and Women's Hospital -
Washington University - Barnes-Jewish Hospital -
Tufts University School of Medicine
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