Physician
Endocrinology
Dr. Elena Ambrogini, MD
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Elena Ambrogini works in the field of Endocrinology. He went to University of Arkansas for Medical Sciences. University of Arkansas for Medical Sciences is ranked 83/35 in Research/PrimaryCare. Doctor Elena Ambrogini, MD is a published MD. He has 25 papers published. The most current publication "FoxO proteins restrain osteoclastogenesis and bone resorption by attenuating H2O2 accumulation."
Publications
- Estrogen receptor-? signaling in osteoblast progenitors stimulates cortical bone accrual.
- FoxO proteins restrain osteoclastogenesis and bone resorption by attenuating H2O2 accumulation.
- MEN1 gene alterations do not correlate with the phenotype of sporadic primary hyperparathyroidism.
- A quick intraoperative parathyroid hormone assay in the surgical management of patients with primary hyperparathyroidism: a study of 206 consecutive cases.
- Spontaneous short-term remission of primary hyperparathyroidism from infarction of a parathyroid adenoma.
- Autoimmune hepatitis during intravenous glucocorticoid pulse therapy for Graves'' ophthalmopathy treated successfully with glucocorticoids themselves.
- Post-menopausal osteoporosis: is it an autoimmune disease?
- Different somatic alterations of the HRPT2 gene in a patient with recurrent sporadic primary hyperparathyroidism carrying an HRPT2 germline mutation.
- Hyperparathyroidism 2 gene (HRPT2, CDC73) and parafibromin studies in two patients with primary hyperparathyroidism and uncertain pathological assessment.
- Identification and functional characterization of loss-of-function mutations of the calcium-sensing receptor in four Italian kindreds with familial hypocalciuric hyper...
- Surgery or surveillance for mild asymptomatic primary hyperparathyroidism: a prospective, randomized clinical trial.
- Should parafibromin staining replace HRTP2 gene analysis as an additional tool for histologic diagnosis of parathyroid carcinoma?
- Glucocorticoids and tumor necrosis factor ? increase oxidative stress and suppress Wnt protein signaling in osteoblasts.
- Decreased oxidative stress and greater bone anabolism in the aged, when compared to the young, murine skeleton with parathyroid hormone administration.
- Oxidative stress stimulates apoptosis and activates NF-kappaB in osteoblastic cells via a PKCbeta/p66shc signaling cascade: counter regulation by estrogens or androgens.
- FoxO-mediated defense against oxidative stress in osteoblasts is indispensable for skeletal homeostasis in mice.
- The estrogen receptor-alpha in osteoclasts mediates the protective effects of estrogens on cancellous but not cortical bone.
- The sulfaphenazole-sensitive pathway acts as a compensatory mechanism for impaired nitric oxide availability in patients with primary hyperparathyroidism. Effect of su...
- Estrogens attenuate oxidative stress and the differentiation and apoptosis of osteoblasts by DNA-binding-independent actions of the ERalpha.
- Increased lipid oxidation causes oxidative stress, increased peroxisome proliferator-activated receptor-gamma expression, and diminished pro-osteogenic Wnt signaling i...
- Morphometric vertebral fractures in postmenopausal women with primary hyperparathyroidism.
- Genetic analyses in familial isolated hyperparathyroidism: implication for clinical assessment and surgical management.
- Genetic analyses of the HRPT2 gene in primary hyperparathyroidism: germline and somatic mutations in familial and sporadic parathyroid tumors.
- Congenital hypothyroidism due to a new deletion in the sodium/iodide symporter protein.
Doctors Specialties
- Endocrinology
Accepted Insurances
- Aetna
- Blue Cross Blue Shield
- Medicare MCR
Education
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University of Arkansas for Medical Sciences -
University of Pisa Faculty of Medicine
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