Professor, Medicine, University of Colorado School of Medicine
Infectious Disease
Dr. Elizabeth Connick, MD
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Elizabeth Connick works in the field of Infectious Disease. She went to Harvard Medical School. Harvard Medical School is a top medical university. Harvard Medical School is ranked 1 in research and 12 in primary care medicine She has 2 awards "Regional Top Doctor" and "CMS Meaningful Use Stage 1 Certification". Dr. Elizabeth Connick has written publications. She has 84 publications published with PubMed. The lastest publication is: 'Immunologic responses associated with 12 weeks of combination antiretroviral therapy consisting of zidovudine, lamivudine, and ritonavir: results of AIDS Clinical Tria...' Elizabeth Connick is registered with Medicare.
Publications
- Germinal Center T Follicular Helper Cells Are Highly Permissive to HIV-1 and Alter Their Phenotype during Virus Replication.
- Distinct Patterns of Bcl-2 Expression Occur in R5- and X4-Tropic HIV-1-Producing Lymphoid Tissue Cells Infected Ex Vivo.
- Compartmentalization of Simian Immunodeficiency Virus Replication within Secondary Lymphoid Tissues of Rhesus Macaques Is Linked to Disease Stage and Inversely Related...
- Impact of circadian misalignment on energy metabolism during simulated nightshift work.
- Engagement-in-Care During the First 5 Years After HIV Diagnosis: Data from a Cohort of Newly HIV-Diagnosed Individuals in a Large US City.
- Impaired Nef function is associated with early control of HIV-1 viremia.
- Safety of Excisional Inguinal Lymph Node Biopsies Performed for Research Purposes in HIV-1-Infected Women and Men.
- PLASMA LL-37 CORRELATES WITH VITAMIN D AND IS REDUCED IN HIV-1 INFECTED INDIVIDUALS NOT RECEIVING ANTIRETROVIRAL THERAPY.
- CCR5 Expression is Reduced in Lymph Nodes of HIV-1-Infected Women Compared to Men, But Does Not Mediate Sex Differences in Viral Loads.
- Initial Linkage and Subsequent Retention in HIV Care for a Newly Diagnosed HIV-Infected Cohort in Denver, Colorado.
- Inadequate T follicular cell help impairs B cell immunity during HIV infection.
- Differential infection patterns of CD4+ T cells and lymphoid tissue viral burden distinguish progressive and nonprogressive lentiviral infections.
- Follicular regulatory T cells impair follicular T helper cells in HIV and SIV infection.
- Suppression of Foxo1 Activity and Down-Modulation of CD62L (L-Selectin) in HIV-1 Infected Resting CD4 T Cells.
- The setpoint study (ACTG A5217): effect of immediate versus deferred antiretroviral therapy on virologic set point in recently HIV-1-infected individuals.
- GagCM9-specific CD8+ T cells expressing limited public TCR clonotypes do not suppress SIV replication in vivo.
- HLA-DR+ CD38+ CD4+ T lymphocytes have elevated CCR5 expression and produce the majority of R5-tropic HIV-1 RNA in vivo.
- Humanized Rag1-/- ?c-/- mice support multilineage hematopoiesis and are susceptible to HIV-1 infection via systemic and vaginal routes.
- Mycobacterium kansasii cutaneous abscesses occurring as immune reconstitution inflammatory syndrome.
- Augmented HIV-specific interferon-gamma responses, but impaired lymphoproliferation during interruption of antiretroviral treatment initiated in primary HIV infection.
- Sex, race, and geographic region influence clinical outcomes following primary HIV-1 infection.
- Effects of HIV-1 gp120 and protease inhibitors on apoptotic susceptibility of CD34+ hematopoietic progenitor cells.
- Blood myeloid dendritic cells from HIV-1-infected individuals display a proapoptotic profile characterized by decreased Bcl-2 levels and by caspase-3+ frequencies that...
- Prevalence of primary resistance at baseline in acutely and recently infected subjects enrolled in AIDS clinical trials group protocol 371.
- Impaired replication capacity of acute/early viruses in persons who become HIV controllers.
- Treatment with recombinant growth hormone is associated with modest improvement in CD4 lymphocyte reconstitution in HIV-infected persons on antiretroviral therapy: res...
- Antiretroviral therapy in acute and recent HIV infection: a prospective multicenter stratified trial of intentionally interrupted treatment.
- Dementia with features of Alzheimer''s disease and HIV-associated dementia in an elderly man with AIDS.
- Phenotypic and functional characterization of HIV-1-specific CD4+CD8+ double-positive T cells in early and chronic HIV-1 infection.
- Tolerability and efficacy of PI versus NNRTI-based regimens in subjects receiving HAART during acute or early HIV infection.
- SDF-1alpha is a potent inducer of HIV-1-Specific CD8+ T-cell chemotaxis, but migration of CD8+ T cells is impaired at high viral loads.
- Plasmacytoid and myeloid dendritic cells with a partial activation phenotype accumulate in lymphoid tissue during asymptomatic chronic HIV-1 infection.
- Mucosal transmission of R5 and X4 tropic HIV-1 via vaginal and rectal routes in humanized Rag2-/- gammac -/- (RAG-hu) mice.
- Fish oil and fenofibrate for the treatment of hypertriglyceridemia in HIV-infected subjects on antiretroviral therapy: results of ACTG A5186.
- Immunophenotypic alterations in acute and early HIV infection.
- Programmed death 1 expression on HIV-specific CD4+ T cells is driven by viral replication and associated with T cell dysfunction.
- CTL fail to accumulate at sites of HIV-1 replication in lymphoid tissue.
- HIV-1 infection and CD4 T cell depletion in the humanized Rag2-/-gamma c-/- (RAG-hu) mouse model.
- alpha-Defensins 1, 2, and 3 are expressed by granulocytes in lymphoid tissues of HIV-1-seropositive and -seronegative individuals.
- Lymphoid follicles are sites of heightened human immunodeficiency virus type 1 (HIV-1) replication and reduced antiretroviral effector mechanisms.
- Challenges in the study of patients with HIV type 1 seroconversion.
- Incomplete antibody evolution and seroreversion after treatment of primary HIV type 1 infection: what is the clinical significance?
- Persistence of Kaposi sarcoma-associated herpesvirus (KSHV)-infected cells in KSHV/HIV-1-coinfected subjects without KSHV-associated diseases.
- Immune reconstitution inflammatory syndrome associated with Kaposi sarcoma during potent antiretroviral therapy.
- Long-term changes in circulating CD4 T lymphocytes in virologically suppressed patients after 6 years of highly active antiretroviral therapy.
- Genetic basis of hypersusceptibility to protease inhibitors and low replicative capacity of human immunodeficiency virus type 1 strains in primary infection.
- Incomplete immune reconstitution after initiation of highly active antiretroviral therapy in human immunodeficiency virus-infected patients with severe CD4+ cell deple...
- MDR1 gene polymorphisms and phase 1 viral decay during HIV-1 infection: an adult AIDS Clinical Trials Group study.
- Relationship between in vitro human immunodeficiency virus type 1 replication rate and virus load in plasma.
- Role of Baseline pol Genotype in HIV-1 Fitness Evolution.
- Suppression of leukemia inhibitor factor in lymphoid tissue in primary HIV infection: absence of HIV replication in gp130-positive cells.
- CD8+ T cells that express CD4 on their surface (CD4dimCD8bright T cells) recognize an antigen-specific target, are detected in vivo, and can be productively infected b...
- Microanatomic relationships between CD8+ cells and HIV-1-producing cells in human lymphoid tissue in vivo.
- Limited immune restoration after 3 years'' suppression of HIV-1 replication in patients with moderately advanced disease.
- Antiretroviral-drug resistance among patients recently infected with HIV.
- Immune reconstitution in HIV-1-infected individuals treated with potent antiretroviral therapy.
- Relationship between human immunodeficiency virus type 1 (HIV-1)-specific memory cytotoxic T lymphocytes and virus load after recent HIV-1 seroconversion.
- Multiple CD4+ cell kinetic patterns and their relationships with baseline factors and virological responses in HIV type 1 patients receiving highly active antiretrovir...
- A potential role for interleukin-7 in T-cell homeostasis.
- Ototoxicity associated with use of nucleoside analog reverse transcriptase inhibitors: a report of 3 possible cases and review of the literature.
- Productive infection of T cells in lymphoid tissues during primary and early human immunodeficiency virus infection.
- Reduced susceptibility of human immunodeficiency virus type 1 (HIV-1) from patients with primary HIV infection to nonnucleoside reverse transcriptase inhibitors is ass...
- Phase 1 trial of a single dose of recombinant human interleukin-12 in human immunodeficiency virus-infected patients with 100-500 CD4 cells/microL.
- Shipment impairs lymphocyte proliferative responses to microbial antigens.
- Prolonged survival and tissue trafficking following adoptive transfer of CD4zeta gene-modified autologous CD4(+) and CD8(+) T cells in human immunodeficiency virus-inf...
- Response to immunization with recall and neoantigens after prolonged administration of an HIV-1 protease inhibitor-containing regimen. ACTG 375 team. AIDS Clinical Tri...
- Effect of zidovudine resistance mutations on virologic response to treatment with zidovudine-lamivudine-ritonavir: genotypic analysis of human immunodeficiency virus t...
- Immune reconstitution in the first year of potent antiretroviral therapy and its relationship to virologic response.
- Rapid accumulation of human immunodeficiency virus (HIV) in lymphatic tissue reservoirs during acute and early HIV infection: implications for timing of antiretroviral...
- Thymic size and lymphocyte restoration in patients with human immunodeficiency virus infection after 48 weeks of zidovudine, lamivudine, and ritonavir therapy.
- Alteration of complement protein levels after antiretroviral therapy in HIV-infected persons.
- Reduced antiretroviral drug susceptibility among patients with primary HIV infection.
- Perforin is not co-expressed with granzyme A within cytotoxic granules in CD8 T lymphocytes present in lymphoid tissue during chronic HIV infection.
- Efficacy and safety of delavirdine mesylate with zidovudine and didanosine compared with two-drug combinations of these agents in persons with HIV disease with CD4 cou...
- HIV infection perturbs DNA content of lymphoid cells: partial correction after ''suppression'' of virus replication.
- Increases in T cell telomere length in HIV infection after antiretroviral combination therapy for HIV-1 infection implicate distinct population dynamics in CD4+ and CD...
- Small differences in CD4+ T-cell production may go unnoticed.
- Characterization of viral dynamics in human immunodeficiency virus type 1-infected patients treated with combination antiretroviral therapy: relationships to host fact...
- Immunologic responses associated with 12 weeks of combination antiretroviral therapy consisting of zidovudine, lamivudine, and ritonavir: results of AIDS Clinical Tria...
- In vivo analysis of Fas/FasL interactions in HIV-infected patients.
- Tension myalgia versus myoadenylate deaminase deficiency: a case report.
- HIV-specific cellular and humoral immune responses in primary HIV infection.
- In vitro effect of interleukin-12 on antigen-specific lymphocyte proliferative responses from persons infected with human immunodeficiency virus type 1.
Schools
Harvard Medical School
New York Presbyterian Hospital Columbia Campus Program
University Of Colorado University Hospital Program
Doctors Specialties
- Infectious Disease Medicine
- Infectious Disease
Accepted Insurances
Awards
- Regional Top Doctor
- CMS Meaningful Use Stage 1 Certification
Education
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University of Colorado -
New York Presbyterian Hospital - Columbia Campus -
Harvard Medical School
Hospital
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University of Colorado Hospital
Drug Facts
| NPI NUMBER | 1255421939 | |
| NPPES Provider LastName | CONNICK | |
| NPPES Provider FirstName | ELIZABETH | |
| NPPES Provider ZIPCode | 800452545 | |
| NPPES Provider State | CO | |
| Specialty Description | Infectious Disease | |
| Total Claim Count | 483.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1255421939 |
| Last Name Of The Provider | CONNICK |
| First Name Of The Provider | ELIZABETH |
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