Chair, Department of Neuroscience Director, Friedman Brain Institute Icahn School of Medicine at Mount Sinai
Psychiatry
Dr. Eric J. Nestler, MD
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Eric J. Nestler, MD practices Psychiatry. He researched medicine at Yale University School of Medicine. Yale University School of Medicine is one of the top medical schools in the US. Yale University School of Medicine is ranked 7 in research and 57 in primary care medicine He received awards:"Elected Member". Eric Nestler is also a published physician. He has 48 publication published. The most recent publication is: Essential Role of Mesolimbic Brain-Derived Neurotrophic Factor in Chronic Social Stress-Induced Depressive Behaviors.
Publications
- In vivo imaging identifies temporal signature of D1 and D2 medium spiny neurons in cocaine reward.
- Bidirectional Synaptic Structural Plasticity after Chronic Cocaine Administration Occurs through Rap1 Small GTPase Signaling.
- Essential Role of Mesolimbic Brain-Derived Neurotrophic Factor in Chronic Social Stress-Induced Depressive Behaviors.
- The role of ventral striatal cAMP signaling in stress-induced behaviors.
- Role of Tet1 and 5-hydroxymethylcytosine in cocaine action.
- Epigenetic basis of opiate suppression of Bdnf gene expression in the ventral tegmental area.
- Functional profile of a novel modulator of serotonin, dopamine, and glutamate neurotransmission.
- SIRT1-FOXO3a Regulate Cocaine Actions in the Nucleus Accumbens.
- Morphine and cocaine increase serum- and glucocorticoid-inducible kinase 1 activity in the ventral tegmental area.
- Locus-specific epigenetic remodeling controls addiction- and depression-related behaviors.
- Regulation of chromatin states by drugs of abuse.
- Cell-type specific expression of p11 controls cocaine reward.
- Analytical tools and current challenges in the modern era of neuroepigenomics.
- β-catenin mediates stress resilience through Dicer1/microRNA regulation.
- Functional Role of the N-Terminal Domain of ΔFosB in Response to Stress and Drugs of Abuse.
- Epigenetic signaling in psychiatric disorders: stress and depression.
- Threonine 149 Phosphorylation Enhances ΔFosB Transcriptional Activity to Control Psychomotor Responses to Cocaine.
- The neural rejuvenation hypothesis of cocaine addiction.
- Loss of BDNF Signaling in D1R-Expressing NAc Neurons Enhances Morphine Reward by Reducing GABA Inhibition.
- Epigenetic Signaling in Psychiatric Disorders.
- Neurodegenerative dementias: connecting psychiatry and neurology through a shared neurobiology.
- Prefrontal Cortical Circuit for Depression- and Anxiety-Related Behaviors Mediated by Cholecystokinin: Role of ΔFosB.
- G9a influences neuronal subtype specification in striatum.
- Essential role of poly(ADP-ribosyl)ation in cocaine action.
- Epigenetic Mechanisms of Depression.
- Fluoxetine exposure during adolescence alters responses to aversive stimuli in adulthood.
- Effects of Striatal ΔFosB Overexpression and Ketamine on Social Defeat Stress-Induced Anhedonia in Mice.
- Enduring Deficits in Brain Reward Function after Chronic Social Defeat in Rats: Susceptibility, Resilience, and Antidepressant Response.
- Cellular basis of memory for addiction.
- ΔFosB Induction in Striatal Medium Spiny Neuron Subtypes in Response to Chronic Pharmacological, Emotional, and Optogenetic Stimuli.
- Stress and CRF gate neural activation of BDNF in the mesolimbic reward pathway.
- Fluoxetine Epigenetically Alters the CaMKIIα Promoter in Nucleus Accumbens to Regulate ΔFosB Binding and Antidepressant Effects.
- Essential role of SIRT1 signaling in the nucleus accumbens in cocaine and morphine action.
- The brain reward circuitry in mood disorders.
- ΔFosB Induction in Prefrontal Cortex by Antipsychotic Drugs is Associated with Negative Behavioral Outcomes.
- Human Transcriptome and Chromatin Modifications: An ENCODE Perspective.
- Epigenetic mechanisms of drug addiction.
- Rapid regulation of depression-related behaviours by control of midbrain dopamine neurons.
- Transcriptional mechanisms of drug addiction.
- Proenkephalin mediates the enduring effects of adolescent cannabis exposure associated with adult opiate vulnerability.
- Epigenetic mechanisms of depression and antidepressant action.
- An Essential Role for {Delta}FosB in the Median Preoptic Nucleus in the Sustained Hypertensive Effects of Chronic Intermittent Hypoxia.
- CREB regulation of nucleus accumbens excitability mediates social isolation-induced behavioral deficits.
- PSMC5, a 19S Proteasomal ATPase, Regulates Cocaine Action in the Nucleus Accumbens.
- The origins of molecular psychiatry.
- Overexpression of DeltaFosB in nucleus accumbens mimics the protective addiction phenotype, but not the protective depression phenotype of environmental enrichment.
- Self-administration of ethanol, cocaine, or nicotine does not decrease the soma size of ventral tegmental area dopamine neurons.
Schools
Yale University
Doctors Specialties
- Psychiatry
Accepted Insurances
Awards
- Elected Member
Education
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Yale University School of Medicine
Hospital
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Mount Sinai Hospital
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