Physician
Infectious Disease
Dr. Fayyaz Sutterwala, MD
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Fayyaz Sutterwala, MD works in the field of Infectious Disease. He has 1 awards "Elected Member". Doctor Fayyaz Sutterwala, MD is also published. He has 42 publications published. The lastest publication: "Reversal of proinflammatory responses by ligating the macrophage Fcgamma receptor type I.' Dr. Fayyaz Sutterwala accepts all Medicare patients.
Publications
- Cutting edge: NLRP12 controls dendritic and myeloid cell migration to affect contact hypersensitivity.
- Cutting edge: mutation of Francisella tularensis mviN leads to increased macrophage absent in melanoma 2 inflammasome activation and a loss of virulence.
- Fungal pathogen recognition by the NLRP3 inflammasome.
- A Yersinia effector protein promotes virulence by preventing inflammasome recognition of the type III secretion system.
- Caspase-1 independent IL-1beta production is critical for host resistance to mycobacterium tuberculosis and does not require TLR signaling in vivo.
- Necrotic cells trigger a sterile inflammatory response through the Nlrp3 inflammasome.
- Cutting edge: Candida albicans hyphae formation triggers activation of the Nlrp3 inflammasome.
- Malarial hemozoin activates the NLRP3 inflammasome through Lyn and Syk kinases.
- Inflammasome-mediated regulation of hepatic stellate cells.
- Acetaminophen-induced hepatotoxicity in mice is dependent on Tlr9 and the Nalp3 inflammasome.
- NLRC4/IPAF: a CARD carrying member of the NLR family.
- The Nalp3 inflammasome is essential for the development of silicosis.
- The tiny conductor: immune regulation via commensal organisms.
- Immune recognition of Pseudomonas aeruginosa mediated by the IPAF/NLRC4 inflammasome.
- ASC/PYCARD and caspase-1 regulate the IL-18/IFN-gamma axis during Anaplasma phagocytophilum infection.
- The inflammasome in pathogen recognition and inflammation.
- MDP-induced interleukin-1beta processing requires Nod2 and CIAS1/NALP3.
- Expression of interleukin-10 in intestinal lymphocytes detected by an interleukin-10 reporter knockin tiger mouse.
- The inflammasome: first line of the immune response to cell stress.
- Role of the caspase-1 inflammasome in Salmonella typhimurium pathogenesis.
- Critical role for NALP3/CIAS1/Cryopyrin in innate and adaptive immunity through its regulation of caspase-1.
- TLR11 activation of dendritic cells by a protozoan profilin-like protein.
- Transforming growth factor-beta controls T helper type 1 cell development through regulation of natural killer cell interferon-gamma.
- Sensing damage by the NLRP3 inflammasome.
- Inhibitors of apoptosis proteins and IL-1β: a tangled relationship.
- Sterile inflammatory responses mediated by the NLRP3 inflammasome.
- The NLRP3 inflammasome: a sensor of immune danger signals.
- Autoinflammatory syndromes and infections: pathogenetic and clinical implications.
- Sensing pathogens and danger signals by the inflammasome.
- Maturation modulates caspase-1-independent responses of dendritic cells to Anthrax lethal toxin.
- Immunology: cascade into clarity.
- Crucial role for the Nalp3 inflammasome in the immunostimulatory properties of aluminium adjuvants.
- IL-12/23p40-dependent clearance of Anaplasma phagocytophilum in the murine model of human anaplasmosis.
- NALP3: a key player in caspase-1 activation.
- Leishmania major-human macrophage interactions: cooperation between Mac-1 (CD11b/CD18) and complement receptor type 1 (CD35) in promastigote adhesion.
- Cooperation between CR1 (CD35) and CR3 (CD 11b/CD18) in the binding of complement-opsonized particles.
- Selective suppression of interleukin-12 induction after macrophage receptor ligation.
- Structural model of cytochrome b559 in photosystem II based on a mutant with genetically fused subunits.
- Reversal of proinflammatory responses by ligating the macrophage Fcgamma receptor type I.
- The taming of IL-12: suppressing the production of proinflammatory cytokines.
- Suppression of Il-12 transcription in macrophages following Fc gamma receptor ligation.
Schools
Temple University
Doctors Specialties
- Infectious Disease Medicine
- Infectious Disease
Accepted Insurances
- Choice Plus POS II
- Blue Card PPO
- Blue Cross Blue Shield of Illinois PPO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- First Health (Coventry Health Care) PPO
- Health Alliance PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- Alliance Select
Awards
- Elected Member
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1790866200 |
| Last Name Of The Provider | SUTTERWALA |
| First Name Of The Provider | FAYYAZ |
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