Professor, Internal Medicine, University of Cincinnati College of Medicine
Hematology
Dr. George F. Atweh, MD
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George F. Atweh, MD practices Hematology. He has 2 awards "Elected Member" and "Top MD". Doctor George F. Atweh, MD has written publications. He has 65 publications published with PubMed. The lastest paper is: 'WT1 expression induces features of renal epithelial differentiation in mesenchymal fibroblasts.' George F. Atweh is registered with Medicare.
Publications
- p27(Kip1) and STMN1: Partners again?
- Down-regulation of stathmin expression is required for megakaryocyte maturation and platelet production.
- Hydroxyurea in sickle cell Disease: What will it take to change practice?
- Differences in response to fetal hemoglobin induction therapy in beta-thalassemia and sickle cell Disease.
- Epigenetic analysis of the human alpha- and beta-globin gene clusters.
- Role of epigenetic modifications in normal globin gene regulation and butyrate-mediated induction of fetal hemoglobin.
- Synergistic antiangiogenic effects of stathmin inhibition and taxol exposure.
- Sickle cell Disease: old discoveries, new concepts, and future promise.
- Therapeutic interactions between stathmin inhibition and chemotherapeutic agents in proState cancer.
- Prevention and management of strokes in patients with sickle cell Disease.
- Induction of fetal hemoglobin in the treatment of sickle cell Disease.
- Stathmin prevents the transition from a normal to an endomitotic Cell cycle during megakaryocytic differentiation.
- Targeting stathmin in proState cancer.
- Butyrate increases the efficiency of translation of gamma-globin mRNA.
- Pharmacological induction of fetal hemoglobin: Why haven't we been more successful in Thalassemia?
- The role of stathmin in the regulation of the Cell cycle.
- Role of stathmin in the regulation of the mitotic spindle: potential applications in cancer therapy.
- Pharmacological induction of fetal hemoglobin in sickle cell Disease and beta-thalassemia.
- Stathmin inhibition enhances okadaic acid-induced mitotic arrest: a potential role for stathmin in mitotic exit.
- In vivo silencing of the human gamma-globin gene in murine erythroid Cells following retroviral transduction.
- Development of ribozymes that target stathmin, a major regulator of the mitotic spindle.
- Pharmacologic induction of fetal hemoglobin: raising the therapeutic bar in sickle cell Disease.
- Effects of stathmin inhibition on the mitotic spindle.
- Taxol and anti-stathmin therapy: a synergistic combination that targets the mitotic spindle.
- Resolving conflicts: misconceptions and myths in the care of the patient with sickle cell Disease.
- Stathmin expression in immortalized and oncogene transformed Cells.
- Sustained induction of fetal hemoglobin by pulse butyrate therapy in sickle cell Disease.
- WT1 expression induces features of renal epithelial differentiation in mesenchymal fibroblasts.
- Congenital erythropoietic porphyria: prolonged high-level expression and correction of the heme biosynthetic defect by retroviral-mediated gene transfer into porphyric...
- Role for protein phosphatases in the Cell-cycle-regulated phosphorylation of stathmin.
- Cellular and molecular effects of a pulse butyrate regimen and new inducers of globin gene expression and hematopoiesis.
- Antisense RNA inhibition of phosphoprotein p18 expression abrogates the transformed phenotype of leukemic Cells.
- Production of genetically stable high-titer retroviral vectors that carry a human gamma-globin gene under the control of the alpha-globin locus control region.
- CACCC and GATA-1 sequences make the constitutively expressed alpha-globin gene erythroid-responsive in mouse erythroLeukemia cells.
- Transcriptional regulation of phosphoprotein p18 during monocytic differentiation of U937 leukemic Cells.
- Expression of the Leukemia-associated gene, p18, in normal and malignant tissues; inactivation of expression in a patient with cleaved B cell lymphoma/Leukemia.
- The major regulatory element upstream of the alpha-globin gene has classical and inducible enhancer activity.
- Regulation of phosphoprotein p18 in leukemic Cells. Cell cycle regulated phosphorylation by p34cdc2 kinase.
- The tumor suppressor gene WT1 inhibits ras-mediated transformation.
- Increased expression of the G gamma and A gamma globin genes associated with a mutation in the A gamma enhancer.
- Tumoral calcinosis.
- Intracranial solitary extraskeletal plasmacytoma resembling meningioma.
- Clinical and molecular correlations in the sickle/beta+-thalassemia Syndrome.
- New amber mutation in a beta-thalassemic gene with nonmeasurable levels of mutant messenger RNA in vivo.
- Identification of a beta-Thalassemia mutation associated with a novel haplotype of RFLPs.
- Beta-Thalassemia resulting from a single nucleotide substitution in an acceptor splice site.
- A novel basis for delta beta-Thalassemia in a Chinese family.
- A silencer element from the alpha-globin gene inhibits expression of beta-like genes.
- Detecting small mutations in expressed genes by a combination of S1 nuclease and RNase A.
- The beta-globin gene on the Chinese delta beta-Thalassemia chromosome carries a promoter mutation.
- Abnormal processing of beta-Malay globin RNA.
- Molecular cloning of a novel human Leukemia-associated gene. Evidence of conservation in animal species.
- A new mutation in IVS-1 of the human beta globin gene causing beta Thalassemia due to abnormal splicing.
- A gene that encodes for a Leukemia-associated phosphoprotein (p18) maps to chromosome bands 1p35-36.1.
- Regulated expression of p18, a major phosphoprotein of leukemic Cells.
- A novel regulatory element of the human alpha-globin gene responsible for its constitutive expression.
- Spontaneous delta- to beta-globin switching in K562 human Leukemia cells.
- Coronary care experience in a university hospital.
- p27(Kip1) and stathmin share the stage for the first time.
- DNA hypomethylation therapy for hemoglobin Disorders: molecular mechanisms and clinical applications.
- Histone deacetylase inhibitors and hemoglobin F induction in beta-Thalassemia.
- Stathmin expression and megakaryocyte differentiation: a potential role in polyploidy.
- Pharmacologic induction of fetal hemoglobin production.
- Biomarkers for early detection of Sickle nephropathy.
Schools
Duke University
American University Med Center
Conditions Treated
- Acute Leukemia
- Anemia
- Antiphospholipid Syndrome (APS)
- Central Nervous System Lymphoma
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Doctors Specialties
- Hematology
Accepted Insurances
Awards
- Elected Member
- Top MD
Education
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Duke University Hospital -
American University of Beirut Faculty of Medicine
Drug Facts
| NPI NUMBER | 1023031929 | |
| NPPES Provider LastName | ATWEH | |
| NPPES Provider FirstName | GEORGE | |
| NPPES Provider ZIPCode | 05401 | |
| NPPES Provider State | VT | |
| Specialty Description | Hematology/Oncology | |
| Total Claim Count | 320.0 | |
| Distinct Opioid Count | 5.0 | |
| Opioid Claim Count | 194.0 | |
| Percent Opioid Claims | 60.63 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1023031929 |
| Last Name Of The Provider | ATWEH |
| First Name Of The Provider | GEORGE |
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