Assistant Professor, Pediatrics, Mayo Medical School
Pediatrics
Dr. Grace M. Arteaga, MD
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Grace Arteaga practices Pediatrics. She went to medical school at University of Illinois College of Medicine at Chicago. University of Illinois College of Medicine at Chicago is ranked 49 in research and 85 in primary care medicine Grace M. Arteaga, MD is also a published doctor. She has 25 publication published. The most recent publication is: Implementation of Patient-Centered Bedside Rounds in the Pediatric Intensive Care Unit.
Publications
- Implementation of Patient-Centered Bedside Rounds in the Pediatric Intensive Care Unit.
- Semiautomatic Intraosseous Devices in Pediatric Prehospital Care.
- Expression of slow skeletal TnI in adult mouse hearts confers metabolic protection to ischemia.
- An unusual presentation of a congenital intrahepatic arterioportal fistula in an infant with Down syndrome.
- An internal domain of beta-tropomyosin increases myofilament Ca(2+) sensitivity.
- Rituximab for successful management of probable pediatric catastrophic antiphospholipid syndrome.
- Rescue of tropomyosin-induced familial hypertrophic cardiomyopathy mice by transgenesis.
- Dilated cardiomyopathy mutant tropomyosin mice develop cardiac dysfunction with significantly decreased fractional shortening and myofilament calcium sensitivity.
- Heart failure, ischemia/reperfusion injury and cardiac troponin.
- Specific enhancement of sarcomeric response to Ca2+ protects murine myocardium against ischemia-reperfusion dysfunction.
- Expression of slow skeletal troponin I in hearts of phospholamban knockout mice alters the relaxant effect of beta-adrenergic stimulation.
- Striated muscle tropomyosin isoforms differentially regulate cardiac performance and myofilament calcium sensitivity.
- Functional effects of a tropomyosin mutation linked to FHC contribute to maladaptation during acidosis.
- The effect of myosin regulatory light chain phosphorylation on the frequency-dependent regulation of cardiac function.
- Use of 2-D DIGE analysis reveals altered phosphorylation in a tropomyosin mutant (Glu54Lys) linked to dilated cardiomyopathy.
- Molecular actions of drugs that sensitize cardiac myofilaments to Ca2+.
- Altered signaling surrounding the C-lobe of cardiac troponin C in myofilaments containing an alpha-tropomyosin mutation linked to familial hypertrophic cardiomyopathy.
- Physiological significance of troponin T binding domains in striated muscle tropomyosin.
- Tropomyosin 3 expression leads to hypercontractility and attenuates myofilament length-dependent Ca(2+) activation.
- Attenuation of length dependence of calcium activation in myofilaments of transgenic mouse hearts expressing slow skeletal troponin I.
- Expression of slow skeletal troponin I in adult transgenic mouse heart muscle reduces the force decline observed during acidic conditions.
- Non-catheter related internal jugular vein thrombosis in a patient with severe haemophilia A.
- A familial hypertrophic cardiomyopathy alpha-tropomyosin mutation causes severe cardiac hypertrophy and death in mice.
- Increased cardiomyocyte intracellular calcium during endotoxin-induced cardiac dysfunction in guinea pigs.
Schools
Universidad Autonoma De Coahuila Torreon Escuela De Medicina De Torreon
Institute Mex Seguro Social
University Illinois
University Illinois Chicago Med Center
Dallas Southwest Med Center
Doctors Specialties
- Pediatric Critical Care Medicine
- Pediatrics
Accepted Insurances
Education
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University of Texas Southwestern Medical School -
University of Illinois College of Medicine at Chicago
Hospital
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Mayo Clinic - Rochester, Minnesota
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