Associate Professor, Critical Care Medicine, University of Pittsburgh School of Medicine

Pediatrics

Dr. Hulya Bayir, MD

Dr. Hulya Bayir works in the field of Pediatrics. Hulya Bayir has written publications. She has 30 publications published with PubMed. The lastest paper is: 'Therapies targeting lipid peroxidation in traumatic brain injury.'

Publications

Mitochondrial Redox Opto-Lipidomics Reveals Mono-Oxygenated Cardiolipins as Pro-Apoptotic Death Signals.
Probenecid and N-acetylcysteine Prevent Loss of Intracellular Glutathione and Inhibit Neuronal Death after Mechanical Stretch Injury In Vitro.
Therapies targeting lipid peroxidation in traumatic brain injury.
Emerging therapies in traumatic brain injury.
Cardiolipin Signaling Mechanisms: Collapse of Asymmetry and Oxidation.
Design and Synthesis of a Mitochondria-Targeted Mimic of Glutathione Peroxidase, MitoEbselen-2, as a Radiation Mitigator.
Deciphering of mitochondrial cardiolipin oxidative signaling in cerebral ischemia-reperfusion.
A mitochondrial pathway for biosynthesis of lipid mediators.
Hemorrhagic shock shifts the serum cytokine profile from pro-to anti-inflammatory after experimental traumatic brain injury in mice.
Authors' Response to Letter to the Editor, "Effect of a single dose of propofol and lack of dextrose administration in a child with mitochondrial disease - A Case Report"
Designing Inhibitors of Cytochrome c/Cardiolipin Peroxidase Complexes: Mitochondria-Targeted Imidazole-Substituted Fatty Acids.
Improved spatial resolution of matrix-assisted laser desorption/ionization imaging of lipids in the brain by alkylated derivatives of 2,5-dihydroxybenzoic acid.
Characterization of cardiolipins and their oxidation products by LC-MS analysis.
LC3 binds externalized cardiolipin on injured mitochondria to signal mitophagy in neurons: Implications for Parkinson disease.
Brain tissue oxygen monitoring identifies cortical hypoxia and thalamic hyperoxia after experimental pediatric cardiac arrest.
Cardiolipin asymmetry, oxidation and signaling.
Cardiolipin externalization to the outer mitochondrial membrane acts as an elimination signal for mitophagy in neuronal cells.
Polynitroxylated-pegylated hemoglobin attenuates fluid requirements and brain edema in combined traumatic brain injury plus hemorrhagic shock in mice.
Effect of a Single Dose of Propofol and Lack of Dextrose Administration in a Child With Mitochondrial Disease: A Case Report.
Screening of Biochemical and Molecular Mechanisms of Secondary Injury and Repair in the Brain after Experimental Blast-Induced Traumatic Brain Injury in Rats.
SEVERE BRIEF PRESSURE CONTROLLED HEMORRHAGIC SHOCK AFTER TRAUMATIC BRAIN INJURY EXACERBATES FUNCTIONAL DEFICITS AND LONG-TERM NEUROPATHOLOGICAL DAMAGE IN MICE.
Therapeutic hypothermia preserves antioxidant defenses after severe traumatic brain injury in infants and children.
Protein nitration and poly-ADP-ribosylation in brain after rapid exsanguination cardiac arrest in a rat model of emergency preservation and resuscitation.
Unmasking Sex-Based Disparity in Neuronal Metabolism.
CSF Bcl-2 and cytochrome C temporal profiles in outcome prediction for adults with severe TBI.
Magnetic resonance imaging assessment of macrophage accumulation in mouse brain after experimental traumatic brain injury.
Mitochondria-targeted disruptors and inhibitors of cytochrome c/cardiolipin peroxidase complexes: a new strategy in anti-apoptotic drug discovery.
The hierarchy of structural transitions induced in cytochrome c by anionic phospholipids determines its peroxidase activation and selective peroxidation during apoptos...
Assessment of antioxidant reserves and oxidative stress in cerebrospinal fluid after severe traumatic brain injury in infants and children.

Doctors Specialties

Pediatric Critical Care Medicine
Pediatrics

Accepted Insurances

Education

University of Pittsburgh Medical Center
SUNY Stony Brook University Hospital & Medical Center

Hospital

UPMC Presbyterian Shadyside

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