Physician
Pediatric Hematology & Oncology
Dr. Ilonna J. Rimm, MD
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Ilonna Rimm practices Pediatric Hematology & Oncology. She researched medicine at Harvard Medical School. Harvard Medical School is one of the top medical schools in the US. Harvard Medical School is ranked 1 in research and 12 in primary care medicine Ilonna J. Rimm, MD is a published doctor. She published 36 publications, including: 'Tacrolimus (FK506) increases neuronal expression of GAP-43 and improves functional recovery after spinal cord injury in rats.'
Publications
- Oligomerization of p53 is necessary to inhibit its transcriptional transactivation property at high protein concentration.
- Impact of V beta 8+/+ T cells on the development of increased airway reactivity and IgE production in SJL mice.
- V(beta)8(+) T cells protect from demyelinating disease in a viral model of multiple sclerosis.
- In the absence of T cells, natural killer cells protect from mortality due to HSV-1 encephalitis.
- B cell-deficient mice have increased susceptibility to HSV-1 encephalomyelitis and mortality.
- Tacrolimus (FK506) increases neuronal expression of GAP-43 and improves functional recovery after spinal cord injury in rats.
- Recombinant CD40L treatment protects allogeneic murine bone marrow transplant recipients from death caused by herpes simplex virus-1 infection.
- Detection of rare apoptotic T cells in vivo.
- A role for transforming growth factor-beta1 in the increased pneumonitis in murine allogeneic bone marrow transplant recipients with graft-versus-host disease after pu...
- Bone marrow transplantation for therapy-related myelodysplasia: comparison with primary myelodysplasia.
- High-dose cyclophosphamide, carmustine, and etoposide with autologous transplantation in Hodgkin's disease: a prognostic model for treatment outcomes.
- Experimental autoimmune myasthenia gravis in B10.BV8S2 transgenic mice: preferential usage of TCRAV1 gene by lymphocytes responding to acetylcholine receptor.
- Suppression of herpes simplex virus type 1 (HSV-1)-induced pneumonia in mice by inhibition of inducible nitric oxide synthase (iNOS, NOS2).
- TCR-beta transgenic mice fail to mediate a GVHR due to defects of allorecognition and subsequent IL-2 generation.
- Restriction of the TCR repertoire inhibits the development of memory T cells and prevents autoimmunity in lpr mice.
- V beta 8.2 transgene expression interferes with development of experimental autoimmune thyroiditis in CBA k/q but not k/k mice.
- Plasticity of the T cell receptor repertoire in TCR beta-chain transgenic mice.
- Insights into the mechanism of allograft acceptance in T-cell receptor (beta-chain) transgenic mice.
- Superantigen-induced peripheral tolerance inhibits T cell responses to immunogenic peptides in TCR (beta-chain) transgenic mice.
- Recombinant human interleukin-1 receptor antagonist in the treatment of steroid-resistant graft-versus-host disease.
- Differential expression of activation markers during tolerance induction by superantigens in T-cell receptor (beta-chain) transgenic mice.
- T cell receptor (beta chain) transgenic mice have selective deficits in gamma delta T cell subpopulations.
- Differentiation of V beta 8.2+ CD4+ T cells induced by a superantigen: roles of antigen-presenting cells and cytokines.
- Effect of constrained T-cell receptor repertoires on collagen-induced arthritis in T-cell receptor V beta transgenic and congenic mice.
- Expression of an ovalbumin-specific V beta 8.2 TCR transgene inhibits collagen arthritis in B10.Q mice.
- Antibody-dependent cellular cytotoxicity and natural-killer-like activity are mediated by subsets of activated T cells.
- Natural killer-like activity mediated by activated T lymphocytes.
- Ontogeny of human hematopoietic cells: analysis utilizing monoclonal antibodies.
- Characterization of human thymic epithelial cells grown in serum-free medium.
- Brain tumors after cranial irradiation for childhood acute lymphoblastic leukemia. A 13-year experience from the Dana-Farber Cancer Institute and the Children's Hospital.
- Allelic exclusion and lymphocyte development. Lessons from transgenic mice.
- Bone marrow transplantation for the Wiskott-Aldrich syndrome. Long-term follow-up.
- Allorecognition in T-cell receptor (beta-chain) transgenic mice.
- Immunodominance is altered in T cell receptor (beta-chain) transgenic mice without the generation of a hole in the repertoire.
- Alloreactive lymphocytes from T cell receptor (beta-chain) transgenic mice do not mediate a graft-versus-host reaction.
Schools
Harvard Medical School
Dana Farber Cancer Institute
Doctors Specialties
- Pediatric Hematology
- Oncology
Accepted Insurances
Education
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Boston Children's Hospital -
Harvard Medical School
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