Professor of Medicine, Chief, Center Director, Vice Chair
Cardiology
Dr. Ivor J. Benjamin, MD
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Dr. Ivor J. Benjamin is an expert in the field of Cardiology. He attended Johns Hopkins University School of Medicine. Johns Hopkins University School of Medicine is a top medical university. Johns Hopkins University School of Medicine is ranked 3 in research and 29 in primary care medicine He over the years received 2 awards: "Elected Member" and "Fellow (FACC)". Ivor J. Benjamin is a published physician as well. He has 79 publications published. The latest was: Stress (heat shock) proteins: molecular chaperones in cardiovascular biology and disease.
Publications
- American Heart Association Cardiovascular Genome-Phenome Study: foundational basis and program.
- A near-infrared fluorescent voltage-sensitive dye allows for moderate-throughput analyses of human induced pluripotent stem cell-derived cardiomyocytes.
- Aggregate-prone R120GCRYAB triggers multifaceted modifications of the Thioredoxin System.
- The NADPH metabolic network regulates human αB-crystallin Cardiomyopathy and reductive stress in Drosophila melanogaster.
- HSPB2 Is Dispensable for the Cardiac Hypertrophic Response but Reduces Mitochondrial Energetics following Pressure Overload In Mice.
- The heat shock transcription factor Hsf1 is downregulated in DNA damage-associated senescence, contributing to the maintenance of senescence phenotype.
- Small Heat Shock Proteins in Redox Metabolism: Implications for Cardiovascular Diseases.
- Targeting endoglin, an auxiliary transforming growth factor β coreceptor, to prevent fibrosis and Heart failure.
- Heat Shock Transcription Factor Hsf1 is Involved in Tumor Progression via Regulation of HIF -1 and RNA-binding Protein HuR.
- Glutathione-dependent reductive stress triggers mitochondrial oxidation and cytotoxicity.
- HSF1-Dependent Upregulation of Hsp70 by Sulfhydryl-Reactive Inducers of the KEAP1/NRF2/ARE Pathway.
- Proteostasis and Redox state in the Heart.
- Sustained activation of nuclear erythroid 2-related factor 2/antioxidant response element signaling promotes reductive stress in the human mutant protein aggregation c...
- Involvement of reductive stress in the Cardiomyopathy in transgenic mice with cardiac-specific overexpression of heat shock protein 27.
- Febrile-range temperature modifies cytokine gene expression in LPS-stimulated macrophages by differentially modifying NF-{kappa}B recruitment to cytokine gene promoters.
- Global expression profiling identifies a novel biosignature for protein aggregation R120GCryAB Cardiomyopathy in mice.
- Heat shock co-activates interleukin-8 transcription.
- Autophagy is an adaptive response in desmin-related Cardiomyopathy.
- Differential display of DNA-binding proteins reveals heat-shock factor 1 as a circadian transcription factor.
- Unmasking different mechanical and energetic roles for the small heat shock proteins CryAB and HSPB2 using genetically modified mouse Hearts.
- CRYAB and HSPB2 deficiency alters cardiac metabolism and paradoxically confers protection against Myocardial ischemia in aging mice.
- Human alpha B-crystallin mutation causes oxido-reductive stress and protein aggregation Cardiomyopathy in mice.
- Viewing a stressful episode of ER: is ATF6 the triage nurse?
- RANK ligand expression in heat shock factor-2 deficient mouse bone marrow stromal/preosteoblast cells.
- Induction of KLF4 in response to heat stress.
- Heat shock factor 1-deficient mice exhibit decreased recovery of hearing following noise overstimulation.
- Heat shock protein-70 mediates the cytoprotective effect of carbon monoxide: involvement of p38 beta MAPK and heat shock factor-1.
- Mouse HSF1 disruption perturbs redox state and increases mitochondrial oxidative stress in Kidney.
- Learning from failure: Congestive heart failure in the postgenomic age.
- The stress or heat shock (HS) response: insights from transgenic mouse models.
- A murine world without HSFs: meeting report.
- Use of Hsf1(-/-) mice reveals an essential role for HSF1 to protect lung against cadmium-induced injury.
- Localized recruitment of a chromatin-remodeling activity by an activator in vivo drives transcriptional elongation.
- Heat shock transcription factor 2 is not essential for embryonic development, fertility, or adult cognitive and psychomotor function in mice.
- Exercise, estrogen, and ischemic cardioprotection by heat shock protein 70.
- Heat shock factor 1 and heat shock proteins: critical partners in protection against Acute cell injury.
- Effects of redox state on the efficient uptake of cell permeable Peptide in Mammalian cells.
- Administration of a World Wide Web site during a period of rapid growth: the OncoLink experience.
- beta Adrenergic receptor desensitisation may serve a cardioprotective role.
- Endometrial cancer surgery in Arizona: a statewide analysis of access to care.
- An economic analysis of dose dense weekly paclitaxel plus carboplatin versus every-3-week paclitaxel plus carboplatin in the treatment of advanced ovarian cancer.
- Diabetes and cardiovascular Disease: a statement for healthcare professionals from the American Heart Association.
- Isoproterenol-induced Myocardial fibrosis in relation to myocyte necrosis.
- Synchronization of circadian Per2 rhythms and HSF1-BMAL1:CLOCK interaction in mouse fibroblasts after short-term heat shock pulse.
- Immuno and functional characterization of CFTR in submandibular and pancreatic acinar and duct cells.
- Matrix metalloproteinases: from biology to therapeutic strategies in cardiovascular Disease.
- Bimoclomol, a heat shock protein co-inducer, acts by the prolonged activation of heat shock factor-1.
- Stress proteins and cardiovascular Disease.
- Induction of a heat shock factor 1-dependent stress response alters the cytotoxic activity of hsp90-binding agents.
- Cardiac hypertrophy in idiopathic dilated congestive Cardiomyopathy: a clinicopathologic study.
- Activation of the heat shock transcription factor by hypoxia in mammalian cells.
- Heat shock factor 1-mediated thermotolerance prevents cell death and results in G2/M cell cycle arrest.
- Maternal effect of Hsf1 on reproductive success.
- Continuous contractile activity induces fiber type specific expression of HSP70 in skeletal muscle.
- Differential expression of B-crystallin and Hsp27 in skeletal muscle during continuous contractile activity. Relationship to myogenic regulatory factors.
- [Effects of hsf1 genotype on the constitutive expression of alphaB-crystallin in mice myocardium].
- Protective responses in the ischemic myocardium.
- Stress-response proteins in cardiovascular Disease.
- Cardioprotective effects of 70-kDa heat shock protein in transgenic mice.
- Human heat shock protein 70 (hsp70) protects murine cells from injury during metabolic stress.
- Induction of stress proteins in cultured myogenic cells. Molecular signals for the activation of heat shock transcription factor during ischemia.
- Disruption of heat shock factor 1 reveals an essential role in the ubiquitin proteolytic pathway.
- Febrile-range hyperthermia augments neutrophil accumulation and enhances lung injury in experimental gram-negative bacterial pneumonia.
- Temporospatial expression of the small HSP/alpha B-crystallin in cardiac and skeletal muscle during mouse development.
- Heat shock response: lessons from mouse knockouts.
- Targeted disruption of heat shock transcription factor 1 abolishes thermotolerance and protection against heat-inducible apoptosis.
- HSF1 is required for extra-embryonic development, postnatal growth and protection during inflammatory responses in mice.
- Stress (heat shock) proteins: molecular chaperones in cardiovascular biology and Disease.
- Mouse heat shock transcription factor 1 deficiency alters cardiac redox homeostasis and increases mitochondrial oxidative damage.
- Stress pathways in the rat cochlea and potential for protection from acquired deafness.
- Heat shock factor 1-independent activation of dendritic cells by heat shock: implication for the uncoupling of heat-mediated immunoregulation from the heat shock respo...
- Small heat shock proteins: a new classification scheme in mammals.
- Heat shock proteins in mammalian development.
- Discovering the full spectrum of cardiovascular Disease: Minority Health Summit 2003: report of the Basic Science Writing Group.
- Reductive stress on life span extension in C. elegans.
- CRYAB and HSPB2 deficiency increases myocyte mitochondrial permeability transition and mitochondrial calcium uptake.
- Induction of heat shock proteins by heregulin beta1 leads to protection from apoptosis and anchorage-independent growth.
- Selective degradation of aggregate-prone CryAB mutants by HSPB1 is mediated by ubiquitin-proteasome pathways.
Schools
Johns Hopkins University School Of Medicine
Yale New Haven HospYale University
Yale New Haven Hospital
Duke University
Michael Reese Hospital and Med Center
Procedures Preformed
- Cardiac Catheterization (incl. Coronary Angiography)
- Cardiac Imaging
Conditions Treated
- Angina and Acute Coronary Syndrome
- Aortic Valve Disease
- Arrhythmias (incl. Atrial Fibrillation)
- Cardiomyopathy
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Doctors Specialties
- Cardiology
Accepted Insurances
Awards
- Elected Member
- Fellow (FACC)
Education
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Yale New Haven Children's Center -
Yale-New Haven Hospital -
Johns Hopkins University School of Medicine
Hospital
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University of Utah Health Care - Hospital and Clinics
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1851481485 |
| Last Name Of The Provider | BENJAMIN |
| First Name Of The Provider | IVOR |
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