Physician
Nephrology
Dr. Jeremy S. Duffield, MD
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Jeremy Duffield is an expert in the field of Nephrology. Jeremy S. Duffield is a published MD. He has 59 papers published. The most current publication "CYR61 (CCN1) OVEREXPRESSION INDUCES LUNG INJURY IN MICE."
Publications
- Dicer1 activity in the stromal compartment regulates nephron differentiation and vascular patterning during mammalian Kidney organogenesis.
- CYR61 (CCN1) OVEREXPRESSION INDUCES LUNG INJURY IN MICE.
- Deficient Autophagy Results in Mitochondrial Dysfunction and FSGS.
- Cells of renin lineage take on a podocyte phenotype in aging nephropathy.
- Translational Profiles of Medullary Myofibroblasts during Kidney Fibrosis.
- Chronic Ifosfamide Toxicity: Kidney Pathology and Pathophysiology.
- Myofibroblasts in Fibrotic Kidneys.
- Cysteamine Modulates Oxidative Stress and Blocks Myofibroblast Activity in CKD.
- Changes in macrophage phenotype as the immune response evolves.
- Cells of Renin Lineage Are Progenitors of Podocytes and Parietal Epithelial Cells in Experimental Glomerular Disease.
- Coronary adventitial cells are linked to perivascular cardiac fibrosis via TGFβ1 signaling in the mdx mouse model of Duchenne muscular dystrophy.
- Role of Lung Pericytes and Resident Fibroblasts in the Pathogenesis of Pulmonary Fibrosis.
- Resident Renal Mononuclear Phagocytes Comprise Five Discrete Populations with Distinct Phenotypes and Functions.
- Resident mesenchymal cells and fibrosis.
- Pericytes in Kidney fibrosis.
- EphrinB2 Reverse Signaling Protects against Capillary Rarefaction and Fibrosis after Kidney Injury.
- Cellular Mechanisms of Tissue Fibrosis. 3. Novel mechanisms of Kidney fibrosis.
- LPA1-induced cytoskeleton reorganization drives fibrosis through CTGF-dependent fibroblast proliferation.
- LRP-6 is a coreceptor for multiple fibrogenic signaling pathways in pericytes and myofibroblasts that are inhibited by DKK-1.
- Activation of pericytes: recent insights into Kidney fibrosis and microvascular rarefaction.
- Wnt signalling in Kidney diseases: dual roles in renal injury and repair.
- Host responses in tissue repair and fibrosis.
- Anti-proteinase 3 anti-neutrophil cytoplasm autoantibodies recapitulate systemic Vasculitis in mice with a humanized immune system.
- Elastin accumulation is regulated at the level of degradation by macrophage metalloelastase (MMP-12) during experimental liver fibrosis.
- Dendritic cells take on more tasks in the liver?
- Metchnikoff's policemen: macrophages in development, homeostasis and regeneration.
- Macrophages are essential contributors to kidney injury in murine cryoglobulinemic membranoproliferative Glomerulonephritis.
- Platelet-derived growth factor receptor signaling activates pericyte-myofibroblast transition in obstructive and post-ischemic Kidney fibrosis.
- Frontiers in Research: Chronic Kidney Diseases: The pivotal role of pericytes in Kidney fibrosis.
- TNF-{alpha}-dependent Regulation of Acute Pancreatitis Severity by Ly-6Chi Monocytes in Mice.
- Origin of new cells in the adult Kidney: results from genetic labeling techniques.
- The origin of interstitial myofibroblasts in chronic Kidney disease.
- Targeting endothelium-pericyte cross talk by inhibiting VEGF receptor signaling attenuates Kidney microvascular rarefaction and fibrosis.
- Macrophages in Kidney repair and regeneration.
- The melanoma-associated transmembrane glycoprotein Gpnmb controls trafficking of cellular debris for degradation and is essential for tissue repair.
- Epithelial to mesenchymal transition in injury of solid organs: fact or artifact?
- Resolved: EMT produces fibroblasts in the Kidney.
- PRM-151 (recombinant human serum amyloid P/pentraxin 2) for the treatment of fibrosis.
- Mobilized human hematopoietic stem/progenitor cells promote Kidney repair after ischemia/reperfusion injury.
- Macrophages and immunologic inflammation of the Kidney.
- Macrophage Wnt7b is critical for Kidney repair and regeneration.
- Fate tracing reveals the pericyte and not epithelial origin of myofibroblasts in Kidney fibrosis.
- Bone marrow Ly6Chigh monocytes are selectively recruited to injured Kidney and differentiate into functionally distinct populations.
- Serum amyloid P inhibits fibrosis through Fc gamma R-dependent monocyte-macrophage regulation in vivo.
- CD11b+ myeloid cells are the key mediators of Th2 cell homing into the airway in allergic inflammation.
- Pericytes and perivascular fibroblasts are the primary source of collagen-producing cells in obstructive fibrosis of the Kidney.
- Kidney injury molecule-1 is a phosphatidylserine receptor that confers a phagocytic phenotype on epithelial cells.
- Intrinsic epithelial cells repair the Kidney after injury.
- Scar-associated macrophages are a major source of hepatic matrix metalloproteinase-13 and facilitate the resolution of murine hepatic fibrosis.
- Resolvin D series and protectin D1 mitigate Acute kidney injury.
- cPLA2-interacting protein, PLIP, causes apoptosis and decreases G1 phase in mesangial cells.
- Kidney tubular epithelium is restored without replacement with bone marrow-derived cells during repair after ischemic injury.
- Conditional ablation of macrophages halts progression of crescentic Glomerulonephritis.
- Mutations in complement factor I predispose to development of atypical hemolytic uremic Syndrome.
- Restoration of tubular epithelial cells during repair of the postischemic Kidney occurs independently of bone marrow-derived stem cells.
- Quantitative morphometry of lupus nephritis: the significance of collagen, tubular space, and inflammatory infiltrate.
- Selective depletion of macrophages reveals distinct, opposing roles during liver injury and repair.
- TLR-2/TLR-4 TREM-1 Signaling Pathway Is Dispensable in Inflammatory Myeloid Cells during Sterile Kidney Injury.
Schools
University Of Oxford Medical School Office
Procedures Preformed
- Hemodialysis
- Peritoneal Dialysis
Conditions Treated
- Acute Glomerulonephritis
- Acute Kidney Failure
- Calcium Metabolism Disorders
- Chronic Kidney Diseases
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Doctors Specialties
- Nephrology
Accepted Insurances
Education
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Brigham and Women's Hospital
Hospital
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Massachusetts General Hospital -
Brigham and Women's Hospital
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