Professor, Neurology, Uniformed Services Hebert SOM
Neurology
Dr. John M. Hallenbeck, MD
John Hallenbeck works in the field of Neurology. He attended Perelman School of Medicine at the University of Pennsylvania. John Hallenbeck is a published MD. He published 33 publications, including: 'A novel quantitative high-throughput screen identifies drugs that both activate SUMO conjugation via the inhibition of microRNAs 182 and 183 and facilitate neuroprotec...' He accepts all Medicare patients.
Publications
- A novel quantitative high-throughput screen identifies drugs that both activate SUMO conjugation via the inhibition of microRNAs 182 and 183 and facilitate neuroprotec...
- Anti-lymphoproliferative activity of alpha-2-macroglobulin in the plasma of hibernating 13-lined ground squirrels and woodchucks.
- Poly(ADP-ribose) polymerase-1 and its cleavage products differentially modulate cellular protection through NF-kB-dependent signaling.
- SUMO and Ischemic Tolerance.
- A new role for downstream Toll-like receptor signaling in mediating immediate early gene expression during focal cerebral ischemia.
- A method for hypothermia-induction and maintenance allows precise body and brain temperature control in mice.
- Immunologic privilege in the central nervous system and the blood-brain barrier.
- Global Protein Conjugation by Ubiquitin-Like-Modifiers during Ischemic Stress Is Regulated by MicroRNAs and Confers Robust Tolerance to Ischemia.
- Downstream Toll-like receptor signaling mediates adaptor-specific cytokine expression following focal cerebral ischemia.
- Tracks of a Non-Main Path Traveler: 2011 Thomas Willis Lecture.
- Elevated Global SUMOylation in Ubc9 Transgenic Mice Protects Their Brains against Focal Cerebral Ischemic Damage.
- Intranasal Delivery of E-Selectin Reduces Atherosclerosis in ApoE-/- Mice.
- Increased plasma and tissue MMP levels are associated with BCSFB and BBB disruption evident on post-contrast FLAIR after experimental stroke.
- Blood-brain barrier disruption in humans is independently associated with increased matrix metalloproteinase-9.
- Feridex preloading permits tracking of CNS-resident macrophages after transient middle cerebral artery occlusion.
- Mucosal tolerance to E-selectin promotes the survival of newly generated neuroblasts via regulatory T-cell induction after stroke in spontaneously hypertensive rats.
- Reperfusion-associated hemorrhagic transformation in SHR rats: evidence of symptomatic parenchymal hematoma.
- Cardiovascular, rheumatologic, and pharmacologic predictors of stroke in patients with rheumatoid arthritis: a nested, case-control study.
- Mucosal tolerization to E-selectin protects against memory dysfunction and white matter damage in a vascular cognitive impairment model.
- Spatial and temporal activation of brain regions in hibernation: c-fos expression during the hibernation bout in thirteen-lined ground squirrel.
- Protein SUMOylation is massively increased in hibernation torpor and is critical for the cytoprotection provided by ischemic preconditioning and hypothermia in SHSY5Y ...
- Involvement of Akt in preconditioning-induced tolerance to ischemia in PC12 cells.
- Development of an ischemic tolerance model in a PC12 cell line.
- Determining the fetal inflammatory response in an experimental model of intrauterine inflammation in rats.
- Glucosylceramide synthase activity and ceramide levels are modulated during cerebral ischemia after ischemic preconditioning.
- Mucosal tolerance to E-selectin provides cell-mediated protection against ischemic brain injury.
- Overexpression of monocyte chemoattractant protein 1 in the brain exacerbates ischemic brain injury and is associated with recruitment of inflammatory cells.
- The many faces of tumor necrosis factor in stroke.
- Effects of intrauterine inflammation on developing rat brain.
- TNF-alpha-induced tolerance to ischemic injury involves differential control of NF-kappaB transactivation: the role of NF-kappaB association with p300 adaptor.
- Global SUMOylation is a molecular mechanism underlying hypothermia-induced ischemic tolerance.
- How implementation of systems biology into clinical trials accelerates understanding of diseases.
Schools
Perelman School of Medicine University of Pennsylvania
Doctors Specialties
- Neurology
Accepted Insurances
Education
-
University of Michigan Hospitals and Health Centers -
Perelman School of Medicine at the University of Pennsylvania
Drug Facts
| NPI NUMBER | 1033152277 | |
| NPPES Provider LastName | HALLENBECK | |
| NPPES Provider FirstName | JOHN | |
| NPPES Provider ZIPCode | 070552840 | |
| NPPES Provider State | NJ | |
| Specialty Description | Emergency Medicine | |
| Total Claim Count | 73.0 | |
| Distinct Opioid Count | 1.0 | |
| Opioid Claim Count | 12.0 | |
| Percent Opioid Claims | 16.44 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1033152277 |
| Last Name Of The Provider | HALLENBECK |
| First Name Of The Provider | JOHN |
| View All |
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