Professor, Neurosciences, UCSD School of Medicine
Neurology
Dr. John M. Ravits, MD
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Doctor John M. Ravits, MD practices Neurology. He graduated from Mayo Medical School. Mayo Medical School has a rank of 27 in research and a 42 in primary care He received 2 awards: "Top MD" and "Super Doctor". Doctor John M. Ravits, MD has written publications. He has 47 publications published with PubMed. The lastest publication is: 'Recent advances in the treatment of neuropathies.' John Ravits accepts Medicare payments and is registered with Medicare.gov.
Publications
- Targeted axonal import (TAxI) peptide delivers functional proteins into spinal cord motor neurons after peripheral administration.
- Dysregulated miRNA biogenesis downstream of cellular stress and ALS-causing mutations: a new mechanism for ALS.
- Neuropathology of Amyotrophic Lateral Sclerosis and Its Variants.
- Exome sequencing in amyotrophic lateral sclerosis identifies risk genes and pathways.
- An α2-Na/K ATPase/α-adducin complex in astrocytes triggers non-cell autonomous neurodegeneration.
- Amyotrophic lateral sclerosis onset is influenced by the burden of rare variants in known amyotrophic lateral sclerosis genes.
- TREM2 Variant p.R47H as a Risk Factor for Sporadic Amyotrophic Lateral Sclerosis.
- Direct conversion of patient fibroblasts demonstrates non-cell autonomous Toxicity of astrocytes to motor neurons in familial and sporadic ALS.
- Targeting RNA foci in iPSC-derived motor neurons from ALS patients with a C9ORF72 repeat expansion.
- Targeted degradation of sense and antisense C9orf72 RNA foci as therapy for ALS and frontotemporal degeneration.
- Deciphering amyotrophic lateral sclerosis: What phenotype, neuropathology and genetics are telling us about pathogenesis.
- Lack of C9ORF72 coding mutations supports a gain of function for repeat expansions in amyotrophic lateral sclerosis.
- Divergent roles of ALS-linked proteins FUS/TLS and TDP-43 intersect in processing long pre-mRNAs.
- ALS motor phenotype heterogeneity, focality, and spread: deconstructing motor neuron degeneration.
- Aberrant neuregulin 1 signaling in amyotrophic lateral sclerosis.
- TDP-43 and ubiquitinated cytoplasmic aggregates in sporadic ALS are low frequency and widely distributed in the lower motor neuron columns independent of Disease spread.
- Sporadic ALS has compartment-specific aberrant exon splicing and altered cell-matrix adhesion biology.
- No evidence of HIV pol gene in spinal cord tissues in sporadic ALS by real-time RT-PCR.
- Current and future directions in genomics of amyotrophic lateral sclerosis.
- An aggregate-inducing peripherin isoform generated through intron retention is upregulated in amyotrophic lateral sclerosis and associated with Disease pathology.
- Efficacy of minocycline in patients with amyotrophic lateral sclerosis: a phase III randomised trial.
- Implications of ALS focality: rostral-caudal distribution of lower motor neuron loss postmortem.
- Focality of upper and lower motor neuron degeneration at the clinical onset of ALS.
- Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations.
- Amyotrophic lateral sclerosis microgenomics.
- Sporadic amyotrophic lateral sclerosis: a hypothesis of persistent (non-lytic) enteroviral infection.
- Sixteen novel mutations in the Cu/Zn superoxide dismutase gene in amyotrophic lateral sclerosis: a decade of Discoveries, defects and disputes.
- Electrophysiological tests of Autonomic function in patients with idiopathic Autonomic failure syndromes.
- Myasthenic (Eaton-Lambert) Syndrome associated with pulmonary large-cell neuroendocrine carcinoma.
- Transient monocular visual loss from narrow-angle glaucoma.
- Primary writing tremor and myoclonic writer's cramp.
- Stimulus artifact compensation using biphasic stimulation.
- Myasthenia gravis. A well-understood neuroMuscular disorder.
- Pathophysiology of hemifacial spasm.
- A long-term follow-up study of patients with post-poliomyelitis neuroMuscular symptoms.
- Botulinum toxin therapy in hemifacial spasm: clinical and electrophysiologic studies.
- Clinical and electromyographic studies of postpoliomyelitis Muscular atrophy.
- A double-blind trial of isoniazid for essential tremor and other action tremors.
- Phase III randomized trial of gabapentin in patients with amyotrophic lateral sclerosis.
- Late-onset SCA2: 33 CAG repeats are sufficient to cause Disease.
- Recent advances in the treatment of neuropathies.
- AAEM minimonograph #48: Autonomic nervous system testing.
- Placebo-controlled trial of gabapentin in patients with amyotrophic lateral sclerosis. WALS Study Group. Western Amyotrophic Lateral Sclerosis Study Group.
- No morphometric Abnormality recurrent laryngeal nerve in spastic dysphonia.
- Prostatic adenocarcinoma of ductal origin.
- A4V superoxide dismutase mutation in apparently sporadic ALS resembling neuralgic amyotrophy.
Schools
Mayo Medical School
Univ Of California San Diego Medical Center
Brigham and Women39s Hospital
Harvard Medical School
National Institutes Of Health
Procedures Preformed
- EMG (Electromyography)
Conditions Treated
- Anterior Horn Disease
- Ataxia
- Autonomic Disorders
- Brachial Plexus Palsy
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Doctors Specialties
- Clinical Neurophysiology
- Neurology
Accepted Insurances
Awards
- Top MD
- Super Doctor
Education
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National Institutes of Health Clinical Center -
Brigham and Women's Hospital -
University of California San Diego -
Mayo Medical School
Hospital
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UC San Diego Health
Drug Facts
| NPI NUMBER | 1396858965 | |
| NPPES Provider LastName | RAVITS | |
| NPPES Provider FirstName | JOHN | |
| NPPES Provider ZIPCode | 920935004 | |
| NPPES Provider State | CA | |
| Specialty Description | Neurology | |
| Total Claim Count | 81.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1396858965 |
| Last Name Of The Provider | RAVITS |
| First Name Of The Provider | JOHN |
| View All |
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