Professor of Medicine, Georgetown University Medical Center
Endocrinology
Dr. Joseph Verbalis, MD
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Joseph Verbalis is an expert in the field of Endocrinology. He went to University of Pittsburgh School of Medicine. He over the years received 3 awards: "Top Doctors:Washington DC Area", "Top Doctors:Washington-Baltimore" and "Elected Member". Joseph Verbalis is also a published doctor. He has 98 paper published. The most recent paper is: Escape from vasopressin-induced antidiuresis: role of vasopressin resistance of the collecting duct. Joseph Verbalis is registered with Medicare and accepts Medicare payments.
Publications
- Diagnosing and Treating the Syndrome of Inappropriate Antidiuretic Hormone Secretion.
- Statement of the 3rd International Exercise-Associated Hyponatremia Consensus Development Conference, Carlsbad, California, 2015.
- Current treatment practice and outcomes. Report of the hyponatremia registry.
- Sodium homeostasis and bone.
- Review and analysis of differing regulatory indications and expert panel guidelines for the treatment of hyponatremia.
- Renal physiology of nocturia.
- Hyponatremia With intracranial disease: not often cerebral salt wasting.
- Diagnosis, Evaluation, and Treatment of Hyponatremia: Expert Panel Recommendations.
- Melanocortin signaling in the brainstem influences vagal outflow to the stomach.
- The Need for Salt: Does a Relationship Exist between Cystic Fibrosis and Exercise-Associated Hyponatremia?
- Age-associated abnormalities of water homeostasis.
- Challenges in assessing the impact of hyponatremia management on length of stay: Interim results from a global, multi-center, prospective, observational registry of ho...
- Chronic hyponatremia exacerbates multiple Manifestations of senescence in male rats.
- Changes in Steroid Concentrations With the Timing of ACTH Stimulation Testing in Normal Subjects.
- Changes in copeptin and bioactive vasopressin in runners With and Without hyponatremia.
- Osteoclast response to low extracellular sodium and the mechanism of hyponatremia-induced bone loss.
- Sex differences in vasopressin Vâ receptor expression and vasopressin-induced antidiuresis.
- Hyponatremia-induced Osteoporosis.
- Osmotic and nonosmotic regulation of arginine vasopressin during prolonged endurance exercise.
- Statement of the Second International Exercise-Associated Hyponatremia Consensus Development Conference, New Zealand, 2007.
- Characterization of noradrenergic transmission at the dorsal motor nucleus of the vagus involved in reflex control of fundus tone.
- Disorders of water and salt metabolism associated With pituitary disease.
- Dorsal motor nucleus of the vagus: a site for evoking simultaneous changes in crural diaphragm activity, lower esophageal sphincter pressure, and fundus tone.
- How does the brain sense osmolality?
- Hyponatremia treatment guidelines 2007: expert panel recommendations.
- Dysnatremia predicts a delayed recovery in collapsed ultramarathon runners.
- Hyponatremia in marathon runners due to inappropriate arginine vasopressin secretion.
- A reevaluation of the effects of stimulation of the dorsal motor nucleus of the vagus on gastric motility in the rat.
- Use of steroid profiles in determining the cause of Adrenal insufficiency.
- Disorders of body water homeostasis in critical illness.
- AVP receptor antagonists as aquaretics: review and assessment of clinical data.
- Comments on: "Esophageal-gastric relaxation reflex in rat: dual control of peripheral nitrergic and cholinergic transmission".
- Whole-body volume regulation and escape from antidiuresis.
- Increased Renal alpha-ENaC and NCC abundance and elevated blood pressure are independent of hyperaldosteronism in vasopressin escape.
- Control of brain volume during hypoosmolality and hyperosmolality.
- Abnormalities of water homeostasis in aging.
- Hindbrain chemical mediators of reflex-induced inhibition of gastric tone produced by esophageal distension and intravenous nicotine.
- Small interfering RNA-mediated functional silencing of vasopressin V2 receptors in the mouse kidney.
- Collecting duct-specific knockout of endothelin-1 alters vasopressin regulation of urine osmolality.
- Glucose does not activate nonadrenergic, noncholinergic inhibitory neurons in the rat stomach.
- Regulation of gene expression in magnocellular neurons in rat supraoptic nucleus during sustained hypoosmolality.
- Sodium status of collapsed marathon runners.
- Increased blood pressure, aldosterone activity, and regional differences in Renal ENaC protein during vasopressin escape.
- Downregulation of Renal vasopressin V2 receptor and aquaporin-2 expression parallels age-associated defects in urine concentration.
- Selective gene expression in magnocellular neurons in rat supraoptic nucleus.
- Inhibition of salt appetite in rats by central oxytocin.
- Rosiglitazone activates Renal sodium- and water-reabsorptive pathways and lowers blood pressure in normal rats.
- Increased renal ENaC subunit and sodium transporter abundances in streptozotocin-induced type 1 Diabetes.
- Glucose acts in the CNS to regulate gastric motility during hypoglycemia.
- Estrogen regulates Adrenal angiotensin AT1 receptors by modulating AT1 receptor translation.
- Evaluation and management of hypo-osmolality in hospitalized patients.
- Diabetes insipidus.
- Synergistic effects of nitric oxide and prostaglandins on Renal escape from vasopressin-induced antidiuresis.
- Dysregulation of Renal salt and water transport proteins in diabetic Zucker rats.
- Role of thromboxane receptors in the dipsogenic response to central angiotensin II.
- Systemic diseases associated With disorders of water homeostasis.
- Management of disorders of water metabolism in patients With pituitary tumors.
- Escape from Vasopressin-Induced Antidiuresis: Insights at the Molecular Level.
- Hyponatremia: epidemiology, pathophysiology, and therapy.
- Control of appetite and satiety: insights from biologic and behavioral studies.
- Brain buffering is restored in hyponatremic rats by correcting their plasma sodium concentration.
- Sodium deprivation blunts hypovolemia-induced pituitary secretion of vasopressin and oxytocin in rats.
- Acute effects of ethanol on ingestive behavior in rats.
- Hyponatremia inhibits sprouting of oxytocinergic axons following bilateral lesions of the paraventricular nucleus in rats.
- Rapid correction of hyponatremia produces differential effects on brain osmolyte and electrolyte reaccumulation in rats.
- Exogenous cholecystokinin activates cFos expression in medullary but not hypothalamic neurons in neonatal rats.
- Distribution and neurochemical phenoTypes of caudal medullary neurons activated to express cFos following peripheral administration of cholecystokinin.
- Inhibitory effects of estrogen on stimulated salt appetite in rats.
- Central oxytocin inhibition of salt appetite in rats: evidence for differential sensing of plasma sodium and osmolality.
- Effect of acute and chronic hyponatremia on brain buffering in rats.
- c-Fos and Fos-related antigens as markers for neuronal activity: perspectives from neuroendocrine systems.
- c-Fos and related immediate early gene products as markers of activity in neuroendocrine systems.
- Osmotic inhibition of neurohypophysial secretion.
- Cholecystokinin stimulates gonadotropin-releasing hormone release in the monkey (Macaca mulatta).
- Control of brain volume during hyperosmolar and hypoosmolar conditions.
- Effect of acute and chronic hyponatremia on blood-brain barrier function in the rat.
- Hyponatremia induced by vasopressin or desmopressin in female and male rats.
- Central oxytocin inhibition of food and salt ingestion: a mechanism for intake regulation of solute homeostasis.
- Cholecystokinin activates catecholaminergic neurons in the caudal medulla that innervate the paraventricular nucleus of the hypothalamus in rats.
- Dehydration anorexia in decerebrate rats.
- Characterization of oxytocin receptor expression and distribution in the pregnant sheep uterus.
- Chronic hyponatremia reduces survival of magnocellular vasopressin and oxytocin neurons after axonal injury.
- Establishing behavioral and physiological functions of central oxytocin: insights from studies of oxytocin and ingestive behaviors.
- The rat oxytocin receptor. cDNA cloning and immunocytochemical localization in brain, pituitary, mammary gland and uterus.
- Area postrema lesions in rats appear to disrupt rapid feedback inhibition of fluid intake.
- Central inhibition of salt appetite by oxytocin in rats.
- Role of Renal aquaporins in escape from vasopressin-induced antidiuresis in rat.
- Central c-Fos expression in neonatal and adult rats after subcutaneous injection of hypertonic saline.
- Role of aquaporins in water balance Disorders.
- Role of AVP in pressor responses during activation of central TxA2/PGH2 receptors.
- Cholecystokinin induces Fos expression in catecholaminergic neurons of the macaque monkey caudal medulla.
- Adaptation to acute and chronic hyponatremia: implications for symptomatology, diagnosis, and therapy.
- The treatment of severe hyponatremia.
- Medullary c-Fos activation in rats after ingestion of a satiating meal.
- Oxytocin is elevated in plasma of 10-day-old rats following gastric distension.
- Escape from vasopressin-induced antidiuresis: role of vasopressin resistance of the collecting duct.
- Studies of Renal aquaporin-2 expression during Renal escape from vasopressin-induced antidiuresis.
Schools
University of Pittsburgh Main Campus
Conditions Treated
- AdRenal Insufficiency
- Craniopharyngioma
- Diabetes Insipidus
- Diabetes Type 1
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Doctors Specialties
- Endocrinology
Accepted Insurances
Awards
- Top Doctors:Washington DC Area
- Top Doctors:Washington-Baltimore
- Elected Member
Education
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University of Pennsylvania Health System -
University of Pittsburgh School of Medicine
Hospital
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MedStar Washington Hospital Center -
MedStar Georgetown University Hospital
Drug Facts
| NPI NUMBER | 1194724047 | |
| NPPES Provider LastName | VERBALIS | |
| NPPES Provider FirstName | JOSEPH | |
| NPPES Provider ZIPCode | 200072113 | |
| NPPES Provider State | DC | |
| Specialty Description | Endocrinology | |
| Total Claim Count | 45.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1194724047 |
| Last Name Of The Provider | VERBALIS |
| First Name Of The Provider | JOSEPH |
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