Physician
Emergency Medicine
Dr. Julia M. Quinn, MD
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Julia Quinn works in the field of Emergency Medicine. She went to medical school at Texas Tech University Health Sciences Center School of Medicine. Dr. Julia M. Quinn is a published MD as well. She has 25 publications published. The latest was: Interleukin-33, a target of parathyroid hormone and oncostatin m, increases osteoblastic matrix mineral deposition and inhibits osteoclast formation in vitro. She accepts Medicare.
Publications
- A role for alphaV integrin subunit in TGF-beta-stimulated osteoclastogenesis.
- T-cells mediate an inhibitory effect of interleukin-4 on osteoclastogenesis.
- Osteoclast inhibitory lectin, a family of new osteoclast inhibitors.
- Interleukin-33, a target of parathyroid hormone and oncostatin m, increases osteoblastic matrix mineral deposition and inhibits osteoclast formation in vitro.
- A new sol-gel process for producing Na(2)O-containing bioactive glass ceramics.
- Ciliary neurotrophic factor inhibits bone formation and plays a sex-specific role in bone growth and remodeling.
- Germline deletion of AMP-activated protein kinase beta subunits reduces bone mass without altering osteoclast differentiation or function.
- The chemokine Cxcl1 is a novel target gene of parathyroid hormone (PTH)/PTH-related protein in committed osteoblasts.
- Modulation of osteoclast function in bone by the immune system.
- IL-23 inhibits osteoclastogenesis indirectly through lymphocytes and is required for the maintenance of bone mass in mice.
- Osteoclast inhibitory lectin, an immune cell product that is required for normal bone physiology in vivo.
- Cardiotrophin-1 is an osteoclast-derived stimulus of bone formation required for normal bone remodeling.
- EphrinB2 regulation by PTH and PTHrP revealed by molecular profiling in differentiating osteoblasts.
- A novel mutation in the Nfkb2 gene generates an NF-kappa B2 "super repressor".
- Osteoclast inhibitory lectin (OCIL) inhibits osteoblast differentiation and function in vitro.
- Interleukin-6 modulates production of T lymphocyte-derived cytokines in antigen-induced arthritis and drives inflammation-induced osteoclastogenesis.
- Interleukin-11 receptor signaling is required for normal bone remodeling.
- The heat shock protein 90 inhibitor, 17-allylamino-17-demethoxygeldanamycin, enhances osteoclast formation and potentiates bone metastasis of a human breast cancer cel...
- Modulation of osteoclast formation.
- Osteopenia in Siah1a mutant mice.
- Characterization of sugar binding by osteoclast inhibitory lectin.
- Glycoprotein 130 regulates bone turnover and bone size by distinct downstream signaling pathways.
- Isolation of a human homolog of osteoclast inhibitory lectin that inhibits the formation and function of osteoclasts.
- Generation and analysis of Siah2 mutant mice.
Schools
Texas Technical University
University Of Louisville
University Louisville Humana Hospital
Doctors Specialties
- Emergency Medicine
Accepted Insurances
Education
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University of Louisville -
Texas Tech University Health Sciences Center School of Medicine
Drug Facts
| NPI NUMBER | 1437187788 | |
| NPPES Provider LastName | QUINN | |
| NPPES Provider FirstName | JULIA | |
| NPPES Provider ZIPCode | 306776004 | |
| NPPES Provider State | GA | |
| Specialty Description | Emergency Medicine | |
| Total Claim Count | 210.0 | |
| Distinct Opioid Count | 1.0 | |
| Opioid Claim Count | 18.0 | |
| Percent Opioid Claims | 8.57 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1437187788 |
| Last Name Of The Provider | QUINN |
| First Name Of The Provider | JULIA |
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