Physician
Pulmonology
Dr. Karen A. Fagan, MD
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Dr. Karen A. Fagan is an expert in the field of Pulmonology. She researched medicine at University of California San Diego School of Medicine. University of California San Diego School of Medicine is ranked 17 in research and 19 in primary care medicine She over the years received 1 awards: "Regional Top Doctor". Karen A. Fagan has written publications. She has 62 publications published with PubMed. The lastest paper is: 'The pulmonary circulation of homozygous or heterozygous eNOS-null mice is hyperresponsive to mild hypoxia.' She accepts Medicare payments and is registered with Medicare.gov.
Publications
- Dehydroepiandrosterone Restores Right Ventricular Structure and Function in Rats with Severe Pulmonary Arterial Hypertension.
- Misclassification of Pulmonary hypertension in adults with sickle hemoglobinopathies using Doppler echocardiography.
- Cytoskeletal defects in Bmpr2-associated Pulmonary arterial hypertension.
- Tyrosine Kinase Inhibitors are Potent Acute Pulmonary Vasodilators in Rats.
- Formation of plexiform lesions in experimental severe Pulmonary arterial hypertension.
- Rho kinase-mediated vasoconstriction in Pulmonary hypertension.
- Future perspectives for the treatment of Pulmonary arterial hypertension.
- Neprilysin null mice develop exaggerated pulmonary vascular remodeling in response to Chronic hypoxia.
- Vascular dysfunction in a murine model of severe hemolysis.
- Involvement of RhoA/Rho kinase signaling in protection against monocrotaline-induced Pulmonary hypertension in pneumonectomized rats by dehydroepiandrosterone.
- Hypoxia-induced Pulmonary vascular remodeling: cellular and molecular mechanisms.
- Metastatic Cancer while receiving continuous prostacyclin therapy.
- Suppression of type II bone morphogenic protein receptor in vascular smooth muscle induces Pulmonary arterial hypertension in transgenic mice.
- Interleukin-6 causes mild Pulmonary hypertension and augments hypoxia-induced Pulmonary hypertension in mice.
- Pulmonary vascular manifestations of mixed connective tissue disease.
- Inhaled Rho kinase inhibitors are potent and selective vasodilators in rat Pulmonary hypertension.
- Cardioprotective and vasomotor effects of HO activity during acute and Chronic hypoxia.
- Attenuation of acute hypoxic Pulmonary vasoconstriction and hypoxic Pulmonary hypertension in mice by inhibition of Rho-kinase.
- Protein kinase C-epsilon-null mice have decreased hypoxic Pulmonary vasoconstriction.
- Hypoxia and Rho/Rho-kinase signaling. Lung development versus hypoxic Pulmonary hypertension.
- Pulmonary hypertension in TNF-alpha-overexpressing mice is associated with decreased VEGF gene expression.
- Pulmonary hypertension associated with connective tissue disease.
- Translocations involving 4p16.3 in three families: deletion causing the Pitt-Rogers-Danks syndrome and duplication resulting in a new overgrowth syndrome.
- Potential genetic contributions to control of the Pulmonary circulation and ventilation at high altitude.
- A uniform extracellular stimulus triggers distinct cAMP signals in different compartments of a simple cell.
- Regulation of the Ca2+-inhibitable adenylyl cyclase type VI by capacitative Ca2+ entry requires localization in cholesterol-rich domains.
- Adenovirus-mediated expression of an olfactory cyclic nucleotide-gated channel regulates the endogenous Ca2+-inhibitable adenylyl cyclase in C6-2B glioma cells.
- Ca(2+)-sensitive adenylyl cyclases.
- Regulation of a Ca2+-sensitive adenylyl cyclase in an excitable cell. Role of voltage-gated versus capacitative Ca2+ entry.
- Construction of a full-length Ca2+-sensitive adenylyl cyclase/aequorin chimera.
- Functional co-localization of transfected Ca(2+)-stimulable adenylyl cyclases with capacitative Ca2+ entry sites.
- Adenovirus encoded cyclic nucleotide-gated channels: a new methodology for monitoring cAMP in living cells.
- Dependence of the Ca2+-inhibitable adenylyl cyclase of C6-2B glioma cells on capacitative Ca2+ entry.
- Cyclic nucleotide-gated channels colocalize with adenylyl cyclase in regions of restricted cAMP diffusion.
- Adenoviral gene transfer to the neonatal rat Pulmonary circulation.
- Strategic plan for Lung vascular research: An NHLBI-ORDR Workshop Report.
- The low-voltage-activated calcium channel CAV3.1 controls proliferation of human Pulmonary artery myocytes.
- Sustained endothelial nitric-oxide synthase activation requires capacitative Ca2+ entry.
- Use of fat-fed rats to study the metabolic and vascular sequelae of obesity and beta-adrenergic antagonism.
- Role of endothelin-1 in lung Disease.
- Upregulation of nitric oxide synthase in mice with severe hypoxia-induced Pulmonary hypertension.
- Relative contributions of endothelial, inducible, and neuronal NOS to tone in the murine Pulmonary circulation.
- Disrupted Pulmonary vascular development and Pulmonary hypertension in transgenic mice overexpressing transforming growth factor-alpha.
- Pulmonary fibrosis and Chronic lung inflammation in ET-1 transgenic mice.
- p27(Kip1) is important in modulating Pulmonary artery smooth muscle cell proliferation.
- Vascular inflammation inhibits gene transfer to the Pulmonary circulation in vivo.
- Selected Contribution: Pulmonary hypertension in mice following intermittent hypoxia.
- Divergent contractile and structural responses of the murine PKC-epsilon null pulmonary circulation to Chronic hypoxia.
- Low-voltage-activated (T-type) calcium channels control proliferation of human Pulmonary artery myocytes.
- Pulmonary prostacyclin synthase overexpression in transgenic mice protects against development of hypoxic Pulmonary hypertension.
- Pulmonary hypertension in transgenic mice expressing a dominant-negative BMPRII gene in smooth muscle.
- Therapeutic potential of RhoA/Rho kinase inhibitors in Pulmonary hypertension.
- Neonatal Lung side population cells demonstrate endothelial potential and are altered in response to hyperoxia-induced Lung simplification.
- Nitric oxide synthase in Pulmonary hypertension: lessons from knockout mice.
- The Pulmonary circulation of homozygous or heterozygous eNOS-null mice is hyperresponsive to mild hypoxia.
- Effect of nitric oxide on transgene expression in vascular smooth muscle cells.
- Overexpression of tumor necrosis factor-alpha produces an increase in lung volumes and Pulmonary hypertension.
- Mice expressing BMPR2R899X transgene in smooth muscle develop Pulmonary vascular lesions.
- Divergent contractile and structural responses of the murine protein kinase C-epsilon null pulmonary circulation to Chronic hypoxia.
- Interaction of interleukin-6 and the BMP pathway in Pulmonary smooth muscle.
- Dehydroepiandrosterone upregulates soluble guanylate cyclase and inhibits hypoxic Pulmonary hypertension.
Schools
David Geffen School Of Medicine At UCLA University Of California Los Angeles
University Of Co School Of Med
University Tx Southwestern Med School
Procedures Preformed
- Bronchoscopy
- Thoracentesis
- Ventilation Management
Conditions Treated
- Acidosis
- Asbestosis
- Asthma
- Atelectasis
- View All
Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
Awards
- Regional Top Doctor
Education
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University of Colorado -
University of Texas Southwestern Medical School -
University of California San Diego School of Medicine
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