Associate Professor, Medicine, Nephrology; Genetics, Albert Einstein College of Medicine
Nephrology
Dr. Katalin Susztak, MD
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Katalin Susztak is an expert in the field of Nephrology. She went to medical school at Albert Einstein College of Medicine. Katalin Susztak is a published doctor and has 47 publications published. The lastest was: 'Sox9-Positive Progenitor Cells Play a Key Role in Renal Tubule Epithelial Regeneration in Mice.'
Publications
- Genome-wide Association Studies Identify Genetic Loci Associated With Albuminuria in Diabetes.
- Deletion of Lkb1 in Renal Tubular Epithelial Cells Leads to CKD by Altering Metabolism.
- Sox9-Positive Progenitor Cells Play a Key Role in Renal Tubule Epithelial Regeneration in Mice.
- Validation and genomic interrogation of the MET variant rs11762213 as a predictor of adverse outcomes in clear cell renal cell carcinoma.
- Diet-Induced Podocyte Dysfunction in Drosophila and Mammals.
- Defective fatty acid oxidation in renal tubular epithelial cells has a key role in kidney fibrosis development.
- Functional Genomic Annotation of Genetic Risk Loci Highlights Inflammation and Epithelial Biology Networks in CKD.
- Sweet Debate: Fructose versus Glucose in Diabetic Kidney Disease.
- Epigenetics: a new way to look at kidney diseases.
- Notch Ties a Knot on Fistula Maturation.
- The hyperglycemic and hyperinsulinemic combo gives you diabetic kidney disease immediately.
- Sirt1-Claudin-1 crosstalk regulates renal function.
- A multicolor podocyte reporter highlights heterogeneous podocyte changes in focal segmental glomerulosclerosis.
- Cytosine methylation changes in enhancer regions of core pro-fibrotic genes characterize kidney fibrosis development.
- Understanding the Epigenetic Syntax for the Genetic Alphabet in the Kidney.
- Epigenomics: The Science of No-Longer-Junk DNA. Why Study it in Chronic Kidney Disease?
- Diabetic Nephropathy: A National Dialogue.
- For better or worse: a niche for Notch in parietal epithelial cell activation.
- Adiponectin promotes functional recovery after podocyte ablation.
- Notch in the kidney: development and disease.
- Transcriptome analysis of human diabetic kidney disease.
- Wnt/?-catenin pathway in podocytes integrates cell adhesion, differentiation, and survival.
- Tracing the footsteps of glomerular insulin signaling in diabetic kidney disease.
- Fetal environment, epigenetics, and pediatric renal disease.
- The story of Notch and chronic kidney disease.
- Fine tuning gene expression: the epigenome.
- Epithelial Notch signaling regulates interstitial fibrosis development in the kidneys of mice and humans.
- Getting a notch closer to understanding diabetic kidney disease.
- Expression of Notch pathway proteins correlates with albuminuria, glomerulosclerosis, and renal function.
- A susceptibility gene for kidney disease in an obese mouse model of type II diabetes maps to chromosome 8.
- Epithelial-mesenchymal transition and podocyte loss in diabetic kidney disease.
- Mouse models of diabetic nephropathy.
- The pathogenic role of Notch activation in podocytes.
- Human and murine kidneys show gender- and species-specific gene expression differences in response to injury.
- Smad1 as a biomarker for diabetic nephropathy.
- The role of osteopontin in the development of albuminuria.
- The Notch pathway in podocytes plays a role in the development of glomerular disease.
- Animal models of renal disease.
- Diabetic nephropathy: a frontier for personalized medicine.
- Glucose-induced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy.
- Localization of the GLUT8 glucose transporter in murine kidney and regulation in vivo in nondiabetic and diabetic conditions.
- Multiple metabolic hits converge on CD36 as novel mediator of tubular epithelial apoptosis in diabetic nephropathy.
- Molecular profiling of diabetic mouse kidney reveals novel genes linked to glomerular disease.
- Genomic strategies for diabetic nephropathy.
- Smad proteins and transforming growth factor-beta signaling.
- Smad3 and Smad4 mediate transcriptional activation of the human Smad7 promoter by transforming growth factor beta.
Schools
University Of Medicine Of Budapest
Albert Einstein College Med
Doctors Specialties
- Nephrology
Accepted Insurances
Education
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Albert Einstein College of Medicine -
Semmelweis University
Hospital
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Philadelphia Veterans Affairs Medical Center -
Hospital of the University of Pennsylvania
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