Attending physician , Children's Hospital Oakland

Anesthesiology

Doctor Ken Zuckerman, MD works in the field of Anesthesiology. He went to medical school at Tulane University School of Medicine. Zuckerman is also published. He has 27 publications published. The lastest publication: "Characterization of a unique factor-independent variant derived from human factor-dependent TF-1 cells: a transformed event.'

Publications

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• Constitutive activation of the JAK2/STAT5 signal transduction pathway correlates with growth factor independence of megakaryocytic leukemic cell lines.
• Adult Chediak-Higashi parkinsonian syndrome with dystonia.
• Chemotherapy of intermediate- and high-grade non-Hodgkin's lymphomas with a high-dose doxorubicin-containing regimen.
• Chemotherapy of intermediate- and high-grade non-Hodgkin's lymphomas with an intensive epirubicin-containing regimen.
• Clonality in juvenile chronic myelogenous leukemia.
• Cloning and sequencing of an alternative splicing-derived cDNA variant of the GM-CSF receptor alpha subunit, which encodes a truncated protein.
• Characterization of a unique factor-independent variant derived from human factor-dependent TF-1 cells: a transformed event.
• The role of type I and type II tumor necrosis factor (TNF) receptors in the ability of TNF-alpha to transduce a proliferative signal in the human megakaryoblastic leuk...
• Prolonged activation of the mitogen-activated protein kinase pathway is required for macrophage-like differentiation of a human myeloid leukemic cell line.
• Cloning and sequencing of the cDNAs encoding two alternative splicing-derived variants of the alpha subunit of the granulocyte-macrophage colony-stimulating factor rec...
• Activation of p38 mitogen-activated protein kinase is required for tumor necrosis factor-alpha -supported proliferation of leukemia and lymphoma cell lines.
• Transforming growth factor: signal transduction pathways, cell cycle mediation, and effects on hematopoiesis.
• Tumor necrosis factor-alpha-induced proliferation of human Mo7e leukemic cells occurs via activation of nuclear factor kappaB transcription factor.
• An IkappaBalpha inhibitor causes leukemia cell death through a p38 MAP kinase-dependent, NF-kappaB-independent mechanism.
• Cell cycle and transcriptional control of human myeloid leukemic cells by transforming growth factor beta.
• Transforming growth factor beta inhibits growth of more differentiated myeloid leukemia cells and retinoblastoma protein phosphorylation at serine 795.
• Differential effects of transforming growth factor on cell cycle regulatory molecules in human myeloid leukemia cells.
• TNF-alpha-induced growth suppression of CD34+ myeloid leukemic cell lines signals through TNF receptor type I and is associated with NF-kappa B activation.
• TGFbeta inhibits GM-CSF-induced phosphorylation of ERK and MEK in human myeloid leukaemia cell lines via inhibition of phosphatidylinositol 3-kinase (PI3-k).
• Transforming growth factor beta inhibits the phosphorylation of pRB at multiple serine/threonine sites and differentially regulates the formation of pRB family-E2F com...
• Value of positron emission tomography scan in staging cancers, and an unusual presentation of acute myeloid leukemia. Case 3. Acute myeloid leukemia presenting with ly...
• Identification of a novel inhibitor of JAK2 tyrosine kinase by structure-based virtual screening.
• Gap junction mediated transport of shRNA between human embryonic stem cells.
• TGFbeta regulates the expression and activities of G2 checkpoint kinases in human myeloid leukemia cells.
• Using local solutions to address a national issue: ASP's recommendations to ensure the vitality of clinical research.
• Ubiquitin/proteasome-dependent degradation of D-type cyclins is linked to tumor necrosis factor-induced cell cycle arrest.

Schools

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• Tulane University School Of Medicine

• Stanford University

• Chldns National Med Center

Doctors Specialties

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• Anesthesiology

Accepted Insurances

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Education

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• Stanford University
• UCSF
• Tulane University School of Medicine

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