Physician
Internal Medicine
Dr. Kenji Asakura, MD
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Kenji Asakura works in the field of Internal Medicine. He researched medicine at Johns Hopkins University School of Medicine. Johns Hopkins University School of Medicine is a top medical college. Johns Hopkins University School of Medicine is ranked 3/29 in Research/PrimaryCare. Kenji Asakura is a published MD. He has 44 papers published. The most current publication "P/Q-type Ca2+ channel blocker omega-agatoxin IVA protects against brain injury after focal ischemia in rats." He accepts all Medicare patients.
Publications
- A multifaceted hospitalist quality improvement intervention: Decreased frequency of common labs.
- Is your clinical documentation improvement program compliant?
- Successful treatment of post-MRSA infection glomerulonephritis with steroid therapy.
- IgA nephropathy associated with hyper IgAnemia, psoriasis or pustulosis and ossification.
- Goodpasture''s syndrome: a report of an autopsy case and a review of Japanese cases.
- Acute renal failure following hypokalemic rhabdomyolysis due to chronic glycyrrhizic acid administration.
- [A case of myelofibrosis with multiple tumorous formation at the terminal stage].
- Improved search for nu(mu) --> nu(e) oscillation in a long-baseline accelerator experiment.
- Search for coherent charged pion production in neutrino-carbon interactions.
- Evidence for muon neutrino oscillation in an accelerator-based experiment.
- Electrophoretic karyotypes of clinically isolated yeasts of Candida albicans and C. glabrata.
- Internalization and sorting of plasma membrane sphingolipid analogues in differentiating oligodendrocytes.
- Effects of transforming growth factor-beta and platelet-derived growth factor on oligodendrocyte precursors: insights gained from a neuronal cell line.
- Effects of S-2474, a novel nonsteroidal anti-inflammatory drug, on amyloid beta protein-induced neuronal cell death.
- Gas6 rescues cortical neurons from amyloid beta protein-induced apoptosis.
- alpha-eudesmol, a P/Q-type Ca(2+) channel blocker, inhibits neurogenic vasodilation and extravasation following electrical stimulation of trigeminal ganglion.
- kappa-opioid agonist U50488 inhibits P-type Ca2+ channels by two mechanisms.
- P/Q-type Ca2+ channel blocker omega-agatoxin IVA protects against brain injury after focal ischemia in rats.
- Synthesis and structure-activity relationships of potent and orally active sulfonamide ETB selective antagonists.
- The nonpeptide alpha-eudexp6l from Juniperus virginiana Linn. (Cupressaceae) inhibits omega-agatoxin IVA-sensitive Ca2+ currents and synaptosomal 45Ca2+ uptake.
- Partially oxygenated sickled cells: sickle-shaped red cells found in circulating blood of patients with sickle cell disease.
- Deterioration of axotomy-induced neurodegeneration by group IIA secretory phospholipase A2.
- omega-agatoxin IVA-sensitive Ca(2+) channel blocker, alpha-eudesmol, protects against brain injury after focal ischemia in rats.
- Chlorpromazine reduces toxicity and Ca2+ uptake induced by amyloid beta protein (25-35) in vitro.
- Amyloid beta protein potentiates Ca2+ influx through L-type voltage-sensitive Ca2+ channels: a possible involvement of free radicals.
- Adrenergic regulation of AMP-activated protein kinase in brown adipose tissue in vivo.
- Irbesartan enhances GLUT4 translocation and glucose transport in skeletal muscle cells.
- Identification of glycosylated exendin-4 analogue with prolonged blood glucose-lowering activity through glycosylation scanning substitution.
- Neuronal protein tyrosine phosphatase 1B deficiency results in inhibition of hypothalamic AMPK and isoform-specific activation of AMPK in peripheral tissues.
- Chemoenzymatic synthesis of glycosylated glucagon-like peptide 1: effect of glycosylation on proteolytic resistance and in vivo blood glucose-lowering activity.
- A high-fat, ketogenic diet induces a unique metabolic state in mice.
- Diet-induced obesity alters AMP kinase activity in hypothalamus and skeletal muscle.
- Exposure of blood from patients with sickle cell disease to air changes the morphological, oxygen-binding, and sickling properties of sickled erythrocytes.
- Blood samples collected under venous oxygen pressure from patients with sickle cell disease contain a significant number of a new type of reversibly sickled cells: con...
- Novel binding sites of 15-deoxy-Delta12,14-prostaglandin J2 in plasma membranes from primary rat cortical neurons.
- Effect of Gas6 on secretory phospholipase A(2)-IIA-induced apoptosis in cortical neurons.
- Human group IIA secretory phospholipase A2 potentiates Ca2+ influx through L-type voltage-sensitive Ca2+ channels in cultured rat cortical neurons.
- Porcine pancreatic group IB secretory phospholipase A2 potentiates Ca2+ influx through L-type voltage-sensitive Ca2+ channels.
- Prostaglandin E2 rescues cortical neurons from amyloid beta protein-induced apoptosis.
- Effects of endothelin B receptor agonists on amyloid beta protein (25-35)-induced neuronal cell death.
- Group IB secretory phospholipase A(2)induces cell death in the cultured cortical neurons: a possible involvement of its binding sites.
- Group IB secretory phospholipase A2 induces neuronal cell death via apoptosis.
- Human group IIA secretory phospholipase A2 induces neuronal cell death via apoptosis.
Schools
Johns Hopkins University School of Medicine
Doctors Specialties
- Internal Medicine
Accepted Insurances
Education
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University of Washington -
Johns Hopkins University School of Medicine
Hospital
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Swedish Medical Center-Cherry Hill Campus
Drug Facts
| NPI NUMBER | 1538179965 | |
| NPPES Provider LastName | ASAKURA | |
| NPPES Provider FirstName | KENJI | |
| NPPES Provider ZIPCode | 981224379 | |
| NPPES Provider State | WA | |
| Specialty Description | Internal Medicine | |
| Total Claim Count | 60.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1538179965 |
| Last Name Of The Provider | ASAKURA |
| First Name Of The Provider | KENJI |
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