Professor, Pediatrics, Indiana University School of Medicine
Neonat/Perinatology
Dr. Laura S. Haneline, MD
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Laura Haneline works in the field of Neonat/Perinatology. He went to Indiana University School of Medicine. Indiana University School of Medicine is ranked 45/47 in Research/PrimaryCare. He has 1 awards "Elected Member". Laura Haneline is a published MD. He published 42 publications, including: 'Multiple inhibitory cytokines induce deregulated progenitor growth and apoptosis in hematopoietic cells from Fac-/- mice.'
Publications
- Epigenetic regulation of PLAC8 contributes to altered function of endothelial colony forming cells exposed to intrauterine gestational diabetes mellitus.
- Lung parenchymal development in premature infants without bronchopulmonary dysplasia.
- Fetal hyperglycemia and a high fat diet contribute to aberrant glucose tolerance and hematopoiesis in adult rats.
- Gestational diabetes induces alterations in the function of neonatal endothelial colony forming cells.
- Membrane and Capillary Components of Lung Diffusion and Pro-Angiogenic Cells in Infants.
- Impaired function of Fanconi anemia type C-deficient macrophages.
- Restrictive loss of Plakoglobin in cardiomyocytes leads to Arrhythmogenic Cardiomyopathy.
- Fanconi anemia D2 protein is an apoptotic target mediated by caspases.
- Gestational diabetes mellitus alters maternal and neonatal circulating endothelial progenitor cell subsets.
- Endothelial abnormalities in adolescents with type 1 diabetes: a biomarker for vascular sequelae?
- TERTrific hormones promote hematopoiesis.
- Distinct roles of stress-activated protein kinases in Fanconi anemia-type C-deficient hematopoiesis.
- Suppressed hindlimb perfusion in Rac2-/- and Nox2-/- mice does not result from impaired collateral growth.
- Oxidative stress impairs endothelial progenitor cell function.
- Redox regulation of stem and progenitor cells.
- Endothelial colony forming cells and mesenchymal stem cells are enriched at different gestational ages in human umbilical cord blood.
- In vitro hyperglycemia or a diabetic intrauterine environment reduces neonatal endothelial colony-forming cell numbers and function.
- Cul4A is required for hematopoietic stem-cell engraftment and self-renewal.
- Clonogenic endothelial progenitor cells are sensitive to oxidative stress.
- Continuous in vivo infusion of interferon-gamma (IFN-gamma) enhances engraftment of syngeneic wild-type cells in Fanca-/- and Fancg-/- mice.
- Genetic reduction of class IA PI-3 kinase activity alters fetal hematopoiesis and competitive repopulating ability of hematopoietic stem cells in vivo.
- Enhanced TNF-alpha-induced apoptosis in Fanconi anemia type C-deficient cells is dependent on apoptosis signal-regulating kinase 1.
- Ex vivo culture of Fancc-/- stem/progenitor cells predisposes cells to undergo apoptosis, and surviving stem/progenitor cells display cytogenetic abnormalities and an ...
- Lentiviral vectors pseudotyped with glycoproteins from Ross River and vesicular stomatitis viruses: variable transduction related to cell type and culture conditions.
- Continuous in vivo infusion of interferon-gamma (IFN-gamma) preferentially reduces myeloid progenitor numbers and enhances engraftment of syngeneic wild-type cells in ...
- Oxidant hypersensitivity of Fanconi anemia type C-deficient cells is dependent on a redox-regulated apoptotic pathway.
- Fanconi anemia type C and p53 cooperate in apoptosis and tumorigenesis.
- Distinct pathways involving the FK506-binding proteins 12 and 12.6 underlie IL-2-versus IL-15-mediated proliferation of T cells.
- Fanconi anemia type C-deficient hematopoietic stem/progenitor cells exhibit aberrant cell cycle control.
- Retroviral-mediated expression of recombinant Fancc enhances the repopulating ability of Fancc-/- hematopoietic stem cells and decreases the risk of clonal evolution.
- Phenotypic correction of primary Fanconi anemia T cells with retroviral vectors as a diagnostic tool.
- FANCC suppresses short telomere-initiated telomere sister chromatid exchange.
- Human Fanconi A cells are susceptible to TRAIL-induced apoptosis.
- Human CD34+AC133+VEGFR-2+ cells are not endothelial progenitor cells but distinct, primitive hematopoietic progenitors.
- BMP10 is essential for maintaining cardiac growth during murine cardiogenesis.
- The highest concentration of primitive hematopoietic progenitor cells in cord blood is found in extremely premature infants.
- Multiple inhibitory cytokines induce deregulated progenitor growth and apoptosis in hematopoietic cells from Fac-/- mice.
- Expression and functional characterization of the beta-isoform of the folate receptor on CD34(+) cells.
- Loss of FancC function results in decreased hematopoietic stem cell repopulating ability.
- Dishevelled-associated activator of morphogenesis 1 (Daam1) is required for heart morphogenesis.
- The Epigenetic Regulator CXXC Finger Protein 1 is Essential for Murine Hematopoiesis.
Schools
Indiana University School of Medicine
Clarian Indiana University Hospital
In University School Of Med
Doctors Specialties
- Neonatal Medicine
- Neonat/Perinatology
Accepted Insurances
- Aetna
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Cofinity PPO
- Commercial Insurance Company
- HealthSmart
- Choice Care PPO
- Medicaid
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- Priority PPO
- Sagamore Plus
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Awards
- Elected Member
Education
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Indiana University School of Medicine
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