Physician
Pulmonology
Dr. Leland G. Dobbs, MD
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Leland Dobbs works in the field of Pulmonology. He graduated from Columbia University College of Physicians & Surgeons. Columbia University College of Physicians & Surgeons is a top medical college. Columbia University College of Physicians & Surgeons has a rank of 8 in research and a 52 in primary care He has 2 awards "Regional Top Doctor" and "CMS Meaningful Use Stage 1 Certification". Leland Dobbs is also published. He has 73 publications published. The lastest publication: "Purification and analysis of RTI40, a type I alveolar epithelial cell apical membrane protein.' He accepts Medicare payments and is listed with Medicare.gov.
Publications
- High Efficiency Type II Cell EGFP Expression Facilitates Cellular Identification, Tracking, and Isolation.
- Claudin-18 deficiency results in alveolar barrier dysfunction and impaired alveologenesis in mice.
- Isolation and culture of alveolar epithelial Type I and Type II cells from rat Lungs.
- HTII-280, a biomarker specific to the apical plasma membrane of human Lung alveolar type II cells.
- In vivo intravital endoscopic confocal fluorescence microscopy of normal and acutely injured rat Lungs.
- Rat alveolar type I cells proliferate, express OCT-4, and exhibit phenotypic plasticity in vitro.
- Directed expression of transgenes to alveolar type I cells in the mouse.
- Adenosine regulation of alveolar fluid clearance.
- Functional ion channels in Pulmonary alveolar type I cells support a role for type I cells in lung ion transport.
- CXC chemokines and their receptors are expressed in type II cells and upregulated following Lung injury.
- Continuous mechanical contraction modulates expression of alveolar epithelial cell phenotype.
- Effects of oligohydramnios on Lung growth and maturation in the fetal rat.
- Alveolar epithelial type I cells contain transport proteins and transport sodium, supporting an active role for type I cells in regulation of Lung liquid homeostasis.
- The great big alveolar TI cell: evolving concepts and paradigms.
- Alveolar epithelial transport in the adult Lung.
- Characteristics of Cl- uptake in rat alveolar type I cells.
- Identification of genes differentially expressed in rat alveolar type I cells.
- Freshly isolated rat alveolar type I cells, type II cells, and cultured type II cells have distinct molecular phenotypes.
- Differential effects of sustained inflation recruitment maneuvers on alveolar epithelial and Lung endothelial injury.
- Low tidal volume reduces epithelial and endothelial injury in acid-injured rat Lungs.
- Highly water-permeable type I alveolar epithelial cells confer high water permeability between the airspace and vasculature in rat Lung.
- Characterization of the gene and promoter for RTI40, a differentiation marker of type I alveolar epithelial cells.
- A novel alveolar type I cell-specific biochemical marker of human acute Lung injury.
- HTI56, an integral membrane protein specific to human alveolar type I cells.
- Mechanical distention modulates alveolar epithelial cell phenotypic expression by transcriptional regulation.
- Purification and analysis of RTI40, a type I alveolar epithelial cell apical membrane protein.
- Monoclonal antibodies specific to apical surfaces of rat alveolar type I cells bind to surfaces of cultured, but not freshly isolated, type II cells.
- Effects of mechanical factors on growth and maturation of the Lung in fetal sheep.
- Biochemical detection of type I cell damage after nitrogen dioxide-induced Lung injury in rats.
- Mechanical distension modulates Pulmonary alveolar epithelial phenotypic expression in vitro.
- HTI56, an integral apical membrane protein of the human alveolar type I cell, is a biochemical marker of acute Lung injury.
- Expression of cell-specific markers for alveolar epithelium in fetal rat Lung.
- Nitric oxide attenuates Lung endothelial injury caused by sublethal hyperoxia in rats.
- Synthesis of surfactant components by cultured type II cells from human Lung.
- A type I cell-specific protein is a biochemical marker of epithelial injury in a rat model of Pneumonia.
- Localization of a candidate surfactant convertase to type II cells, macrophages, and surfactant subfractions.
- Low density lipoprotein- and high density lipoprotein-mediated signal transduction and exocytosis in alveolar type II cells.
- Maintenance of the differentiated type II cell phenotype by culture with an apical air surface.
- Alveolar epithelial injury and pleural Empyema in acute P. aeruginosa pneumonia in anesthetized rabbits.
- [Ca(2+)](i) oscillations regulate type II cell exocytosis in the Pulmonary alveolus.
- Ultrastructure of phospholipid mixtures reconstituted with surfactant proteins B and D.
- Identification of a novel antigen on the apical surface of rat alveolar epithelial type II and Clara cells.
- Activation of G proteins may inhibit or stimulate surfactant secretion in rat alveolar type II cells.
- Evidence for G beta gamma-mediated cross-talk in primary cultures of Lung alveolar cells. Pertussis toxin-sensitive production of cAMP.
- The role of calcium in the secretion of surfactant by rat alveolar type II cells.
- Isolation and culture of alveolar type II cells.
- Pulmonary surfactant and its components inhibit secretion of phosphatidylcholine from cultured rat alveolar type II cells.
- Calcium mobilization and exocytosis after one mechanical stretch of Lung epithelial cells.
- An improved method for isolating type II cells in high yield and purity.
- Impaired Lung branching morphogenesis in the absence of functional EGF receptor.
- Positive correlation between cytosolic free calcium and surfactant secretion in cultured rat alveolar type II cells.
- Mechanical forces modulate alveolar epithelial phenotypic expression.
- Lipoprotein-stimulated surfactant secretion in alveolar type II cells: mediation by heterotrimeric G proteins.
- Prolonged transgene expression in rodent Lung cells.
- Alveoli and airways: possible interactions in the pathogenesis of Asthma.
- Pulmonary alveolar type II cells isolated from rats. Release of phosphatidylcholine in response to beta-adrenergic stimulation.
- Cloning, characterization, and development expression of a rat Lung alveolar type I cell gene in embryonic endodermal and neural derivatives.
- Cholera toxin stimulates secretion of saturated phosphatidylcholine and increases cellular cyclic AMP in isolated rat alveolar type II cells.
- Metabolic properties and ultrastructure of alveolar type II cells isolated with elastase.
- Secretion of surfactant by primary cultures of alveolar type II cells isolated from rats.
- Loss of immunoreactivity for RTI40, a type I cell-specific protein in the alveolar epithelium of rat Lungs with bleomycin-induced fibrosis.
- Alveolar type II cells.
- Stretch-dependent regulation of atrial peptide synthesis and secretion in cultured atrial cardiocytes.
- Changes in biochemical characteristics and pattern of lectin binding of alveolar type II cells with time in culture.
- Regulation of Pulmonary surfactant secretion and clearance.
- Surfactant apoprotein Mr = 26,000-36,000 enhances uptake of liposomes by type II cells.
- Pulmonary surfactant.
- Stimulation of secretion of disaturated phosphatidylcholine from isolated alveolar type II cells by 12-O-tetradecanoyl-13-phorbol acetate.
- Surfactant protein C promoter-driven expression of T1-alpha induces Lung inflammation.
- Regulation of ATP-dependent surfactant secretion and activation of second-messenger systems in alveolar type II cells.
- Synthesis of phosphatidylcholine and phosphatidylglycerol by alveolar type II cells in primary culture.
- The effects of mechanical forces on Lung functions.
Schools
Columbia University College Of Physicians and Surgeons
New York Presbyterian Hospital
Roosevelt Hospital
Procedures Preformed
- Bronchoscopy
- Ventilation Management
Conditions Treated
- Acidosis
- Alkalosis
- Asthma
- Atelectasis
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Power Select HMO
- Blue Card PPO
- CaliforniaCare HMO
- Advantage Preferred DirectAccess Plus PPO
- Blue Cross Blue Shield of California PPO
- Select Plus HMO
- Basic Exclusive PPO for Small Business
- Basic PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- LocalPlus
- Open Access Plus
- First Health (Coventry Health Care) PPO
- Blue & Gold HMO
- Covered California - Bronze PPO
- Employer Group HMO
- Health Net PPO
- Individual and Family PPO
- WholeCare Network HMO
- Kaiser PHCS Network PPO
- MultiPlan PPO
- PHCS Network PPO
- Heritage Signature
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- PacifiCare HMO
Awards
- Regional Top Doctor
- CMS Meaningful Use Stage 1 Certification
Education
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UCSF -
St Luke's-Roosevelt Hospital Center -
New York Presbyterian Hospital - Columbia Campus -
Columbia University College of Physicians & Surgeons
Hospital
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Alta Bates Summit Medical Center - Summit Campus -
Alta Bates Summit Medical Center
Drug Facts
| NPI NUMBER | 1760422356 | |
| NPPES Provider LastName | DOBBS | |
| NPPES Provider FirstName | LELAND | |
| NPPES Provider ZIPCode | 946093424 | |
| NPPES Provider State | CA | |
| Specialty Description | Pulmonary Disease | |
| Total Claim Count | 593.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1760422356 |
| Last Name Of The Provider | DOBBS |
| First Name Of The Provider | LELAND |
| View All |
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