Assistant Professor of Pathology, University of Chicago Medicine
Pathology
Dr. Loren J. Joseph, MD
Doctor Loren J. Joseph, MD works in the field of Pathology. He went to medical school at Stanford University School of Medicine. Stanford University School of Medicine is one of the top medical schools in the US. Stanford University School of Medicine is ranked 2 in research and 25 in primary care medicine Doctor Loren J. Joseph, MD is a published doctor and has 30 publications published. The lastest was: 'Genetic profiling of advanced radioactive iodine-resistant differentiated thyroid cancer and correlation with axitinib efficacy.'
Publications
- Genetic profiling of advanced radioactive iodine-resistant differentiated thyroid cancer and correlation with axitinib efficacy.
- Tumor suppressors miR-143 and miR-145 and predicted target proteins API5, ERK5, K-RAS, and IRS-1 are differentially expressed in proximal and distal colon.
- The Renin-Angiotensin system mediates EGF receptor-vitamin d receptor cross-talk in colitis-associated colon cancer.
- Pancreatic ductal adenocarcinoma with autoimmune pancreatitis-like histologic and immunohistochemical features.
- Detection and Quantification of BCR-ABL1 Fusion Transcripts by Droplet Digital PCR.
- Phase II Study Of The Oral MEK Inhibitor Selumetinib In Advanced Acute Myeloid Leukemia (AML): A University Of Chicago Phase II Consortium Trial.
- GFI1B, EVI5, MYB-Additional genes that cooperate with the human BCL6 gene to promote the development of lymphomas.
- Immune reconstitution after combined haploidentical and umbilical cord blood transplant.
- Gene signature distinguishes patients with chronic ulcerative colitis harboring remote neoplastic lesions.
- Multiplexed Quantification of Nucleic Acids with Large Dynamic Range Using Multivolume Digital RT-PCR on a Rotational SlipChip Tested with HIV and Hepatitis C Viral Load.
- miR-143 and miR-145 are downregulated in ulcerative colitis: Putative regulators of inflammation and protooncogenes.
- Molecular biology underlying the clinical heterogeneity of prostate cancer: an update.
- RNA reference materials for gene expression studies. RNA metrology: forecast calls for partial clearing.
- Reduced-intensity conditioning with combined haploidentical and cord blood transplantation results in rapid engraftment, low GVHD, and durable remissions.
- Epidermal growth factor receptor is required for colonic tumor promotion by dietary fat in the azoxymethane/dextran sulfate sodium model: roles of transforming growth ...
- Patterns and kinetics of T-cell chimerism after allo transplant with alemtuzumab-based conditioning: mixed chimerism protects from GVHD, but does not portend disease r...
- Epidermal growth factor receptor signaling is up-regulated in human colonic aberrant crypt foci.
- COLD-PCR enhanced melting curve analysis improves diagnostic accuracy for KRAS mutations in colorectal carcinoma.
- EGFR signals downregulate tumor suppressors miR-143 and miR-145 in Western diet-promoted murine colon cancer: role of G1 regulators.
- Ursodeoxycholic acid suppresses Cox-2 expression in colon cancer: roles of Ras, p38, and CCAAT/enhancer-binding protein.
- Identification of histologically distinct conventional adenomas that arise predominately in patients with sessile serrated adenomas.
- Paxillin expression and amplification in early lung lesions of high-risk patients, lung adenocarcinoma and metastatic disease.
- Epidermal growth factor receptor signaling is required for microadenoma formation in the mouse azoxymethane model of colonic carcinogenesis.
- Ursodeoxycholic acid inhibits Ras mutations, wild-type Ras activation, and cyclooxygenase-2 expression in colon cancer.
- Microsatellite analysis of sporadic flat and depressed lesions of the colon.
- A vitamin D analogue inhibits colonic carcinogenesis in the AOM/DSS model.
- Epidermal growth factor receptor controls flat dysplastic aberrant crypt foci development and colon cancer progression in the rat azoxymethane model.
- Ursodeoxycholic acid and F(6)-D(3) inhibit aberrant crypt proliferation in the rat azoxymethane model of colon cancer: roles of cyclin D1 and E-cadherin.
- Inducible heat shock protein 70 prevents multifocal flat dysplastic lesions and invasive tumors in an inflammatory model of colon cancer.
Schools
Stanford University School Of Medicine
Northwestern Meml
Stanford
University of Chicago
National Institute Of Health
Doctors Specialties
- Pathology
Accepted Insurances
Education
-
University of Chicago -
National Institutes of Health Clinical Center -
McGaw Medical Center of Northwestern University -
Stanford University -
Stanford University School of Medicine
Hospital
-
University of Chicago Medical Center -
Beth Israel Deaconess Medical Center
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1487706164 |
| Last Name Of The Provider | JOSEPH |
| First Name Of The Provider | LOREN |
| View All |
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