Associate Professor, Dermatology, Stony Brook University SOM
Dermatology
Dr. Marcia Tonnesen, MD
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Marcia Tonnesen works in the field of Dermatology. She graduated from University of Utah School of Medicine. She has 1 awards "Fellow (FAAD)". Marcia Tonnesen has written publications. She has 26 publications published with PubMed. The lastest paper is: 'Human mast cells stimulate vascular tube formation. Tryptase is a novel, potent angiogenic factor.' Doctor Marcia Tonnesen, MD accepts Medicare payments and is registered with Medicare.gov.
Publications
- Loss of Primary Cilia in Melanoma Cells Is Likely Independent of Proliferation and Cell Cycle Progression.
- Fibronectin Peptides that Bind PDGF-BB Enhance Survival of Cells and Tissue Under Stress.
- Nitric oxide contributes to cytokine-induced apoptosis in pancreatic beta cells via potentiation of JNK activity and inhibition of Akt.
- Inhibition of histone deacetylases prevents cytokine-induced toxicity in beta cells.
- Effects of shear stress on adhesive interaction between neutrophils and cultured endothelial cells.
- Human monocyte adherence: a primary effect of chemotactic factors on the monocyte to stimulate adherence to human endothelium.
- Stimulation of eosinophil adherence to human vascular endothelial cells in vitro by platelet-activating factor.
- Isotretinoin produces significant inhibition of monocyte and neutrophil chemotaxis in vivo in patients with cystic Acne.
- Lymphocyte subsets and Langerhans cells/indeterminate cells in erythema multiforme.
- Erythema multiforme: microvascular damage and infiltration of lymphocytes and basophils.
- Human mast cells stimulate vascular tube formation. Tryptase is a novel, potent angiogenic factor.
- Adherence of neutrophils to cultured human microvascular endothelial cells. Stimulation by chemotactic peptides and lipid mediators and dependence upon the Mac-1, LFA-...
- Neutrophil-endothelial cell interactions: mechanisms of neutrophil adherence to vascular endothelium.
- Expression of fibronectin, laminin, and factor VIII-related antigen during development of the human cutaneous microvasculature.
- Transient functional expression of alphaVbeta 3 on vascular cells during wound repair.
- Angiogenesis in wound healing.
- Keratinocytes in human wounds express alpha v beta 6 integrin.
- Mast cells are potent regulators of endothelial cell adhesion molecule ICAM-1 and VCAM-1 expression.
- Mesenchymal cell activation is the rate-limiting step of granulation tissue induction.
- Fibrinogen and fibrin are anti-adhesive for keratinocytes: a mechanism for fibrin eschar slough during wound repair.
- Human fibroblasts bind directly to fibrinogen at RGD sites through integrin alpha(v)beta3.
- Fibrin and collagen differentially regulate human dermal microvascular endothelial cell integrins: stabilization of alphav/beta3 mRNA by fibrin1.
- Fibronectin growth factor-binding domains are required for fibroblast survival.
- Tissue engineering for cutaneous wounds.
- Cooperation of C1q receptors and integrins in C1q-mediated endothelial cell adhesion and spreading.
Schools
University Of Utah School Of Medicine
Barnes Jewish Hospital
Harvard University
Brigham Hospital Harvard
Conditions Treated
- Acne
- Actinic Keratosis
- Athlete's Foot
- Benign Skin Lesion
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Doctors Specialties
- Dermatology
Accepted Insurances
Awards
- Fellow (FAAD)
Education
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Massachusetts General Hospital -
Washington University - Barnes-Jewish Hospital -
University of Utah School of Medicine
Hospital
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Stony Brook University Medical Center
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1205883238 |
| Last Name Of The Provider | TONNESEN |
| First Name Of The Provider | MARCIA |
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