Professor,Medicine, Baylor College of Medicine
Cardiology
Dr. Mark L. Entman, MD
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Mark Entman practices Cardiology. He went to Duke University School of Medicine. Duke University School of Medicine is one of the top medical schools in the US. Duke University School of Medicine is ranked 8 in research and 29 in primary care medicine He received awards:"Elected Member", "Investigator Alumnus" and "Fellow (FACC)". Mark Entman is a published doctor as well. He has 71 publications published. The latest was: Plasma Levels of Endothelial Microparticles Bearing Monomeric C-reactive Protein are Increased in Peripheral Artery Disease.
Publications
- Mesenchymal stem cell-derived inflammatory fibroblasts mediate interstitial fibrosis in the aging heart.
- Plasma Levels of Endothelial Microparticles Bearing Monomeric C-reactive Protein are Increased in Peripheral Artery Disease.
- Left Atrial Volume and Pulmonary Artery Diameter Are Noninvasive Measures of Age-Related Diastolic Dysfunction in Mice.
- Circulating Collagen Biomarkers as Indicators of Disease Severity in Pulmonary Arterial Hypertension.
- CXCR6 Plays a Critical Role in Angiotensin II-Induced Renal Injury and Fibrosis.
- Steroid Receptor Coactivator-2 Is a Dual Regulator of Cardiac Transcription Factor Function.
- The chemokine receptor CXCR6 contributes to recruitment of bone marrow-derived fibroblast precursors in renal fibrosis.
- Critical Role of CXCL16 in Hypertensive Kidney Injury and Fibrosis.
- Adverse fibrosis in the aging heart depends on signaling between myeloid and mesenchymal cells; role of inflammatory fibroblasts.
- AICAR-dependent AMPK activation improves scar formation in the aged heart in a murine model of reperfused myocardial infarction.
- Young Little Mice Express a Premature Cardiovascular Aging Phenotype.
- SRC-2 coactivator deficiency decreases functional reserve in response to pressure overload of mouse heart.
- Aberrant differentiation of fibroblast progenitors contributes to fibrosis in the aged murine heart: role of elevated circulating insulin levels.
- CXCL16 Recruits Bone Marrow-Derived Fibroblast Precursors in Renal Fibrosis.
- Defective Myofibroblast Formation from Mesenchymal Stem Cells in the Aging Murine Heart Rescue by Activation of the AMPK Pathway.
- Doppler Velocity Measurements from Large and Small Arteries of Mice.
- CCR2 Mediates the Uptake of Bone Marrow-Derived Fibroblast Precursors in Angiotensin II-induced Cardiac Fibrosis.
- Cardiac mesenchymal stem cells contribute to scar formation after myocardial infarction.
- Cardiomyocyte PDGFR-beta signaling is an essential component of the mouse cardiac response to load-induced stress.
- The cytoprotective effects of tumor necrosis factor are conveyed through tumor necrosis factor receptor-associated factor 2 in the heart.
- Coronary flow reserve in mice: effects of age, coronary disease, and vascular loading.
- Adaptive and maladptive effects of SMAD3 signaling in the adult heart after hemodynamic pressure overloading.
- Coronary flow reserve as an index of cardiac function in mice with cardiovascular abnormalities.
- Increased myocardial susceptibility to repetitive ischemia with high-fat diet-induced obesity.
- Fc receptor engagement mediates differentiation of cardiac fibroblast precursor cells.
- Cardiac function in young and old Little mice.
- Critical role of monocyte chemoattractant protein-1/CC chemokine ligand 2 in the pathogenesis of ischemic cardiomyopathy.
- The role of platelet-derived growth factor signaling in healing myocardial infarcts.
- Bone marrow-derived fibroblast precursors mediate ischemic cardiomyopathy in mice.
- Effects of diet-induced obesity on inflammation and remodeling after myocardial infarction.
- Signaling mechanism of renal fibrosis in unilateral ureteral obstructive kidney disease in ROCK1 knockout mice.
- Activation of Rho-associated coiled-coil protein kinase 1 (ROCK-1) by caspase-3 cleavage plays an essential role in cardiac myocyte apoptosis.
- Oncostatin M differentially regulates CXC chemokines in mouse cardiac fibroblasts.
- Induction of antioxidant gene expression in a mouse model of ischemic cardiomyopathy is dependent on reactive oxygen species.
- Targeted deletion of ROCK1 protects the heart against pressure overload by inhibiting reactive fibrosis.
- Identification of mast cells in the cellular response to myocardial infarction.
- Pulsed Doppler signal processing for use in mice: applications.
- Regulatable atrial natriuretic peptide gene therapy for hypertension.
- Downregulation of peroxisome proliferator-activated receptor-alpha gene expression in a mouse model of ischemic cardiomyopathy is dependent on reactive oxygen species ...
- Chemokines in myocardial ischemia.
- Critical role of endogenous thrombospondin-1 in preventing expansion of healing myocardial infarcts.
- CCL2/Monocyte Chemoattractant Protein-1 regulates inflammatory responses critical to healing myocardial infarcts.
- Targeting the chemokines in myocardial inflammation.
- Noninvasive ultrasonic measurement of arterial wall motion in mice.
- Vascular mural cells in healing canine myocardial infarcts.
- Determinants of cardiac electrophysiological properties in mice.
- Disruption of Rho signaling results in progressive atrioventricular conduction defects while ventricular function remains preserved.
- Cardiac muscle plasticity in adult and embryo by heart-derived progenitor cells.
- Molecular normalization of dystrophin in the failing left and right ventricle of patients treated with either pulsatile or continuous flow-type ventricular assist devi...
- Of mice and dogs: species-specific differences in the inflammatory response following myocardial infarction.
- Evidence of improved right ventricular structure after LVAD support in patients with end-stage cardiomyopathy.
- Nuclear factor-kappaB protects the adult cardiac myocyte against ischemia-induced apoptosis in a murine model of acute myocardial infarction.
- Cardiac progenitor cells from adult myocardium: homing, differentiation, and fusion after infarction.
- Measurement of aortic input impedance in mice: effects of age on aortic stiffness.
- Effect of age on peripheral vascular response to transverse aortic banding in mice.
- MCSF expression is induced in healing myocardial infarcts and may regulate monocyte and endothelial cell phenotype.
- Inhibitory cardiac transcription factor, SRF-N, is generated by caspase 3 cleavage in human heart failure and attenuated by ventricular unloading.
- Telomere attrition and Chk2 activation in human heart failure.
- Development of murine ischemic cardiomyopathy is associated with a transient inflammatory reaction and depends on reactive oxygen species.
- Soluble adhesion molecules and the search for biomarkers for atherosclerosis.
- Active interstitial remodeling: an important process in the hibernating human myocardium.
- Evidence for an active inflammatory process in the hibernating human myocardium.
- Heart failure and greater infarct expansion in middle-aged mice: a relevant model for postinfarction failure.
- Morphological characteristics of the microvasculature in healing myocardial infarcts.
- Noninvasive cardiovascular phenotyping in mice.
- Rho associated coiled-coil kinase-1 regulates collagen-induced phosphatidylserine exposure in platelets.
- CCR2 regulates the uptake of bone marrow-derived fibroblasts in renal fibrosis.
- microRNA-22 Promotes Heart Failure through Coordinate Suppression of PPAR/ERR-Nuclear Hormone Receptor Transcription.
- Th1/M1 conversion to th2/m2 responses in models of inflammation lacking cell death stimulates maturation of monocyte precursors to fibroblasts.
- Glucose Regulation of Load-Induced mTOR Signaling and ER Stress in Mammalian Heart.
Schools
Duke University
Johns Hopkins Hospital
Duke University Med Center
Doctors Specialties
- Cardiology
Accepted Insurances
Awards
- Elected Member
- Investigator Alumnus
- Fellow (FACC)
Education
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Duke University Hospital -
Johns Hopkins University -
Duke University School of Medicine
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