Professor, Neurology, Massachusetts General Hospital, Harvard Medical School
Neurology
Dr. Michael A. Moskowitz, MD
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Michael Moskowitz is an expert in the field of Neurology. He attended Tufts University School of Medicine. Tufts University School of Medicine has a rank of 49 in research and a 52 in primary care He received 1 awards: "Whos Who in Medicine and Healthcare". Michael Moskowitz has written publications. He has 97 publications published with PubMed. The lastest publication is: 'Supply-Demand Mismatch Transients in Susceptible Peri-infarct Hot Zones Explain the Origins of Spreading Injury Depolarizations.'
Publications
- Supply-Demand Mismatch Transients in Susceptible Peri-infarct Hot Zones Explain the Origins of Spreading Injury Depolarizations.
- Myeloperoxidase propagates damage and is a potential therapeutic target for subacute stroke.
- Ischemic Stroke Activates Hematopoietic Bone Marrow Stem Cells.
- Chaos and commotion in the wake of cortical spreading depression and spreading depolarizations.
- The Innate Immune System After Ischemic Injury: Lessons to Be Learned From the Heart and Brain.
- Diffusion tensor and volumetric magnetic resonance imaging using an MR-compatible hand-induced robotic device suggests training-induced neuroplasticity in patients wit...
- The challenge of connecting the dots in the B.R.A.I.N.
- Bo K. Siesjo, MD, PhD: January 8, 1930, to June 27, 2013.
- fMRI as a molecular imaging procedure for the functional reorganization of motor systems in chronic stroke.
- The NINDS Stroke Progress Review Group Final Analysis and Recommendations.
- Myocardial infarction accelerates atherosclerosis.
- Rapid monocyte kinetics in acute myocardial infarction are sustained by extramedullary monocytopoiesis.
- Migraine Mutations Increase Stroke Vulnerability by Facilitating Ischemic Depolarizations.
- Activation of the migraine pain pathway by cortical spreading depression: Do we need more evidence?
- PNAS Plus: Hypomorphic Notch 3 alleles link Notch signaling to ischemic cerebral small-vessel disease.
- Striatal neurons expressing full-length mutant huntingtin exhibit decreased N-cadherin and altered neuritogenesis.
- Enhanced subcortical spreading depression in familial hemiplegic migraine type 1 mutant mice.
- Remote ischemic preconditioning: making the brain more tolerant, safely and inexpensively.
- Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy syndrome mutations increase susceptibility to spreading depression.
- Brain protection: maybe yes, maybe no.
- High-mobility group box 1 promotes metalloproteinase-9 upregulation through Toll-like receptor 4 after cerebral ischemia.
- Soluble guanylate cyclase alpha1beta1 limits stroke size and attenuates neurological injury.
- The science of stroke: mechanisms in search of treatments.
- Perfusion pressure-dependent recovery of cortical spreading depression is independent of tissue oxygenation over a wide physiologic range.
- Microemboli may link spreading depression, migraine aura, and patent foramen ovale.
- Sonic hedgehog regulates ischemia/hypoxia-induced neural progenitor proliferation.
- Androgenic suppression of spreading depression in familial hemiplegic migraine type 1 mutant mice.
- Context dependent neuroprotective properties of prion protein (PrP).
- Connectivity alterations assessed by combining fMRI and MR-compatible hand robots in chronic stroke.
- p27Kip1 constrains proliferation of neural progenitor cells in adult brain under homeostatic and ischemic conditions.
- Rac1 is a critical mediator of endothelium-derived neurotrophic activity.
- Genetic and hormonal factors modulate spreading depression and transient hemiparesis in mouse models of familial hemiplegic migraine type 1.
- Obesity increases vascular senescence and susceptibility to ischemic injury through chronic activation of Akt and mTOR.
- Inhibition of cerebral vasoconstriction by dantrolene and nimodipine.
- Tracking the inflammatory response in stroke in vivo by sensing the enzyme myeloperoxidase.
- Necrostatin-1 reduces histopathology and improves functional outcome after controlled cortical impact in mice.
- Hypoxia and hypotension transform the blood flow response to cortical spreading depression from hyperemia into hypoperfusion in the rat.
- Additive effects of statin and dipyridamole on cerebral blood flow and stroke protection.
- Simultaneous multispectral reflectance imaging and laser speckle flowmetry of cerebral blood flow and oxygen metabolism in focal cerebral ischemia.
- The impact of anesthetics and hyperoxia on cortical spreading depression.
- Defining a pathway to discovery from bench to bedside: the trigeminovascular system and sensitization.
- Mild induced hypertension improves blood flow and oxygen metabolism in transient focal cerebral ischemia.
- Early release of HMGB-1 from neurons after the onset of brain ischemia.
- Stroke program review group: an interim report.
- Linking Notch signaling to ischemic stroke.
- Acute plasmalemma permeability and protracted clearance of injured cells after controlled cortical impact in mice.
- Adiponectin prevents cerebral ischemic injury through endothelial nitric oxide synthase dependent mechanisms.
- Blood vessels, migraine, and stroke.
- Cortical spreading depression and estrogen.
- MR contrast probes that trace gene transcripts for cerebral ischemia in live animals.
- The phosphorylation state of eNOS modulates vascular reactivity and outcome of cerebral ischemia in vivo.
- Rho-kinase inhibition acutely augments blood flow in focal cerebral ischemia via endothelial mechanisms.
- The 2006 Thomas Willis lecture: the adventures of a translational researcher in stroke and migraine.
- Pathophysiology of headache--past and present.
- fMRI of delayed albumin treatment during stroke recovery in rats: implication for fast neuronal habituation in recovering brains.
- Rapid nongenomic actions of thyroid hormone.
- Vasoconstrictive neurovascular coupling during focal ischemic depolarizations.
- Recommendations of the National Heart, Lung, and Blood Institute working group on cerebrovascular biology and disease.
- Cortical spreading depression confounds concentration-dependent pial arteriolar dilation during N-methyl-D-aspartate superfusion.
- Traumatic brain injury in mice deficient in Bid: effects on histopathology and functional outcome.
- Suppression of cortical spreading depression in migraine prophylaxis.
- The dynamics of statins: from event prevention to neuroprotection.
- Inhibition of Rho kinase (ROCK) leads to increased cerebral blood flow and stroke protection.
- Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia.
- Advances in stroke neuroprotection: hyperoxia and beyond.
- Expression of cellular FLICE inhibitory proteins (cFLIP) in normal and traumatic murine and human cerebral cortex.
- Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury.
- Protective effects of statins involving both eNOS and tPA in focal cerebral ischemia.
- Migraine as an inflammatory disorder.
- Exciting, radical, suicidal: how brain cells die after stroke.
- Pronounced hypoperfusion during spreading depression in mouse cortex.
- Laser speckle flowmetry for the study of cerebrovascular physiology in normal and ischemic mouse cortex.
- Cortical spreading depression activates and upregulates MMP-9.
- Regenerative response in ischemic brain restricted by p21cip1/waf1.
- Nuclear translocation of apoptosis-inducing factor after focal cerebral ischemia.
- tPA and proteolysis in the neurovascular unit.
- Deciphering migraine mechanisms: clues from familial hemiplegic migraine genotypes.
- Nasal vaccination with myelin oligodendrocyte glycoprotein reduces stroke size by inducing IL-10-producing CD4+ T cells.
- Effect of basic fibroblast growth factor treatment on brain progenitor cells after permanent focal ischemia in rats.
- Therapeutic implications of central and peripheral neurologic mechanisms in migraine.
- FGF-2 regulates neurogenesis and degeneration in the dentate gyrus after traumatic brain injury in mice.
- Rapid cerebral ischemic preconditioning in mice deficient in endothelial and neuronal nitric oxide synthases.
- EGF amplifies the replacement of parvalbumin-expressing striatal interneurons after ischemia.
- Neurogenesis and apoptotic cell death.
- Simultaneous imaging of total cerebral hemoglobin concentration, oxygenation, and blood flow during functional activation.
- Rapid nontranscriptional activation of endothelial nitric oxide synthase mediates increased cerebral blood flow and stroke protection by corticosteroids.
- Caspase activation and neuroprotection in caspase-3- deficient mice after in vivo cerebral ischemia and in vitro oxygen glucose deprivation.
- STAT1 is activated in neurons after ischemia and contributes to ischemic brain injury.
- The HD mutation does not alter neuronal death in the striatum of Hdh(Q92) knock-in mice after mild focal ischemia.
- Mechanisms of pain modulation in chronic syndromes.
- Cell biology. PARP-1--a perpetrator of apoptotic cell death?
- Acute cardiovascular protective effects of corticosteroids are mediated by non-transcriptional activation of endothelial nitric oxide synthase.
- Upregulation of the Fas receptor death-inducing signaling complex after traumatic brain injury in mice and humans.
- Nuclear factor-kappaB as a molecular target for migraine therapy.
- Neurotrophin-3 promotes cell death induced in cerebral ischemia, oxygen-glucose deprivation, and oxidative stress: possible involvement of oxygen free radicals.
- Intrinsic brain activity triggers trigeminal meningeal afferents in a migraine model.
Schools
Tufts University
Yale New Haven Hospital
Peter Bent Brigham Hospital
Mass Institute Tech
Doctors Specialties
- Internal Medicine
- Neurology
Accepted Insurances
Awards
- Whos Who in Medicine and Healthcare
Education
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Boston Children's Hospital -
Yale-New Haven Hospital -
Tufts University School of Medicine
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