Physician

Neurology

Dr. Michael G. Schlossmacher, MD

Michael Schlossmacher is an expert in the field of Neurology. Michael G. Schlossmacher is a published doctor. He published 50 publications, including: 'The life of Eugene Braunwald: a remarkable journey that began in Vienna.'

Publications

Association between Î±-synuclein blood transcripts and early, neuroimaging-supported Parkinson's disease.
Parkin-Dependent Degradation of the F-box protein Fbw7Î² Promotes Neuronal Survival in response to oxidative stress by Stabilizing Mcl-1.
Association of SNCA with Parkinson: replication in the Harvard NeuroDiscovery Center Biomarker Study.
CNS expression of glucocerebrosidase corrects {alpha}-synuclein pathology and memory in a mouse model of Gaucher-related synucleinopathy.
Acid Î²-glucosidase mutants linked to gaucher disease, parkinson disease, and lewy body dementia alter Î±-synuclein processing.
Translational Research in Neurology and Neuroscience 2011: Movement Disorders.
Direct quantification of CSF alpha-synuclein by ELISA and first cross-sectional study in patients with neurodegeneration.
Multiple system atrophy: a primary oligodendrogliopathy.
GATA transcription factors directly regulate the Parkinson's disease-linked gene alpha-synuclein.
Parkinson disease, 10 years after its genetic revolution: multiple clues to a complex disorder.
Aggregated alpha-synuclein mediates dopaminergic neurotoxicity in vivo.
Structure of acid beta-glucosidase with pharmacological chaperone provides insight into Gaucher disease.
Molecular markers of early Parkinson's disease based on gene expression in blood.
Decreased alpha-synuclein in cerebrospinal fluid of aged individuals and subjects with Parkinson's disease.
Phosphorylation of Ser-129 is the dominant pathological modification of alpha-synuclein in familial and sporadic Lewy body disease.
Parkin protects against mitochondrial toxins and beta-amyloid accumulation in skeletal muscle cells.
Detection of oligomeric forms of alpha-synuclein protein in human plasma as a potential biomarker for Parkinson's disease.
Synphilin-1 and parkin show overlapping expression patterns in human brain and form aggresomes in response to proteasomal inhibition.
Dopamine covalently modifies and functionally inactivates parkin.
Lewy body Parkinson's disease in a large pedigree with 77 Parkin mutation carriers.
The glucocerebrosidase gene and Parkinson's disease in Ashkenazi Jews.
Parkinson's disease: assays for the ubiquitin ligase activity of neural Parkin.
Dimerization of Parkinson's disease-causing DJ-1 and formation of high molecular weight complexes in human brain.
Case records of the Massachusetts General Hospital. Weekly clinicopathological exercises. Case 27-2004. A 79-year-old woman with disturbances in gait, cognition, and a...
Seizures after epidural blood patch.
Parkin localizes to the Lewy bodies of Parkinson disease and dementia with Lewy bodies.
Parkinson''s disease-linked LRRK2 is expressed in circulating and tissue immune cells and upregulated following recognition of microbial structures.
Cathepsin D expression level affects alpha-synuclein processing, aggregation, and toxicity in vivo.
Increased DJ-1 expression under oxidative stress and in Alzheimer''s disease brains.
Expansion of the Parkinson disease-associated SNCA-Rep1 allele upregulates human alpha-synuclein in transgenic mouse brain.
CSF synuclein: adding to the biomarker footprint of dementia with Lewy bodies.
Biomarker research in Parkinson''s disease: objective measures needed for patient stratification in future cause-directed trials.
Quantification of Î±-synuclein in cerebrospinal fluid as a biomarker candidate: review of the literature and considerations for future studies.
Î±-Synuclein and tau concentrations in cerebrospinal fluid of patients presenting with parkinsonism: a cohort study.
The effects of oxidative stress on parkin and other E3 ligases.
RING finger 1 mutations in Parkin produce altered localization of the protein.
Deciphering the role of heterozygous mutations in genes associated with parkinsonism.
Serum heart-type fatty acid-binding protein and cerebrospinal fluid tau: marker candidates for dementia with Lewy bodies.
The genetics of Parkinson disease: Implications for neurological care.
Amyloid beta-peptide is produced by cultured cells during normal metabolism.
Detection of distinct isoform patterns of the beta-amyloid precursor protein in human platelets and lymphocytes.
Detection of soluble forms of the beta-amyloid precursor protein in human plasma.
Normal cellular processing of the beta-amyloid precursor protein results in the secretion of the amyloid beta peptide and related molecules.
beta-Amyloid peptide and a 3-kDa fragment are derived by distinct cellular mechanisms.
The life of Eugene Braunwald: a remarkable journey that began in Vienna.
Ubiquitination of a new form of alpha-synuclein by parkin from human brain: implications for Parkinson's disease.
Isolation and quantification of soluble Alzheimer''s beta-peptide from biological fluids.
Hashimoto''s encephalopathy mimicking Creutzfeldt-Jakob disease: brain biopsy findings.
Vertebral artery dissection with a normal-appearing lumen at multisection CT angiography: the importance of identifying wall hematoma.

Schools

University of Vienna
Harvard Med School
Harvard Med SchMa Genl HospBrigham and Womens Hospital

Doctors Specialties

Neurology

Accepted Insurances

Education

Beth Israel Deaconess Medical Center

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