Professor of Medicine, University Mass School of Medicine
Endocrinology
Dr. Neil M. Aronin, MD
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Neil Aronin is an expert in the field of Endocrinology. He studied medicine at Mount Sinai School of Medicine. He received awards:"Regional Top Doctor", "Meaningful Use Stage 1 Certification" and "Fellow (FACE)". Neil Aronin is a published doctor. He published 101 publications, including: 'Mutant huntingtin expression in clonal striatal cells: dissociation of inclusion formation and neuronal survival by caspase inhibition.' Neil Aronin accepts all Medicare patients.
Publications
- Excitatory transmission onto AgRP neurons is regulated by cJun NH2-terminal kinase 3 in response to metabolic stress.
- Huntingtin-lowering strategies in Huntington's Disease: Antisense oligonucleotides, small RNAs, and gene editing.
- A synthetic luciferin improves bioluminescence imaging in live mice.
- Striatal Synaptosomes from Hdh(140Q/140Q) Knock-in Mice have Altered Protein Levels, Novel Sites of Methionine Oxidation, and Excess Glutamate Release after Stimulation.
- Frameless multimodal image guidance of localized convection-enhanced delivery of therapeutics in the brain.
- Hunting down huntingtin.
- Native Mutant Huntingtin in Human Brain: EVIDENCE FOR PREVALENCE OF FULL-LENGTH MONOMER.
- Huntingtin is present in the nucleus, interacts with the transcriptional corepressor C-terminal binding protein, and represses transcription.
- Lysosomal proteases are involved in generation of N-terminal huntingtin fragments.
- Novel Pheochromocytoma susceptibility loci identified by integrative genomics.
- Huntingtin associates with acidic phosphoLipids at the plasma membrane.
- Photoinducible and rhythmic ICER-CREM immunoreactivity in the rat suprachiasmatic nucleus.
- Striatal potassium channel dysfunction in Huntington's Disease transgenic mice.
- Huntingtin bodies sequester vesicle-associated proteins by a polyproline-dependent interaction.
- Autophagy regulates the processing of amino terminal huntingtin fragments.
- Asymmetry in the assembly of the RNAi enzyme complex.
- Huntingtin is degraded to small fragments by calpain after ischemic injury.
- siRNAs knock down hepatitis.
- Increased huntingtin protein length reduces the number of polyglutamine-induced gene expression changes in mouse models of Huntington''s Disease.
- Striatal neurochemical changes in transgenic models of Huntington's Disease.
- Argonaute protein identity and pairing geometry determine cooperativity in mammalian RNA silencing.
- Oligonucleotide therapeutic approaches for Huntington Disease.
- Huntingtin cleavage product A forms in neurons and is reduced by gamma-secretase inhibitors.
- Aberrant Rab11-dependent trafficking of the neuronal glutamate transporter EAAC1 causes oxidative stress and cell death in Huntington's Disease.
- Germline mutations in TMEM127 confer susceptibility to Pheochromocytoma.
- Mutant huntingtin impairs vesicle formation from recycling endosomes by interfering with Rab11 activity.
- Disruption of Rab11 activity in a knock-in mouse model of Huntington's Disease.
- Mutant huntingtin and glycogen synthase kinase 3-beta accumulate in neuronal lipid rafts of a presymptomatic knock-in mouse model of Huntington's Disease.
- Polyglutamine expansion in huntingtin alters its interaction with phosphoLipids.
- Expanded CAG repeats in the crosshairs.
- Five siRNAs targeting three SNPs may provide therapy for three-quarters of Huntington''s Disease patients.
- Linking SNPs to CAG repeat length in Huntington's Disease patients.
- A function of huntingtin in guanine nucleotide exchange on Rab11.
- Designing siRNA that distinguish between genes that differ by a single nucleotide.
- Target selectivity in mRNA silencing.
- Therapeutic silencing of mutant huntingtin with siRNA attenuates striatal and cortical neuropathology and behavioral deficits.
- Maintenance of susceptibility to neurodegeneration following intrastriatal injections of quinolinic acid in a new transgenic mouse model of Huntington's Disease.
- Changes in cortical and striatal neurons predict behavioral and electrophysiological abnormalities in a transgenic murine model of Huntington's Disease.
- Caspase 3-cleaved N-terminal fragments of wild-Type and mutant huntingtin are present in normal and Huntington's disease brains, associate with membranes, and undergo ...
- Early and progressive accumulation of reactive microglia in the Huntington Disease brain.
- Pro-caspase-8 is predominantly localized in mitochondria and released into cytoplasm upon apoptotic stimulation.
- Huntingtin expression stimulates endosomal-lysosomal activity, endosome tubulation, and autophagy.
- Differential regulation of fos family genes in the ventrolateral and dorsomedial subdivisions of the rat suprachiasmatic nucleus.
- Are there multiple pathways in the pathogenesis of Huntington''s Disease?
- Forskolin and dopamine D1 receptor activation increase huntingtin's association with endosomes in immortalized neuronal cells of striatal origin.
- Axonal transport of N-terminal huntingtin suggests early pathology of corticostriatal projections in Huntington Disease.
- Mutant huntingtin expression in clonal striatal cells: dissociation of inclusion formation and neuronal survival by caspase inhibition.
- Analysis of Huntingtin-associated protein 1 in mouse brain and immortalized striatal neurons.
- Wild-Type and mutant huntingtins function in vesicle trafficking in the secretory and endocytic pathways.
- Are neuronal intranuclear inclusions the common neuropathology of triplet-repeat Disorders with polyglutamine-repeat expansions?
- Huntingtin localization in brains of normal and Huntington's Disease patients.
- Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brain.
- Fast transport and retrograde movement of huntingtin and HAP 1 in axons.
- Dde-I restriction endonuclease fragmentation: a novel method of generating cDNA probes for in situ hybridization in brain.
- A kindred with a variant of multiple endocrine neoplasia Type 1 demonstrating frequent expression of pituitary tumors but not linked to the multiple endocrine neoplasi...
- In vitro stimulation of c-Fos protein expression in the suprachiasmatic nucleus of hypothalamic slices.
- c-Fos expression in the rat intergeniculate leaflet: photic regulation, co-localization with Fos-B, and cellular identification.
- Expression of normal and mutant huntingtin in the developing brain.
- Unexpected c-fos gene expression in the suprachiasmatic nucleus of mice entrained to a skeleton photoperiod.
- Metastatic papillary Thyroid carcinoma to lung diagnosed by bronchoalveolar lavage.
- The myopathy of Cushing's Syndrome.
- Compositional changes of AP-1 DNA-binding proteins are regulated by light in a mammalian circadian clock.
- Colocalization of D1 and D2 dopamine receptor mRNAs in striatal neurons.
- Temporal regulation of light-induced Fos and Fos-like protein expression in the ventrolateral subdivision of the rat suprachiasmatic nucleus.
- Circadian regulation of Fos B is different from c-Fos in the rat suprachiasmatic nucleus.
- Functional role for Sp1 in the transcriptional amplification of a cell cycle regulated histone H4 gene.
- CAG expansion affects the expression of mutant Huntingtin in the Huntington's Disease brain.
- Monoamines in the brain cerebrospinal fluid of facial pain patients.
- Prognostic differences in subgroups of patients with electrographic evidence of subendocardial or transmural myocardial infarction. The favorable outlook for patients ...
- The stability and metabolism of intravenously administered neurotensin in the rat.
- Environmental iodine intake and thyroid Dysfunction during chronic amiodarone therapy.
- Localization of immunoreactive enkephalins in GABA synthesizing neurons of the rat neostriatum.
- Ultrastructural localization of immunoreactive neurotensin in the monkey superficial dorsal horn.
- Plasma immunoreactive beta-endorphin is elevated in uraemia.
- Ultrastructural localization and biochemical features of immunoreactive LEU-enkephalin in monkey dorsal horn.
- Failure of bromocriptine to lower plasma catecholamines in normal men and women.
- Comparative metabolic clearance rates of beta-endorphin and beta-lipotropin in humans.
- Impaired clearance of beta-lipotropin in uremia.
- Sustained remission of Nelson's Syndrome after stopping cyproheptadine treatment.
- The effects of gonadal steroids on the content of substance P in the rat anterior pituitary.
- Regulation by Thyroid hormone of the concentration of substance P in the rat anterior pituitary.
- The distribution of substance P-containing neurons in the cat Edinger-Westphal nucleus: relationship to efferent projection systems.
- Somatostatin is increased in the basal ganglia in Huntington Disease.
- Substance P and neurotensin.
- Light microscopic and ultrastructural localization of immunoreactive substance P in the dorsal horn of monkey spinal cord.
- Immunoreactive substance P in the substantia nigra of the monkey: light and electron microscopic localization.
- Quantitative electron microscopic study of immunoreactive somatostatin axons in the rat neostriatum.
- Immunoreactive leu-enkephalin in the monkey hypothalamus including observations on its ultrastructural localization in the paraventricular nucleus.
- Ultrastructural features of immunoreactive somatostatin neurons in the rat caudate nucleus.
- Light and electron microscopic localization of immunoreactive Leu-enkephalin in the monkey basal ganglia.
- Frequency of painless myocardial ischemia during exercise tolerance testing in patients with and without Diabetes mellitus.
- Glutamic acid decarboxylase and enkephalin immunoreactive axon terminals in the rat neostriatum synapse with striatonigral neurons.
- Immunohistochemical evidence for neural mediation of VIP activity in the dogfish rectal Gland.
- Ultrastructural localization of immunoreactive calbindin-D28k in the rat and monkey basal ganglia, including subcellular distribution with colloidal gold labeling.
- Hypothyroidism increases substance P concentrations in the heterotopic anterior pituitary.
- A Golgi study of the monkey paraventricular nucleus: neuronal Types, afferent and efferent fibers.
- Diminished flare response in neuropathic diabetic patients. Comparison of effects of substance P, histamine, and capsaicin.
- Substance P and neurotensin: their roles in the regulation of anterior pituitary function.
- The differential effects of Thyroid and gonadal hormones on substance P content in the anterior pituitary of the prepubertal rat.
- Diabetes mellitus and silent myocardial ischemia.
Schools
Perelman School of Medicine University of Pennsylvania
Duke University Med Center
Massachusetts General Hospital
Mt Sinai Med Center
Conditions Treated
- Adrenal Incidentaloma
- Adrenal Insufficiency
- Adrenogenital Disorders
- Craniopharyngioma
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Doctors Specialties
- Endocrinology
Accepted Insurances
Awards
- Regional Top Doctor
- Meaningful Use Stage 1 Certification
- Fellow (FACE)
Education
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Massachusetts General Hospital -
Mount Sinai School of Medicine -
Duke University Hospital -
Perelman School of Medicine at the University of Pennsylvania
Hospital
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UMass Memorial Medical Center
Drug Facts
| NPI NUMBER | 1679556252 | |
| NPPES Provider LastName | ARONIN | |
| NPPES Provider FirstName | NEIL | |
| NPPES Provider ZIPCode | 016550002 | |
| NPPES Provider State | MA | |
| Specialty Description | Endocrinology | |
| Total Claim Count | 99.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1679556252 |
| Last Name Of The Provider | ARONIN |
| First Name Of The Provider | NEIL |
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