Professor, Medicine-Massachusetts General Hospital, Harvard Medical School
Cardiology
Dr. Paul L. Huang, MD
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Paul Huang works in the field of Cardiology. He went to Harvard Medical School. Harvard Medical School is a top medical university. Harvard Medical School is ranked 1 in research and 12 in primary care medicine He over the years received 2 awards: "Elected Member" and "Fellow (FACC)". Paul Huang is also a published MD. He has 74 paper published. The most recent paper is: A Novel Dual NO-donating Oxime and c-Jun N-terminal Kinase Inhibitor Protects Against Cerebral Ischemia-Reperfusion Injury in Mice. Paul Huang accepts Medicare payments and is listed with Medicare.gov.
Publications
- Endothelial dysfunction abrogates the efficacy of normobaric hyperoxia in stroke.
- Live-Cell Real-Time Imaging of Mitochondria in HIV-1-Infected Cells.
- Rho-kinase inhibition improves ischemic perfusion deficit in hyperlipidemic mice.
- Phosphomimetic modulation of eNOS improves Myocardial reperfusion and mimics cardiac postconditioning in mice.
- eNOS-derived nitric oxide regulates endothelial barrier function through VE-cadherin and Rho GTPases.
- Metabolic regulation of cancer cell side population by glucose through activation of the Akt pathway.
- Deficient eNOS Phosphorylation Is a Mechanism for Diabetic Vascular Dysfunction Contributing to Increased Stroke Size.
- Induction of Vascular Insulin Resistance and Endothelin-1 Expression and Acceleration of Atherosclerosis by the Overexpression of Protein Kinase C-β Isoform in the End...
- Loss of p53 and altered miR15-a/16-1î²MCL-1 pathway in CLL: insights from TCL1-Tg:p53(-/-) mouse model and primary human leukemia cells.
- C-reactive protein causes insulin resistance in mice through Fcγ receptor IIB-mediated inhibition of skeletal muscle glucose delivery.
- Hyperlipidemia disrupts cerebrovascular reflexes and worsens ischemic perfusion defect.
- Cerebral blood volume affects blood-brain barrier integrity in an Acute transient stroke model.
- eNOS phosphorylation on serine 1176 affects insulin sensitivity and adiposity.
- Endothelial nitric oxide synthase controls the expression of the angiogenesis inhibitor thrombospondin 2.
- Nitric oxide synthase 3 deficiency limits adverse ventricular remodeling after pressure overload in insulin resistance.
- Rhesus macaques develop metabolic Syndrome with reversible vascular dysfunction responsive to pioglitazone.
- Delayed paraplegia after spinal cord ischemic injury requires caspase-3 activation in mice.
- Aldosterone Regulates Vascular Gene Transcription via Oxidative Stress-Dependent and -Independent Pathways.
- Optical coherence tomography for the quantitative study of cerebrovascular physiology.
- Implantable magnetic relaxation sensors measure cumulative exposure to cardiac biomarkers.
- Role of Endothelial Nitric Oxide in Cerebrovascular Regulation.
- Soluble guanylate cyclase alpha1beta1 limits stroke size and attenuates neurological injury.
- Loss of insulin signaling in vascular endothelial cells accelerates atherosclerosis in apolipoprotein E null mice.
- A new activity of anti-HIV and anti-tumor protein GAP31: DNA adenosine glycosidase--structural and modeling insight into its functions.
- eNOS, metabolic Syndrome and cardiovascular disease.
- Oxidative stress and compartment of gene expression determine proatherosclerotic effects of inducible nitric oxide synthase.
- The Akt1-eNOS axis illustrates the specificity of kinase-substrate relationships in vivo.
- Cerebrovascular thromboprophylaxis in mice by erythrocyte-coupled tissue-type plasminogen activator.
- Effects of neuroglobin overexpression on Acute brain injury and long-term outcomes after focal cerebral ischemia.
- Contributions of nitric oxide synthase isoforms to pulmonary oxygen toxicity, local vs. mediated effects.
- The phosphorylation state of eNOS modulates vascular reactivity and outcome of cerebral ischemia in vivo.
- Rho-kinase inhibition Acutely augments blood flow in focal cerebral ischemia via endothelial mechanisms.
- Discovery of small-molecule HIV-1 fusion and integrase inhibitors oleuropein and hydroxytyrosol: part II. integrase inhibition.
- Discovery of small-molecule HIV-1 fusion and integrase inhibitors oleuropein and hydroxytyrosol: Part I. fusion [corrected] inhibition.
- Atheroprotective effects of neuronal nitric oxide synthase in apolipoprotein e knockout mice.
- Alternatively spliced neuronal nitric oxide synthase mediates penile erection.
- Tissue plasminogen activator promotes matrix metalloproteinase-9 upregulation after focal cerebral ischemia.
- NO mediates mural cell recruitment and vessel morphogenesis in murine melanomas and tissue-engineered blood vessels.
- Unraveling the links between diabetes, obesity, and cardiovascular Disease.
- Protective effects of statins involving both eNOS and tPA in focal cerebral ischemia.
- Near-infrared fluorescent imaging of matrix metalloproteinase activity after Myocardial infarction.
- Structural and functional modeling of human lysozyme reveals a unique nonapeptide, HL9, with anti-HIV activity.
- Endothelial nitric oxide synthase regulates microlymphatic flow via collecting lymphatics.
- Role of endothelial nitric oxide synthase in endothelial activation: insights from eNOS knockout endothelial cells.
- Mechanisms of postspaceflight orthostatic Hypotension: low alpha1-adrenergic receptor responses before flight and central autonomic dysregulation postflight.
- Endothelial nitric oxide synthase and endothelial dysfunction.
- Nitric oxide protects cardiac sarcolemmal membrane enzyme function and ion active transport against ischemia-induced inactivation.
- Mice lacking inducible nitric oxide synthase demonstrate impaired killing of Porphyromonas gingivalis.
- Anti-HIV activity of olive leaf extract (OLE) and modulation of host cell gene expression by HIV-1 infection and OLE treatment.
- Inducible nitric-oxide synthase is an important contributor to prolonged protective effects of ischemic preconditioning in the mouse Kidney.
- Rapid cerebral ischemic preconditioning in mice deficient in endothelial and neuronal nitric oxide synthases.
- Effects of Chronic treatment with L-arginine on atherosclerosis in apoE knockout and apoE/inducible NO synthase double-knockout mice.
- Vasculoprotective roles of neuronal nitric oxide synthase.
- Long-term treatment with N(omega)-nitro-L-arginine methyl ester causes arteriosclerotic Coronary lesions in endothelial nitric oxide synthase-deficient mice.
- Deletion of exon 6 of the neuronal nitric oxide synthase gene in mice results in hypogonadism and infertility.
- Lack of nitric oxide synthase depresses ion transporting enzyme function in cardiac muscle.
- In vivo imaging of proteolytic activity in atherosclerosis.
- Stimulation of nitric oxide synthase in cerebral cortex due to elevated partial pressures of oxygen: an oxidative stress response.
- Noncholinergic penile erection in mice lacking the gene for endothelial nitric oxide synthase.
- The Study to Understand the Genetics of the Acute Response to Metformin and Glipizide in Humans (SUGAR-MGH): Design of a pharmacogenetic Resource for Type 2 Diabetes.
- Anti-Inflammatory Effect of Targeted Delivery of SOD to Endothelium: Mechanism, Synergism with NO Donors and Protective Effects In Vitro and In Vivo.
- C-reactive protein, an ''intermediate phenotype'' for inflammation: human twin studies reveal heritability, association with blood pressure and the metabolic Syndrome,...
- Human ISL1 Heart progenitors generate diverse multipotent cardiovascular cell lineages.
- A comprehensive definition for metabolic Syndrome.
- [Mice lacking the eNOS gene].
- Decreased arteriolar density in endothelial nitric oxide synthase knockout mice is due to Hypertension, not to the constitutive defect in endothelial nitric oxide synt...
- Nitric oxide and cerebral ischemic preconditioning.
- Oxygen seizure latency and peroxynitrite formation in mice lacking neuronal or endothelial nitric oxide synthases.
- Cardiovascular roles of nitric oxide: a review of insights from nitric oxide synthase gene disrupted mice.
- Expression of neuronal nitric oxide synthase splice variants in atherosclerotic plaques of apoE knockout mice.
- Nitric oxide regulates the Heart by spatial confinement of nitric oxide synthase isoforms.
- Endothelial nitric oxide synthase transgenic models of endothelial dysfunction.
- A Novel Dual NO-donating Oxime and c-Jun N-terminal Kinase Inhibitor Protects Against Cerebral Ischemia-Reperfusion Injury in Mice.
Schools
Harvard Medical School
Massachusetts General Hospital
Procedures Preformed
- Cardiac Catheterization (incl. Coronary Angiography)
- Cardiac Imaging
- Cardiac Myocardial Perfusion Imaging
- Cardioversion, Elective
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Conditions Treated
- Angina and Acute Coronary Syndrome
- Aortic Aneurysm
- Aortic Valve Disease
- Arrhythmias (incl. Atrial Fibrillation)
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Doctors Specialties
- Cardiology
Accepted Insurances
Awards
- Elected Member
- Fellow (FACC)
Education
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Massachusetts General Hospital -
Harvard Medical School
Hospital
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Massachusetts General Hospital
Drug Facts
| NPI NUMBER | 1174504351 | |
| NPPES Provider LastName | HUANG | |
| NPPES Provider FirstName | PAUL | |
| NPPES Provider ZIPCode | 021142621 | |
| NPPES Provider State | MA | |
| Specialty Description | Cardiology | |
| Total Claim Count | 423.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1174504351 |
| Last Name Of The Provider | HUANG |
| First Name Of The Provider | PAUL |
| View All |
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