Associate Professor of Pediatrics, Boston Children's Hospital

Infectious Disease

Paula I. Watnick, MD practices Infectious Disease. She went to medical school at Yale University School of Medicine. Yale University School of Medicine is a top medical university. Yale University School of Medicine is ranked 7/57 in Research/PrimaryCare. Paula Watnick is also published. She has 37 publications published. The lastest publication: "The interaction of the Vibrio cholerae transcription factors, Fur and IrgB, with the overlapping promoters of two virulence genes, irgA and irgB.'

Publications

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• Regulation of CsrB/C sRNA decay by EIIA(Glc) of the phosphoenolpyruvate: carbohydrate phosphotransferase system.
• The acetate switch of an intestinal pathogen disrupts host insulin signaling and lipid metabolism.
• The transcription factor Mlc promotes Vibrio cholerae biofilm formation through repression of PTS components.
• Mannitol and the Mannitol-Specific Enzyme IIB Subunit Activate Vibrio cholerae Biofilm Formation.
• Glucose-Specific Enzyme IIA Has Unique Binding Partners in The Vibrio cholerae Biofilm.
• The bacterial biofilm matrix as a platform for protein delivery.
• Spatially selective colonization of the arthropod intestine through activation of Vibrio cholerae biofilm formation.
• A communal bacterial adhesin anchors biofilm and bystander cells to surfaces.
• The phosphoenolpyruvate phosphotransferase system regulates Vibrio cholerae biofilm formation through multiple independent pathways.
• Vibrio cholerae phosphoenolpyruvate phosphotransferase system control of carbohydrate transport, biofilm formation, and colonization of the germfree mouse intestine.
• Genetic analysis of Vibrio cholerae monolayer formation reveals a key role for DeltaPsi in the transition to permanent attachment.
• A novel role for enzyme I of the Vibrio cholerae phosphoenolpyruvate phosphotransferase system in regulation of growth in a biofilm.
• NspS, a predicted polyamine sensor, mediates activation of Vibrio cholerae biofilm formation by norspermidine.
• Vibrio cholerae infection of Drosophila melanogaster mimics the human disease cholera.
• Role for glycine betaine transport in Vibrio cholerae osmoadaptation and biofilm formation within microbial communities.
• The Vibrio cholerae O139 O-antigen polysaccharide is essential for Ca2+-dependent biofilm development in sea water.
• Role of ectoine in Vibrio cholerae osmoadaptation.
• Environmental determinants of Vibrio cholerae biofilm development.
• Paula I Watnick--elucidating the role of biofilms. Interview by Pam Das.
• Signals, regulatory networks, and materials that build and break bacterial biofilms.
• Genetic analysis of Drosophila melanogaster susceptibility to intestinal Vibrio cholerae infection.
• A high-throughput screen identifies a new natural product with broad-spectrum antibacterial activity.
• Genetic evidence that the Vibrio cholerae monolayer is a distinct stage in biofilm development.
• Identification and characterization of a Vibrio cholerae gene, mbaA, involved in maintenance of biofilm architecture.
• Vibrio cholerae CytR is a repressor of biofilm development.
• Identification of novel stage-specific genetic requirements through whole genome transcription profiling of Vibrio cholerae biofilm development.
• The absence of a flagellum leads to altered colony morphology, biofilm development and virulence in Vibrio cholerae O139.
• Biofilm, city of microbes.
• Vibrio cholerae VibF is required for vibriobactin synthesis and is a member of the family of nonribosomal peptide synthetases.
• Steps in the development of a Vibrio cholerae El Tor biofilm.
• Genetic approaches to study of biofilms.
• A role for the mannose-sensitive hemagglutinin in biofilm formation by Vibrio cholerae El Tor.
• The interaction of the Vibrio cholerae transcription factors, Fur and IrgB, with the overlapping promoters of two virulence genes, irgA and irgB.
• Purification of Vibrio cholerae fur and estimation of its intracellular abundance by antibody sandwich enzyme-linked immunosorbent assay.
• Characterization of the transverse relaxation rates in lipid bilayers.
• Hydrophobic mismatch in gramicidin A'/lecithin systems.

Schools

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• Yale University

• Beth Israel Hospital

• Mass Genl Hospital Harvard Med School

Doctors Specialties

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• Infectious Disease Medicine
• Infectious Disease

Accepted Insurances

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Education

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• Massachusetts General Hospital
• Beth Israel Deaconess Medical Center
• Yale University School of Medicine

Hospital

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• Boston Children's Hospital

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