Associate Professor, Medicine, Indiana University School of Medicine
Nephrology
Dr. Pierre Dagher, MD
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Pierre Dagher works in the field of Nephrology. He researched medicine at NYU School of Medicine. He received awards:"Fellow (ASNF)". Dr. Pierre Dagher is also published. He has 41 publications published. The lastest publication: "Modulation of chloride secretion in the rat ileum by intracellular bicarbonate.' He accepts Medicare.
Publications
- Tamm-Horsfall Protein Regulates Granulopoiesis and Systemic Neutrophil Homeostasis.
- Ferroxitosis: a cell death from modulation of oxidative phosphorylation and PKM2-dependent glycolysis in melanoma.
- How the Innate Immune System Senses Trouble and Causes Trouble.
- Sisters in arms: Myeloid and tubular epithelial cells shape Renal innate immunity.
- p53 Is Renoprotective after ischemic Kidney injury by reducing inflammation.
- TNF-alpha mediates obstruction-induced Renal tubular cell apoptosis and proapoptotic signaling.
- Guanine nucleosides and Jurkat cell death: roles of ATP depletion and accumulation of deoxyribonucleotides.
- Guanosine supplementation reduces apoptosis and protects Renal function in the setting of ischemic injury.
- Modeling ischemia in vitro: selective depletion of adenine and guanine nucleotide pools.
- Modulation of chloride secretion in the rat ileum by intracellular bicarbonate.
- Short-chain fatty acids inhibit cAMP-mediated chloride secretion in rat colon.
- Acid-base effects on colonic electrolyte transport revisited.
- Dissociation of colonic apical Na/H exchange activity from bulk cytoplasmic pH.
- Effect of intracellular acidification on colonic NaCl absorption.
- Modulation of secretagogue-induced chloride secretion by intracellular bicarbonate.
- Mechanism of bicarbonate secretion in rat (Rattus rattus) colon.
- Modulation of chloride secretion in the rat colon by intracellular bicarbonate.
- Minocycline inhibits apoptosis and inflammation in a rat model of ischemic Renal injury.
- Unilateral ureteral obstruction induces Renal tubular cell production of tumor necrosis factor-alpha independent of inflammatory cell infiltration.
- Tumor necrosis factor-alpha induces intrinsic apoptotic signaling during Renal obstruction through truncated bid activation.
- Pathways of Renal injury in systemic gram-negative sepsis.
- The p53 inhibitor pifithrin-? can stimulate fibrosis in a rat model of ischemic Acute kidney injury.
- Endotoxin uptake by S1 proximal tubular segment causes oxidative stress in the downstream S2 segment.
- Fueling the fire in Acute kidney injury: endothelial cells collect their Toll.
- Tamm-Horsfall protein-deficient thick ascending limbs promote injury to neighboring S3 segments in an MIP-2-dependent mechanism.
- siRNA targeted to p53 attenuates ischemic and cisplatin-induced Acute kidney injury.
- p53 regulates Renal expression of HIF-1{alpha} and pVHL under physiological conditions and after ischemia-reperfusion injury.
- Tamm-Horsfall protein protects the Kidney from ischemic injury by decreasing inflammation and altering TLR4 expression.
- An expanding role of Toll-like receptors in sepsis-induced Acute kidney injury.
- Sepsis induces an increase in thick ascending limb Cox-2 that is TLR4 dependent.
- Renal Toll-like receptors: recent advances and implications for disease.
- Up-regulation of AMP-activated kinase by dysfunctional cystic fibrosis transmembrane conductance regulator in cystic fibrosis airway epithelial cells mitigates excessi...
- Sepsis induces changes in the expression and distribution of Toll-like receptor 4 in the rat Kidney.
- Minocycline reduces Renal microvascular leakage in a rat model of ischemic Renal injury.
- Apoptosis in ischemic Renal injury: roles of GTP depletion and p53.
- Newly developed techniques to study and diagnose Acute renal failure.
- A novel method to determine specificity and sensitivity of the TUNEL reaction in the quantitation of apoptosis.
- Rho GTPases show differential sensitivity to nucleotide triphosphate depletion in a model of ischemic cell injury.
- P53 mediates the apoptotic response to GTP depletion after Renal ischemia-reperfusion: protective role of a p53 inhibitor.
- Functional studies of the Kidney of living animals using multicolor two-photon microscopy.
Schools
University At Buffalo State University Of New York School Of Medicine
Nyu Hospitals Center
Procedures Preformed
- Hemodialysis
- Peritoneal Dialysis
Conditions Treated
- Acidosis
- Acute Kidney Failure
- Calcium Metabolism Disorders
- Chronic Kidney Diseases
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Doctors Specialties
- Nephrology
Accepted Insurances
Awards
- Fellow (ASNF)
Education
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NYU School of Medicine -
American University of Beirut Faculty of Medicine
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1467567677 |
| Last Name Of The Provider | DAGHER |
| First Name Of The Provider | PIERRE |
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