Glaucoma specialist at Texan Eye
Ophthalmology
Dr. Robert E. Marquis, MD
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Robert Marquis is an expert in the field of Ophthalmology. He studied medicine at University of Texas Southwestern Medical School. He received 1 awards: "Fellow (FAAO)". Robert E. Marquis, MD is also published. He has 70 publications published. The lastest publication: "Inactivation of enzymes within spores of Bacillus megaterium ATCC 19213 by hydroperoxides.'
Publications
- Mutation of NADH oxidase (nox) reveals an overlap of the oxygen- and acid-meditated stress responses in Streptococcus mutans.
- Human common salivary protein 1 (CSP-1) promotes binding of Streptococcus mutans to experimental salivary pellicle and glucans formed on hydroxyapatite surface.
- Protocols to study the physiology of oral biofilms.
- Characterization of UV-peroxide killing of bacterial spores.
- Repressed respiration of oral streptococci grown in biofilms.
- Free radical reactions and the inhibitory and lethal actions of high-pressure gases.
- In vitro effects of low intensity direct current generated silver on eukaryotic cells.
- The selectivity of the hair cell's mechanoelectrical-transduction channel promotes Ca2+ flux at low Ca2+ concentrations.
- Effects of extracellular Ca2+ concentration on hair-bundle stiffness and gating-spring integrity in hair cells.
- Arginine deiminase system and bacterial adaptation to acid environments.
- Inhibitory and cidal antimicrobial actions of electrically generated silver ions.
- Acid tolerance, proton permeabilities, and membrane ATPases of oral streptococci.
- Diminished acid tolerance of plaque bacteria caused by fluoride.
- Membrane-associated and solubilized ATPases of Streptococcus mutans and Streptococcus sanguis.
- Oxygen metabolism by Treponema denticola.
- Cloning and expression in Escherichia coli of the genes of the arginine deiminase system of Streptococcus sanguis NCTC 10904.
- Molecular mechanisms of resistance to heat and oxidative damage.
- Inhibition of streptococcal growth, F-ATPase and pyrophosphatase by diphosphonates.
- Sporicidal action of peracetic acid and protective effects of transition metal ions.
- Inhibition of proton-translocating ATPases of Streptococcus mutans and Lactobacillus casei by fluoride and aluminum.
- Fluoride inhibition of proton-translocating ATPases of oral bacteria.
- Membrane ATPases and acid tolerance of Actinomyces viscosus and Lactobacillus casei.
- Role of the arginine deiminase system in protecting oral bacteria and an enzymatic basis for acid tolerance.
- Thermophysiology of Streptococcus mutans and related lactic-acid bacteria.
- Comparative acid tolerances and inhibitor sensitivities of isolated F-ATPases of oral lactic acid bacteria.
- Irreversible paraben inhibition of glycolysis by Streptococcus mutans GS-5.
- Microscopic and thermal characterization of hydrogen peroxide killing and lysis of spores and protection by transition metal ions, chelators, and antioxidants.
- Fluoride, triclosan and organic weak acids as modulators of the arginine deiminase system in biofilms and suspension cells of oral streptococci.
- Oxygen metabolism, oxidative stress and acid-base physiology of dental plaque biofilms.
- Arginine deiminase system and acid adaptation of oral streptococci.
- Catabolite modification of acid tolerance of Streptococcus mutans GS-5.
- Biofilm acid/base physiology and gene expression in oral bacteria.
- Rapid procedure for acid adaptation of oral lactic-acid bacteria and further characterization of the response.
- Mechanisms of inhibition by fluoride of urease activities of cell suspensions and biofilms of Staphylococcus epidermidis, Streptococcus salivarius, Actinomyces naeslun...
- Anti-microbial efficacy and mode of action studies on a new zinc/ Triclosan formulation.
- Effects of apigenin and tt-farnesol on glucosyltransferase activity, biofilm viability and caries development in rats.
- Adaptation of Streptococcus mutans and Enterococcus hirae to acid stress in continuous culture.
- Anaerobic killing of oral streptococci by reduced, transition metal cations.
- Antimicrobial actions of fluoride for oral bacteria.
- Physiologic homeostasis and stress responses in oral biofilms.
- Mineralization and responses of bacterial spores to heat and oxidative agents.
- Quasi-irreversible inhibition of enolase of Streptococcus mutans by fluoride.
- Inactivation of enzymes within spores of Bacillus megaterium ATCC 19213 by hydroperoxides.
- Membrane locus and pH sensitivity of paraben inhibition of alkali production by oral streptococci.
- Fluoride and organic weak acids as respiration inhibitors for oral streptococci in acidified environments.
- Physiologic actions of zinc related to inhibition of acid and alkali production by oral streptococci in suspensions and biofilms.
- Antimicrobial actions of benzimidazoles against oral streptococci.
- Weak acid effects and fluoride inhibition of glycolysis by Streptococcus mutans GS-5.
- Sensitization of Actinomyces naeslundii and Streptococcus sanguis in biofilms and suspensions to acid damage by fluoride and Other weak acids.
- Method to sensitize bacterial spores to subsequent killing by dry heat or ultraviolet irradiation.
- Selective sensitization of bacteria to peroxide damage associated with fluoride inhibition of catalase and pseudocatalase.
- Turning on and turning off the arginine deiminase system in oral streptococci.
- Alkali production by oral bacteria and protection against dental caries.
- Shifts in membrane fatty acid profiles associated with acid adaptation of Streptococcus mutans.
- Acid adaptation in Streptococcus mutans UA159 alleviates sensitization to environmental stress due to RecA deficiency.
- The influence of a novel propolis on mutans streptococci biofilms and caries development in rats.
- Targets for hydrogen-peroxide-induced damage to suspension and biofilm cells of Streptococcus mutans.
- Malolactic fermentation by Streptococcus mutans.
- Triclosan inhibition of membrane enzymes and glycolysis of Streptococcus mutans in suspensions and biofilms.
- Enhanced acid resistance of oral streptococci at lethal pH values associated with acid-tolerant catabolism and with ATP synthase activity.
- Antimicrobial actions of benzimidazoles against the oral anaerobes Fusobacterium nucleatum and Prevotella intermedia.
- Co-operative inhibition by fluoride and zinc of glucosyl transferase production and polysaccharide synthesis by mutans streptococci in suspension cultures and biofilms.
- Multi-target antimicrobial actions of zinc against oral anaerobes.
- Applied and ecological aspects of oxidative-stress damage to bacterial spores and to oral microbes.
- Management of Glaucoma: focus on pharmacological therapy.
- Antimicrobial effects of o-cymen-5-ol and zinc, alone & in combination in simple solutions and toothpaste formulations.
- Antimicrobial actions of α-mangostin against oral streptococci.
- Fluoride and organic weak acids as modulators of microbial physiology.
- Alkali production associated with malolactic fermentation by oral streptococci and protection against acid, oxidative, or starvation damage.
Procedures Preformed
- Cataract Removal Surgery
- Glaucoma Surgery
Conditions Treated
- Aphakia and Other Disorders of Lens
- Benign Neoplasm of Choroid
- Benign Neoplasm of Eye
- Cataract
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Doctors Specialties
- Ophthalmology
Accepted Insurances
Awards
- Fellow (FAAO)
Education
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University of Texas Southwestern Medical School
Hospital
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Seton Medical Center Austin
Drug Facts
| NPI NUMBER | 1902881048 | |
| NPPES Provider LastName | MARQUIS | |
| NPPES Provider FirstName | ROBERT | |
| NPPES Provider ZIPCode | 787314203 | |
| NPPES Provider State | TX | |
| Specialty Description | Ophthalmology | |
| Total Claim Count | 4379.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1902881048 |
| Last Name Of The Provider | MARQUIS |
| First Name Of The Provider | ROBERT |
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