Professor, Surgery, Case Western Reserve University School of Medicine
Orthopaedic Surgery
Dr. Robert T. Ballock, MD
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Robert Ballock practices Orthopaedic Surgery. He graduated from Harvard Medical School. Harvard Medical School is a top medical college. Harvard Medical School is ranked 1 in research and 12 in primary care medicine Robert Ballock is a published MD as well. He has 47 publications published. The latest was: Expression pattern of two Frizzled-related genes, Frzb-1 and Sfrp-1, during mouse embryogenesis suggests a role for modulating action of Wnt family members.
Publications
- A comparison of early versus late conversion from intravenous to oral therapy in the treatment of septic arthritis.
- Carboxypeptidase Z (CPZ) links thyroid hormone and Wnt signaling pathways in growth plate chondrocytes.
- Thyroid hormone interacts with the Wnt/beta-catenin signaling pathway in the terminal differentiation of growth plate chondrocytes.
- Closed reduction for treatment of developmental dysplasia of the hip in children.
- Peroxisome Proliferator-Activated Receptor-gamma Promotes Adipogenic Changes in Growth Plate Chondrocytes In Vitro.
- Thyroid hormone and the growth plate.
- Analysis of gene expression in mineralized skeletal tissues by laser capture microdissection and RT-PCR.
- Peroxisome proliferator activated receptor-gamma (PPARgamma) represses thyroid hormone signaling in growth plate chondrocytes.
- Expression and activation of peroxisome proliferator-activated receptors in growth plate chondrocytes.
- High incidence of hip dysplasia but not slipped capital femoral epiphysis in patients with Prader-Willi syndrome.
- Expression and activity of the CDK inhibitor p57Kip2 in chondrocytes undergoing hypertrophic differentiation.
- Analyzing bone, blood vessels, and biomaterials with microcomputed tomography.
- Physiology and pathophysiology of the growth plate.
- Second cervical vertebrae pedicle Fractures versus synchondrosis in a child.
- The biology of the growth plate.
- Molecular and genetic approaches to musculoskeletal diseases.
- Sternoclavicular joint ganglion cysts in young children.
- Human disease-causing NOG missense mutations: effects on noggin secretion, dimer formation, and bone morphogenetic protein binding.
- Both retinoic acid and 1,25(OH)2 vitamin D3 inhibit thyroid hormone-induced terminal differentiaton of growth plate chondrocytes.
- Expression of cyclin-dependent kinase inhibitors in epiphyseal chondrocytes induced to terminally differentiate with thyroid hormone.
- Salter-Harris type II Fractures of the capital femoral epiphysis.
- Compartment syndrome in ipsilateral humerus and forearm Fractures in children.
- Bizarre parosteal osteochondromatous proliferation of bone (BPOP): an unusual Foot mass in a child.
- Oral antibiotic therapy of bacterial arthritis.
- Expression of thyroid hormone receptor isoforms in rat growth plate cartilage in vivo.
- Cell cycle genes in chondrocyte proliferation and differentiation.
- Expression pattern of two Frizzled-related genes, Frzb-1 and Sfrp-1, during mouse embryogenesis suggests a role for modulating action of Wnt family members.
- Hemihypertrophy. Concepts and controversies.
- Regulation of the expression of the type-II collagen gene in periosteum-derived cells by three members of the transforming growth factor-beta superfamily.
- The prevalence of nonmuscular causes of torticollis in children.
- Length dependence of a halo orthosis on cervical immobilization.
- TGF-beta 1 prevents hypertrophy of epiphyseal chondrocytes: regulation of gene expression for cartilage matrix proteins and metalloproteases.
- Inhibition of the chondrocyte phenotype by retinoic acid involves upregulation of metalloprotease genes independent of TGF-beta.
- Role for autocrine TGF-beta 1 in regulating differentiation of a human leukemic cell line toward osteoclast-like cells.
- Thyroxine is the serum factor that regulates morphogenesis of columnar cartilage from isolated chondrocytes in chemically defined medium.
- Suboptimal (thin) distal cement mantle thickness as a contributory factor in total hip arthroplasty femoral component failure. A retrospective radiographic analysis fa...
- Growth factor modulation of the formation of a molded vascularized bone graft in vivo.
- Structural behavior of the halo orthosis pin-bone interface: biomechanical evaluation of standard and newly designed stainless steel halo fixation pins.
- The quality of magnetic resonance imaging, as affected by the composition of the halo orthosis. A brief note.
- The effect of angled insertion on halo pin fixation.
- Use of patellar Tendon autograft for anterior cruciate ligament reconstruction in the rabbit: a long-term histologic and biomechanical study.
- The effect of pin location on the rigidity of the halo pin-bone interface.
- Can burst Fractures be predicted from plain radiographs?
- Primary cilia modulate Ihh signal transduction in response to hydrostatic loading of growth plate chondrocytes.
- Leptin synergizes with thyroid hormone signaling in promoting growth plate chondrocyte proliferation and terminal differentiation in vitro.
- Thyroid hormone-mediated growth and differentiation of growth plate chondrocytes involves IGF-1 modulation of beta-catenin signaling.
Schools
Harvard Medical School
Tx Scottish Rite Hospital Chldn
University Ca San Diego Med Center
Procedures Preformed
- Achilles Tenotomy
- Dressing and-or Debridement of Wound, Infection, or Burn (incl. Negative Pressure Wound Therapy)
- Elbow Fracture and-or Dislocation Treatment, Closed
- Elbow Fracture and-or Dislocation Treatment, Open
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Conditions Treated
- Achilles Tendinitis
- Ankle Fracture
- Ankle Sprain and Achilles Tendon Sprain or Rupture
- Anterior Cruciate Ligament (ACL) or Posterior Cruciate Ligament (PCL) Tear
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Doctors Specialties
- Orthopedic Surgery
- Orthopaedic Surgery
Accepted Insurances
Education
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University of California San Diego -
Harvard Medical School
Hospital
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Cleveland Clinic
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