Professor, Medicine, University of Louisville School of Medicine
Cardiology
Dr. Roberto Bolli, MD
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Roberto Bolli practices Cardiology. He went to Baylor College of Medicine. He received 2 awards: "Elected Member" and "Fellow (FACC)". Dr. Roberto Bolli is also a published doctor. He has 99 publication published. The most recent publication is: Concise Review: Review and Perspective of Cell Dosage and Routes of Administration From Preclinical and Clinical Studies of Stem Cell Therapy for Heart Disease. He accepts Medicare payments and is listed with Medicare.gov.
Publications
- Concise Review: Review and Perspective of Cell Dosage and Routes of Administration From Preclinical and Clinical Studies of Stem Cell Therapy for Heart Disease.
- Current status of cell therapy for non-ischaemic Cardiomyopathy: a brief overview.
- Cell Therapy Needs Rigorous Translational Studies in Large Animal Models.
- Identification of Heme Oxygenase 1 (HO-1) as a Novel Negative Regulator of Mobilization of Hematopoietic Stem/Progenitor Cells.
- Cardiac stem cell therapy for cardiac repair.
- Bone marrow mononuclear cell therapy for Acute myocardial infarction: a perspective from the cardiovascular cell therapy research network.
- Mechanisms of stem cell effects: insights from MRI.
- Vascular endothelial growth factor in Heart failure.
- IntraCoronary delivery of autologous cardiac stem cells improves cardiac function in a porcine model of chronic ischemic cardiomyopathy.
- A highly sensitive and accurate method to quantify absolute numbers of c-kit+ cardiac stem cells following transplantation in mice.
- Phase II Clinical Research Design in Cardiology: Learning the Right Lessons Too Well: Observations and Recommendations From the Cardiovascular Cell Therapy Research Ne...
- Stem cell therapy: promising treatment in Heart failure?
- Protein O-GlcNAcylation is a Novel Cytoprotective Signal in Cardiac Stem Cells.
- Genetic background, gender, age, body temperature, and arterial blood pH have a major impact on Myocardial infarct size in the mouse and need to be carefully measured ...
- The COX-2/PGI2 Receptor Axis Plays an Obligatory Role in Mediating the Cardioprotection Conferred by the Late Phase of Ischemic Preconditioning.
- Targeting phosphatidylinositol 3-kinase-Akt through hepatocyte growth factor for cardioprotection.
- Oxidant species trigger late preconditioning against Myocardial stunning in conscious rabbits.
- Regeneration of the human Heart--no chimera?
- Ischemic preconditioning upregulates inducible nitric oxide synthase in cardiac myocyte.
- Nitric oxide (NO) induces nitration of protein kinase Cepsilon (PKCepsilon ), facilitating PKCepsilon translocation via enhanced PKCepsilon -RACK2 interactions: a nove...
- Formation of protein kinase C(epsilon)-Lck signaling modules confers cardioprotection.
- Mitochondrial PKCepsilon and MAPK form signaling modules in the murine Heart: enhanced mitochondrial PKCepsilon-MAPK interactions and differential MAPK activation in P...
- Inducible nitric oxide synthase modulates cyclooxygenase-2 activity in the Heart of conscious rabbits during the late phase of ischemic preconditioning.
- Expression of activated PKC epsilon (PKC epsilon) protects the ischemic Heart, without attenuating ischemic H(+) production.
- Role of nitric oxide in Myocardial preconditioning.
- Role of cyclic guanosine monophosphate in late preconditioning in conscious rabbits.
- Role of Src protein tyrosine kinases in late preconditioning against Myocardial infarction.
- Gene dosage-dependent effects of cardiac-specific overexpression of the A3 adenosine receptor.
- Discovery of a new function of cyclooxygenase (COX)-2: COX-2 is a cardioprotective protein that alleviates ischemia/reperfusion injury and mediates the late phase of p...
- Aldose reductase is an obligatory mediator of the late phase of ischemic preconditioning.
- Delta-opioid receptor-induced late preconditioning is mediated by cyclooxygenase-2 in conscious rabbits.
- COX-2-derived prostacyclin mediates opioid-induced late phase of preconditioning in isolated rat Hearts.
- Protein tyrosine kinase signaling is necessary for NO donor-induced late preconditioning against Myocardial stunning.
- Role of the JAK-STAT pathway in protection against Myocardial ischemia/reperfusion injury.
- MCC-134, a single pharmacophore, opens surface ATP-sensitive potassium channels, blocks mitochondrial ATP-sensitive potassium channels, and suppresses preconditioning.
- Protein kinase Cepsilon interacts with and inhibits the permeability transition pore in cardiac mitochondria.
- Use of fractional flow reserve versus stress perfusion scintigraphy after unstable Angina. Effect on duration of hospitalization, cost, procedural characteristics, and...
- Effect of aspirin on late preconditioning against Myocardial stunning in conscious rabbits.
- The role of sodium-hydrogen ion exchange in patients undergoing Coronary artery bypass grafting.
- Gene therapy with inducible nitric oxide synthase protects against Myocardial infarction via a cyclooxygenase-2-dependent mechanism.
- Mechanism of cyclooxygenase-2 upregulation in late preconditioning.
- Cardioprotection involves activation of NF-kappa B via PKC-dependent tyrosine and serine phosphorylation of I kappa B-alpha.
- Nonelectrocardiographic evidence that both ischemic preconditioning and adenosine preconditioning exist in humans.
- Impact of sodium-hydrogen exchange inhibition by cariporide on death or Myocardial infarction in high-risk CABG surgery patients: results of the CABG surgery cohort of...
- Nicorandil induces late preconditioning against Myocardial infarction in conscious rabbits.
- Gender and aging do not impair opioid-induced late preconditioning in rats.
- Administration of a CO-releasing molecule at the time of reperfusion reduces infarct size in vivo.
- New perspectives on vitamin E: gamma-tocopherol and carboxyelthylhydroxychroman metabolites in biology and medicine.
- Cardioprotection during the final stage of the late phase of ischemic preconditioning is mediated by neuronal NO synthase in concert with cyclooxygenase-2.
- Bmx, a member of the Tec family of nonreceptor tyrosine kinases, is a novel participant in pharmacological cardioprotection.
- Molecular genetic advances in Cardiovascular medicine: focus on the myocyte.
- Tumor necrosis factor-alpha does not modulate ischemia/reperfusion injury in naïve myocardium but is essential for the development of late preconditioning.
- Myocardial protection at a crossroads: the need for translation into clinical therapy.
- IL-6 plays an obligatory role in late preconditioning via JAK-STAT signaling and upregulation of iNOS and COX-2.
- Inotrope use at separation from cardioPulmonary bypass and the role of prebypass TEE.
- Hypercholesterolemia blunts NO donor-induced late preconditioning against Myocardial infarction in conscious rabbits.
- Delayed adaptation of the Heart to stress: late preconditioning.
- Overexpression of mitogen-activated protein kinase kinase 6 in the Heart improves functional recovery from ischemia in vitro and protects against myocardial infarction...
- Cells expressing early cardiac markers reside in the bone marrow and are mobilized into the peripheral blood after Myocardial infarction.
- Prostacyclin attenuates oxidative damage of myocytes by opening mitochondrial ATP-sensitive K+ channels via the EP3 receptor.
- Administration of a CO-releasing molecule induces late preconditioning against Myocardial infarction.
- Bone marrow cells for cardiac regeneration: the quest for the protagonist continues.
- Cardiac stem cells delivered intravascularly traverse the vessel barrier, regenerate infarcted myocardium, and improve cardiac function.
- Proapoptotic effects of caspase-1/interleukin-converting enzyme dominate in Myocardial ischemia.
- Short-term caloric restriction improves ischemic tolerance independent of opening of ATP-sensitive K+ channels in both young and aged Hearts.
- Delayed preconditioning-mimetic actions of nitroglycerin in patients undergoing exercise tolerance tests.
- Myocardial regeneration by activation of multipotent cardiac stem cells in ischemic Heart failure.
- Late preconditioning induced by NO donors, adenosine A1 receptor agonists, and delta1-opioid receptor agonists is mediated by iNOS.
- HO-1 induction by HIF-1: a new mechanism for delayed cardioprotection?
- Cardiac preconditioning during percutaneous Coronary interventions.
- Gene therapy with iNOS provides long-term protection against Myocardial infarction without adverse functional consequences.
- Role of the protein kinase C-epsilon-Raf-1-MEK-1/2-p44/42 MAPK signaling cascade in the activation of signal transducers and activators of transcription 1 and 3 and in...
- Hypercholesterolemia abrogates late preconditioning via a tetrahydrobiopterin-dependent mechanism in conscious rabbits.
- Myocardial aging--a stem cell problem.
- Adult bone marrow-derived cells: regenerative potential, plasticity, and tissue commitment.
- Bone marrow cell-mediated cardiac regeneration a veritable revolution.
- Foreword--focused issue on cardiac repair by stem cells.
- The ubiquitous role of nitric oxide in cardioprotection.
- Cardiac progenitor cells: the revolution continues.
- Endothelial progenitor cells: neovascularization or more?
- No pain, no gain: the useful function of Angina.
- Cause of death: suicide.
- Life and death of cardiac stem cells: a paradigm shift in cardiac biology.
- Postinfarct cytokine therapy regenerates cardiac tissue and improves left ventricular function.
- Stem cell niches in the Adult mouse heart.
- Cardioprotection by postconditioning in conscious rats is limited to Coronary occlusions <45 min.
- Sca-1 expression is associated with decreased cardiomyogenic differentiation potential of skeletal muscle-derived Adult primitive cells.
- Preconditioning: a paradigm shift in the biology of Myocardial ischemia.
- Effects of anesthesia on echocardiographic assessment of left ventricular structure and function in rats.
- The telomere-telomerase axis and the Heart.
- Increasing evidence that estrogen is an important modulator of bone marrow-mediated cardiac repair after Acute infarction.
- Concise review: stem cells, Myocardial regeneration, and methodological artifacts.
- Bone marrow for cardiac repair: the importance of characterizing the phenotype and function of injected cells.
- Pretreatment with intraCoronary enalaprilat protects human myocardium during percutaneous Coronary angioplasty.
- The late phase of ischemic preconditioning induces a prosurvival genetic program that results in marked attenuation of apoptosis.
- Adult bone marrow-derived cells for cardiac repair: a systematic review and meta-analysis.
- The late phase of preconditioning and its natural clinical application--gene therapy.
- Endothelial nitric oxide synthase plays an obligatory role in the late phase of ischemic preconditioning by activating the protein kinase C epsilon p44/42 mitogen-acti...
Schools
University Of Perugia
National Institute Of Health
Baylor University Medical Center
Procedures Preformed
- Ablation for Treatment of Cardiac Arrhythmias
- Cardiac Catheterization (incl. Coronary Angiography)
- Cardiac Imaging
- Cardiac MRI (Magnetic Resonance Imaging) of Heart or Chest
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Conditions Treated
- Adult Congenital Heart Disease
- Aneurysm and Dissection of Heart
- Angina and Acute Coronary Syndrome
- Aortic Stenosis
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Doctors Specialties
- Cardiology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Pathway X Bronze Direct CACA IN HIX
- Pathway X Ind Direct Access KY
- Anthem Blue Cross Blue Shield HMO
- Anthem Blue Cross Blue Shield PPO
- Blue Card PPO
- Blue Cross Blue Shield of Illinois PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- First Health (Coventry Health Care) PPO
- Choice Care PPO
- Preferred PPO
- Medicaid
- Medicare MCR
- Sagamore Plus
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Awards
- Elected Member
- Fellow (FACC)
Education
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Baylor College of Medicine -
University of Perugia Faculty of Medicine
Drug Facts
| NPI NUMBER | 1306846696 | |
| NPPES Provider LastName | BOLLI | |
| NPPES Provider FirstName | ROBERTO | |
| NPPES Provider ZIPCode | 402023841 | |
| NPPES Provider State | KY | |
| Specialty Description | Cardiology | |
| Total Claim Count | 14.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1306846696 |
| Last Name Of The Provider | BOLLI |
| First Name Of The Provider | ROBERTO |
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