Instructor in Medicine, Massachusetts General Hospital
Internal Medicine
Dr. Ryan B. Corcoran, MD
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Doctor Ryan B. Corcoran, MD is an expert in the field of Internal Medicine. He attended Stanford University School of Medicine. Stanford University School of Medicine is a top medical college. Stanford University School of Medicine has a rank of 2 in research and a 25 in primary care Ryan B. Corcoran, MD is a published MD. He published 17 publications, including: 'Tumor Heterogeneity and Lesion-Specific Response to Targeted Therapy in Colorectal Cancer.' He accepts Medicare payments and is registered with Medicare.gov.
Publications
- Clinical Acquired Resistance to RAF Inhibitor Combinations in BRAF-Mutant Colorectal Cancer through MAPK Pathway Alterations.
- Combined MEK and PI3K inhibition in a mouse model of pancreatic cancer.
- mTOR Inhibition Specifically Sensitizes Colorectal Cancers with KRAS or BRAF Mutations to BCL-2/BCL-XL Inhibition by Suppressing MCL-1.
- TORC1 Suppression Predicts Responsiveness to RAF and MEK Inhibition in BRAF-Mutant Melanoma.
- Synthetic Lethal Interaction of Combined BCL-XL and MEK Inhibition Promotes Tumor Regressions in KRAS Mutant Cancer Models.
- EGFR-mediated re-activation of MAPK signaling contributes to insensitivity of BRAF mutant colorectal cancers to RAF inhibition with vemurafenib.
- Receptor tyrosine kinases exert dominant control over PI3K signaling in human KRAS mutant colorectal cancers.
- BIM expression in treatment-naive cancers predicts responsiveness to kinase inhibitors.
- STAT3 plays a critical role in KRAS-induced pancreatic tumorigenesis.
- Potential Therapeutic Strategies to Overcome Acquired Resistance to BRAF or MEK Inhibitors in BRAF Mutant Cancers.
- The PI3K pathway as drug target in human cancer.
- BRAF gene amplification can promote acquired resistance to MEK inhibitors in cancer Cells harboring the BRAF V600E mutation.
- Hedgehog signal transduction by Smoothened: pharmacologic evidence for a 2-step activation process.
- Insulin-like growth factor 2 is required for progression to advanced medulloblastoma in patched1 heterozygous mice.
- Oxysterols stimulate Sonic hedgehog signal transduction and proliferation of medulloblastoma Cells.
- Tumor Heterogeneity and Lesion-Specific Response to Targeted Therapy in Colorectal Cancer.
Schools
Stanford University
Massachusetts General Hospital
Conditions Treated
- Anemia
- Coagulation Disorders (incl. Hemophilia)
- Deep Vein Thrombosis (DVT)
- Hodgkin's Disease
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Doctors Specialties
- Hematology
- Internal Medicine
Accepted Insurances
- Blue Card PPO
- BlueConnect HMO
- Blue with Managed Care Behavioral Health HMO
- Preferred Blue PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- GHI PPO
- First Health (Coventry Health Care) PPO
- Choice Care PPO
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- Tufts PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Education
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Brigham and Women's Hospital -
Massachusetts General Hospital -
Stanford University School of Medicine
Hospital
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Massachusetts General Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1508993957 |
| Last Name Of The Provider | CORCORAN |
| First Name Of The Provider | RYAN |
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