Assistant Professor, Medicine, University of California, San Francisco, School of Medicine
Infectious Disease
Dr. Satish K. Pillai, MD
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Satish Pillai works in the field of Infectious Disease. He graduated from Case Western Reserve University School of Medicine. Satish Pillai is a published physician as well. He has 56 publications published. The latest was: Antiviral gene expression in psoriasis.
Publications
- Antiviral gene expression in psoriasis.
- Select host restriction factors are associated with HIV persistence during antiretroviral therapy.
- Dynamic regulation of host restriction factor expression over the course of HIV-1 infection in vivo.
- Decreased HIV-1 Transcription in CCR5 Delta-32 Heterozygotes On Suppressive Antiretroviral Therapy.
- The infectious diseases society of america emerging infections network: bridging the gap between clinical infectious diseases and public health.
- Heat illness: predictors of hospital admissions among emergency department visits-Georgia, 2002-2008.
- Effects of interferon-α treatment on anti-HIV-1 intrinsic immunity in vivo.
- Increased expression of intrinsic antiviral genes in HLA-B*57-positive individuals.
- Effects of Cellular Activation on Anti-HIV-1 Restriction Factor Expression Profile in Primary Cells.
- Challenges in Detecting HIV Persistence during Potentially Curative Interventions: A Study of the Berlin Patient.
- BST-2 is rapidly down-regulated from the cell surface by the HIV-1 protein Vpu: evidence for a post-ER mechanism of Vpu-action.
- Human Endogenous Retrovirus K106 (HERV-K106) Was Infectious after the Emergence of Anatomically Modern Humans.
- In vivo CD8+ T-cell suppression of siv viremia is not mediated by CTL clearance of productively infected cells.
- Cross-sectional dating of novel haplotypes of HERV-K 113 and HERV-K 115 indicate these proviruses originated in Africa before Homo sapiens.
- Development of reduced vancomycin susceptibility in methicillin-susceptible Staphylococcus aureus.
- Antistaphylococcal agents.
- Inferring HIV transmission dynamics from phylogenetic sequence relationships.
- Daptomycin nonsusceptibility in Staphylococcus aureus with reduced vancomycin susceptibility is independent of alterations in MprF.
- Lack of neutralizing antibody response to HIV-1 predisposes to superinfection.
- Emergence of a clinical daptomycin-resistant Staphylococcus aureus isolate during treatment of methicillin-resistant Staphylococcus aureus bacteremia and osteomyelitis.
- Codon volatility does not reflect selective pressure on the HIV-1 genome.
- Semen-specific genetic characteristics of human immunodeficiency virus type 1 env.
- Effects of glucose on fsr-mediated biofilm formation in Enterococcus faecalis.
- Linezolid resistance in sequential Staphylococcus aureus isolates associated with a T2500A mutation in the 23S rRNA gene and loss of a single copy of rRNA.
- Linezolid-resistant Enterococcus faecalis isolated from a cord blood transplant recipient.
- Linezolid-resistant, vancomycin-resistant Enterococcus faecium infection in patients without prior exposure to linezolid.
- Linezolid resistance in Staphylococcus aureus: characterization and stability of resistant phenotype.
- Role of MicroRNA Modulation in the Interferon-α/Ribavirin Suppression of HIV-1 In Vivo.
- Effect of cocaine on chemokine and CCR-5 gene expression by mononuclear cells from normal donors and HIV-1 infected patients.
- Structure and function of CC-chemokine receptor 5 homologues derived from representative primate species and subspecies of the taxonomic suborders Prosimii and Anthrop...
- Estimating selection pressures on HIV-1 using phylogenetic likelihood models.
- Single-strand specificity of APOBEC3G accounts for minus-strand deamination of the HIV genome.
- Broadly increased sensitivity to cytotoxic T lymphocytes resulting from Nef epitope escape mutations.
- Current V3 genotyping algorithms are inadequate for predicting X4 co-receptor usage in clinical isolates.
- Whole genome characterization of the mechanisms of daptomycin resistance in clinical and laboratory derived isolates of Staphylococcus aureus.
- The sequential introduction of HIV-1 subtype B and CRF01AE in Singapore by sexual transmission: accelerated V3 region evolution in a subpopulation of Asian CRF01 viruses.
- A new perspective on V3 phenotype prediction.
- Genetic composition of human immunodeficiency virus type 1 in cerebrospinal fluid and blood without treatment and during failing antiretroviral therapy.
- Rational peptide selection to detect human immunodeficiency virus type 1-specific T-cell responses under resource-limited conditions.
- Clinical rationale for treatment of endocarditis caused by methicillin-susceptible Staphylococcus aureus developing nonsusceptibility to daptomycin.
- Lack of bactericidal antagonism or synergism in vitro between oxacillin and vancomycin against methicillin-susceptible strains of Staphylococcus aureus.
- Latently-infected CD4+ T cells are enriched for HIV-1 Tat variants with impaired transactivation activity.
- Reduced susceptibility to vancomycin influences pathogenicity in Staphylococcus aureus infection.
- Maintenance of Nef-mediated modulation of major histocompatibility complex class I and CD4 after sexual transmission of human immunodeficiency virus type 1.
- Evaluation of endocarditis caused by methicillin-susceptible Staphylococcus aureus developing nonsusceptibility to daptomycin.
- Prevalence of the fsr locus in Enterococcus faecalis infections.
- Modulation of cellular protein trafficking by human immunodeficiency virus type 1 Nef: role of the acidic residue in the ExxxLL motif.
- Genetic attributes of cerebrospinal fluid-derived HIV-1 env.
- Persistence of rRNA operon mutated copies and rapid re-emergence of linezolid resistance in Staphylococcus aureus.
- Turning up the volume on mutational pressure: is more of a good thing always better? (A case study of HIV-1 Vif and APOBEC3).
- Pleocytosis is associated with disruption of HIV compartmentalization between blood and cerebral spinal fluid viral populations.
- HIV rebounds from latently infected cells, rather than from continuing low-level replication.
- Protease polymorphisms in HIV-1 subtype CRF01_AE represent selection by antiretroviral therapy and host immune pressure.
- Association between specific HIV-1 Env traits and virologic control in vivo.
- Interferon-alpha drives monocyte gene expression in chronic unsuppressed HIV-1 infection.
Schools
Case Western Reserve University School of Medicine
Doctors Specialties
- Infectious Disease Medicine
- Infectious Disease
Accepted Insurances
Education
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Beth Israel Deaconess Medical Center -
Case Western Reserve University - University Hospitals -
Case Western Reserve University School of Medicine
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