Wolpow Family Chair in IBD Treatment and Research, Director, IBD Center, Boston Children's Hospital, Brigham and Women's Hospital, Associate Professor of Medicine, Harvard Medical School
Gastroenterology
Dr. Scott B. Snapper, MD
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Doctor Scott B. Snapper, MD works in the field of Gastroenterology. He went to medical school at Albert Einstein College of Medicine of Yeshiva University. He over the years received 1 awards: "Elected Member". Doctor Scott B. Snapper, MD is also a published doctor. He has 49 paper published. The most recent paper is: O-018New Evidence for Structural Brain Differences in Pediatric Crohn's Disease: Impact of Underlying Disease Factors. Scott B. Snapper, MD is registered with Medicare and accepts Medicare payments.
Publications
- O-009 Lymphotoxin Links Microbiota and Group 3 Innate Lymphoid Cells to Protect Against Intestinal Inflammation.
- O-008 Aberrant Anti-inflammatory Macrophage Function and Differentiation in Wiskott-Aldrich Syndrome Protein-Deficient Mice and Humans.
- O-005 YI Microbiota Drives Inflammation by Altering Intestinal Lamina Propria Macrophage Phenotype in a Novel IL10R-Deficient Model of Very Early Onset IBD.
- O-018 New Evidence for Structural Brain Differences in Pediatric Crohn's Disease: Impact of Underlying Disease Factors.
- Individual Intestinal symbionts induce a distinct population of RORγ+ regulatory T cells.
- Large B-Cell Lymphoma in an Adolescent Patient with IL-10 Receptor Deficiency and History of Infantile Inflammatory Bowel Disease.
- CCL25/CCR9 Interactions Are Not Essential for Colitis Development but Are Required for Innate Immune Cell Protection from Chronic Experimental Murine Colitis.
- Interleukin-10 receptor signaling in innate immune cells regulates mucosal immune tolerance and anti-inflammatory macrophage function.
- The ability of an attaching and effacing pathogen to trigger localized actin assembly contributes to virulence by promoting mucosal attachment.
- Protective mucosal immunity mediated by epithelial CD1d and IL-10.
- N-wasp is essential for the negative regulation of B cell receptor signaling.
- Colitis and Colon Cancer in WASP-Deficient Mice Require Helicobacter Species.
- Elevated levels of leukotriene C4 synthase mRNA distinguish a subpopulation of eosinophilic oEsophagitis patients.
- IL-10R Polymorphisms Are Associated with Very-early-onset Ulcerative Colitis.
- EPEC and vaccinia virus do not require WIP family proteins for pathogen-induced actin assembly.
- Wiskott-Aldrich Syndrome Protein Deficiency in Innate Immune Cells Leads to Mucosal Immune Dysregulation and Colitis in Mice.
- Update on biologic pathways in inflammatory bowel Disease and their therapeutic relevance.
- Gut-tropic T Cells that Express Integrin ?4?7 and CCR9 are Required for Induction of Oral Immune Tolerance in Mice.
- Rac1 Drives Melanoblast Organization during Mouse Development by Orchestrating Pseudopod- Driven Motility and Cell-Cycle Progression.
- Decoding inflammatory bowel Disease through the lens of immunodeficiency.
- MyD88-Dependent TLR1/2 Signals Educate Dendritic Cells with Gut-Specific Imprinting Properties.
- Intestinal alkaline phosphatase has beneficial effects in mouse models of chronic colitis.
- N-WASP is required for membrane wrapping and myelination by Schwann cells.
- Activating WASP mutations associated with X-linked neutropenia result in enhanced actin polymerization, altered cytoskeletal responses, and genomic instability in lymp...
- Neural Wiskott-Aldrich Syndrome protein modulates Wnt signaling and is required for hair follicle cycling in mice.
- Enterohemorrhagic E. coli requires N-WASP for efficient type III translocation but not for EspFU-mediated actin pedestal formation.
- Inflammatory bowel Disease and mutations affecting the interleukin-10 receptor.
- Toll-like receptor 4-mediated regulation of spontaneous Helicobacter-dependent colitis in IL-10-deficient mice.
- Phosphorylation of WASp is a key regulator of activity and stability in vivo.
- Activating mutations of N-WASP alter Shigella pathogenesis.
- Targeting Smads to restore transforming growth factor-beta signaling and regulatory T-cell function in inflammatory bowel Disease.
- WASP confers selective advantage for specific hematopoietic cell populations and serves a unique role in marginal zone B-cell homeostasis and function.
- Regulatory T cells in inflammatory bowel Disease.
- Challenges in IBD research: Assessing progress and rethinking the research agenda.
- Lymphocyte-dependent and Th2 cytokine-associated colitis in mice deficient in Wiskott-Aldrich Syndrome protein.
- Wiskott Aldrich Syndrome protein (WASP) and N-WASP are critical for T cell development.
- Teaching tolerance with a probiotic antigen delivery System.
- The Wiskott-Aldrich Syndrome protein is required for the function of CD4(+)CD25(+)Foxp3(+) regulatory T cells.
- Targeted deletion of integrin-linked kinase reveals a role in T-cell chemotaxis and survival.
- Role of the WASP family proteins for Mycobacterium marinum actin tail formation.
- WASP deficiency leads to global defects of directed leukocyte migration in vitro and in vivo.
- The NF2 tumor suppressor Merlin and the ERM proteins interact with N-WASP and regulate its actin polymerization function.
- Induced reactivity of Intestinal CD4(+) T cells with an epithelial cell lectin, galectin-4, contributes to exacerbation of Intestinal inflammation.
- A new method for rapidly generating gene-targeting vectors by engineering BACs through homologous recombination in bacteria.
- A defect in hematopoietic stem cell migration explains the nonrandom X-chromosome inactivation in carriers of Wiskott-Aldrich Syndrome.
- Gene therapy for Wiskott-Aldrich Syndrome: rescue of T-cell signaling and amelioration of colitis upon transplantation of retrovirally transduced hematopoietic stem ce...
- A family of WASPs.
- Interleukin 10 receptor signaling: master regulator of Intestinal mucosal homeostasis in mice and humans.
Schools
Albert Einstein College of Medicine of Yeshiva University
Brigham and Women39s Hospital
Harvard Med School
Massachusetts General Hospital
Procedures Preformed
- Colonoscopy, Proctosigmoidoscopy, and Sigmoidoscopy
- Endoscopy (Esophagus, Stomach, Small Intestine)
Conditions Treated
- Abdominal Pain
- Anemia
- Barrett's Esophagus
- Benign Neoplasm of the Digestive System
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Doctors Specialties
- Gastroenterology
Accepted Insurances
Awards
- Elected Member
Education
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Massachusetts General Hospital -
Brigham and Women's Hospital -
Albert Einstein College of Medicine of Yeshiva University
Hospital
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Brigham and Women's Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1184605537 |
| Last Name Of The Provider | SNAPPER |
| First Name Of The Provider | SCOTT |
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