Physician
Anesthesiology
Dr. Shao H. Yang, MD
|
|
Give Review
Dr. Shao H. Yang works in the field of Anesthesiology. He went to medical school at Shanghai Medical University. Shao H. Yang, MD is also published. He has 28 publications published. The lastest publication: "Mice that express farnesylated versions of prelamin A in neurons develop achalasia.'
Publications
- Mice that express farnesylated versions of prelamin A in neurons develop achalasia.
- Chemical compositions and antiproliferation activities of the chloroform fraction from Pyropolyporus fomentarius in K562 cells.
- An absence of nuclear lamins in keratinocytes leads to ichthyosis, defective epidermal barrier function, and intrusion of nuclear membranes and endoplasmic reticulum i...
- Abstracts of Presentations at the International Conference on Basic and Clinical Multimodal Imaging (BaCI), a Joint Conference of the International Society for Neuroim...
- Nuclear lamins in the brain - new insights into function and regulation.
- Targeting protein prenylation in progeria.
- Are B-type lamins essential in all mammalian cells?
- Severe hepatocellular disease in mice lacking one or both CaaX prenyltransferases.
- An absence of both lamin B1 and lamin B2 in keratinocytes has no effect on cell proliferation or the development of skin and hair.
- Investigating the purpose of prelamin A processing.
- Direct synthesis of lamin A, bypassing prelamin a processing, causes misshapen nuclei in fibroblasts but no detectable pathology in mice.
- Targeting the protein prenyltransferases efficiently reduces tumor development in mice with K-RAS-induced lung cancer.
- Assessing the efficacy of protein farnesyltransferase inhibitors in mouse models of progeria.
- Increasing the length of progerin's isoprenyl anchor does not worsen bone disease or survival in mice with Hutchinson-Gilford progeria syndrome.
- The posttranslational processing of prelamin A and disease.
- Progerin elicits disease phenotypes of progeria in mice whether or not it is farnesylated.
- Eliminating the synthesis of mature lamin A reduces disease phenotypes in mice carrying a Hutchinson-Gilford progeria syndrome allele.
- Requirement of N-myristoyltransferase 1 in the development of monocytic lineage.
- Prelamin A farnesylation and progeroid syndromes.
- A farnesyltransferase inhibitor improves disease phenotypes in mice with a Hutchinson-Gilford progeria syndrome mutation.
- A protein farnesyltransferase inhibitor ameliorates disease in a mouse model of progeria.
- Prelamin A and lamin A appear to be dispensable in the nuclear lamina.
- Blocking protein farnesyltransferase improves nuclear shape in fibroblasts from humans with progeroid syndromes.
- Blocking protein farnesyltransferase improves nuclear blebbing in mouse fibroblasts with a targeted Hutchinson-Gilford progeria syndrome mutation.
- N-myristoyltransferase 1 is essential in early mouse development.
- Heterozygosity for Lmna deficiency eliminates the progeria-like phenotypes in Zmpste24-deficient mice.
- Estradiol protects against cerebellar damage and motor deficit in ethanol-withdrawn rats.
Doctors Specialties
- Anesthesiology
Accepted Insurances
Education
-
SUNY Stony Brook University Hospital & Medical Center -
North Shore-Long Island Jewish Health System -
Shanghai Medical University
Hospital
-
Holy Cross Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1932183290 |
| Last Name Of The Provider | YANG |
| First Name Of The Provider | SHAO |
| View All |
Similar Doctors
|
|
Dr. Maneesh Sharma, MD
 This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Ashanpreet S. Grewal, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Yongbo Liao, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Mary B. Gorman, MD |
|
|
Dr. Neeru Sharma, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Julie A. Garringer, MD |
|
|
Dr. Thomas Tung, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Lisa M. Einhorn, MD |