Professor, Medicine, Brown University Program in Medicine
Pulmonology
Dr. Sharon I. Rounds, MD
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Sharon Rounds is an expert in the field of Pulmonology. He graduated from Tufts University School of Medicine. Tufts University School of Medicine is ranked 49 in research and 52 in primary care medicine Sharon Rounds is also published. He has 80 publications published. The lastest publication: "Autobullectomy: spontaneous improvements in pulmonary function and symptoms in a patient with bullous emphysema.'
Publications
- Cigarette smoke-induced lung endothelial apoptosis and emphysema are associated with impairment of FAK and eIF2α
- Study design and rationale for investigating phosphodiesterase type 5 inhibition for the treatment of Pulmonary hypertension due to chronic obstructive lung disease: t...
- Induction of Pulmonary hypertensive changes by extracellular vesicles from monocrotaline-treated mice.
- Sustained adenosine exposure causes lung endothelial apoptosis: a possible contributor to cigarette smoke-induced endothelial apoptosis and lung injury.
- Strategic plan for lung vascular research: An NHLBI-ORDR Workshop Report.
- Studies on the mechanism of decreased neutrophil adherence to postconfluent cultured endothelial cells.
- The binding of cationic probes to apical and basal surfaces of rat lung capillary endothelium and of endothelial cells in tissue culture.
- Thiourea causes endothelial cells in tissue culture to produce neutrophil chemoattractant activity.
- Effect of long-term hypoxia on cultured aortic and Pulmonary arterial endothelial cells.
- Pulmonary hypertensive response to foreign body microemboli.
- Bovine and human endothelial cell production of neutrophil chemoattractant activity in response to components of the angiotensin system.
- Pulmonary response to foreign body microemboli in dogs: release of neutrophil chemoattractant activity by vascular endothelial cells.
- Hemoptysis in Sarcoidosis: a case report.
- Intensive care services in the Veterans Health Administration.
- Platelet-activating factor (PAF)-induced platelet aggregation. Modulation by plasma adenosine and methylxanthines.
- Vascular reactivity is increased in rat lungs injured with alpha-naphthylthiourea.
- Role of protein kinase C isoforms in rat epididymal microvascular endothelial barrier function.
- Apoptosis and lung injury.
- Isoprenylcysteine carboxyl methyltransferase modulates endothelial monolayer permeability: involvement of RhoA carboxyl methylation.
- Release of soluble E-selectin from activated endothelial cells upon apoptosis.
- Adenosine protected against Pulmonary edema through transporter- and receptor A2-mediated endothelial barrier enhancement.
- Pulmonary endothelial cell signaling and function.
- Lamotrigine overdose presenting as anticonvulsant hypersensitivity syndrome.
- Protein tyrosine phosphatase-dependent proteolysis of focal adhesion complexes in endothelial cell apoptosis.
- Pulmonary vascular reactivity is blunted in pregnant rats.
- Bad blood, bad endothelium: ill fate?
- Transforming growth factor-beta1 causes Pulmonary microvascular endothelial cell apoptosis via ALK5.
- Innovative approaches to healthcare delivery at the Providence VA Medical Center.
- Overview of the Providence VA Medical Center.
- Pulmonary hypertension update.
- Compliance with CPAP therapy in older men with obstructive Sleep apnea.
- Cigarette smoke causes lung vascular barrier dysfunction via oxidative stress-mediated inhibition of RhoA and focal adhesion kinase.
- Transforming growth factor-beta1-induced endothelial barrier dysfunction involves Smad2-dependent p38 activation and subsequent RhoA activation.
- Components of the angiotensin system cause release of a neutrophil chemoattractant from cultured bovine and human endothelial cells.
- Effect of ambient oxygen on cultured endothelial cells from different vascular beds.
- Effects of endothelial cell injury on Pulmonary vascular reactivity.
- Effects of hypoxia on heparan sulfate in bovine aortic and Pulmonary artery endothelial cells.
- Barrier dysfunction and RhoA activation are blunted by homocysteine and adenosine in Pulmonary endothelium.
- Effects of hypoxia and hypercarbia on cultured endothelial cells.
- Isoprenylcysteine carboxyl methyltransferase activity modulates endothelial cell apoptosis.
- FAK blunts adenosine-homocysteine-induced endothelial cell apoptosis: requirement for PI 3-kinase.
- Pulmonary hypertensive diseases.
- Group education sessions and compliance with nasal CPAP therapy.
- Unilateral lung hyperinflation and herniation as a manifestation of intrinsic PEEP.
- Effects of endotoxin injury on endothelial cell adenosine metabolism.
- Effect of hypercarbia on surface proteins of cultured bovine endothelial cells.
- Inhibitors of oxidative ATP production cause transient vasoconstriction and block subsequent pressor responses in rat lungs.
- Heterogeneity in apoptotic responses of microvascular endothelial cells to oxidative stress.
- Characteristics of amphotericin B-induced endothelial cell injury.
- Adenosine induces endothelial apoptosis by activating protein tyrosine phosphatase: a possible role of p38alpha.
- Effects of withdrawal of inhaled steroids in men with severe irreversible airflow obstruction.
- Combined Pulmonary fibrosis and emphysema alters physiology but has similar mortality to Pulmonary fibrosis without emphysema.
- Alterations in molecular chaperones and eIF2alpha during lung endothelial cell apoptosis.
- Inhibition of ICMT induces endothelial cell apoptosis through GRP94.
- Circulating endothelial cells and endothelial progenitor cells in obstructive Sleep apnea.
- Mechanism of ATP-induced leukocyte adherence to cultured Pulmonary artery endothelial cells.
- Amrinone dilates Pulmonary vessels and blunts hypoxic vasoconstriction in isolated rat lungs.
- Extracellular nucleotides stimulate leukocyte adherence to cultured Pulmonary artery endothelial cells.
- Extracellular ATP and adenosine cause apoptosis of Pulmonary artery endothelial cells.
- A simple assay for ecto-5''-nucleotidase using intact Pulmonary artery endothelial cells. Effect of endotoxin-induced cell injury.
- Studies of the mechanism of hypoxic Pulmonary vasoconstriction.
- Autobullectomy: spontaneous improvements in Pulmonary function and symptoms in a patient with bullous emphysema.
- C-type natriuretic peptide does not attenuate the development of Pulmonary hypertension caused by hypoxia and VEGF receptor blockade.
- Nucleotide-induced PMN adhesion to cultured epithelial cells: possible role of MUC1 mucin.
- Heterogeneity in combined Pulmonary fibrosis and emphysema.
- Hydrogen peroxide stimulates sodium-potassium pump activity in cultured Pulmonary arterial endothelial cells.
- Studies on the mechanism of short-term regulation of Pulmonary artery endothelial cell Na/K pump activity.
- Oxidant stress stimulates Na/K pump activity in bovine Pulmonary arterial endothelial cells.
- Effect of ATP-induced permeabilization on loading of the Na+ probe SBFI into endothelial cells.
- Mechanism of extracellular ATP- and adenosine-induced apoptosis of cultured Pulmonary artery endothelial cells.
- Effect of hypoxic exposure on Na+/H+ antiport activity, isoform expression, and localization in endothelial cells.
- Differences in nucleotide effects on intracellular pH, Na+/H+ antiport activity, and ATP-binding proteins in endothelial cells.
- Role of the Na/H antiport in pH-dependent cell death in Pulmonary artery endothelial cells.
- Hypoxic Pulmonary vasoconstriction. Physiologic significance, mechanism, and clinical relevance.
- Combined Pulmonary fibrosis and emphysema syndrome: a review.
- Pulmonary arterial hypertension: new insights and new hope.
- Evidence for expanding physician supply.
- Remodeling of the Na/H antiport and the cytoskeleton of human Pulmonary artery endothelial cells: response to an oxygen deficit.
- Cardiac output is decreased and hypoxic vasoconstriction is intact in chronically hypoxic sheep.
Schools
Tufts University
Conditions Treated
- Pulmonary Hypertension
- Sarcoidosis
- Sleep Apnea
Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
- Aetna
- Blue Cross Blue Shield
- Cigna
- Commercial Insurance Company
- Group Health Cooperative
- Medicare Advantage PFFS
- Medicare MCR
- Tricare
- Tufts Health Plans
- United Healthcare
Education
-
University of Colorado -
UCSF -
Tufts University School of Medicine
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