Physician
Neurology
Dr. Stanley H. Appel, MD
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Stanley H. Appel practices Neurology. He went to Columbia University College of Physicians & Surgeons. Columbia University College of Physicians & Surgeons is a top medical university. Columbia University College of Physicians & Surgeons is ranked 8 in research and 52 in primary care medicine He over the years received 5 awards: "Investigator Alumnus", "Elected Member", "Top MD", "Super Doctor" and "Whos Who in Medicine and Healthcare". Stanley H. Appel, MD has written publications. He has 46 publications published with PubMed. The lastest publication is: 'TDP-43 activates microglia through NF-B and NLRP3 inflammasome.' He accepts Medicare.
Publications
- TDP-43 activates microglia through NF-κB and NLRP3 inflammasome.
- Exome sequencing in amyotrophic lateral Sclerosis identifies risk genes and pathways.
- Protective and Toxic Neuroinflammation in Amyotrophic Lateral Sclerosis.
- An open-label multicenter study to assess the safety of dextromethorphan/quinidine in patients with pseudobulbar affect associated with a range of underlying neurologi...
- Seeking a measure of clinically meaningful change in ALS.
- TREM2 Variant p.R47H as a Risk Factor for Sporadic Amyotrophic Lateral Sclerosis.
- Immune-mediated Mechanisms in the Pathoprogression of Amyotrophic Lateral Sclerosis.
- A tribute to jenny s. Henkel, ph.d. (1955-2013).
- Deciphering amyotrophic lateral Sclerosis: What phenotype, neuropathology and genetics are telling us about pathogenesis.
- An antisense oligonucleotide against SOD1 delivered intrathecally for patients with SOD1 familial amyotrophic lateral Sclerosis: a phase 1, randomised, first-in-man st...
- Regulatory T-lymphocytes mediate amyotrophic lateral Sclerosis progression and survival.
- Blood-spinal cord barrier breakdown and pericyte reductions in amyotrophic lateral Sclerosis.
- Regulatory T lymphocytes from ALS mice suppress microglia and effector T lymphocytes through different cytokine-mediated mechanisms.
- Inflammation in Parkinson's Disease: Cause or consequence?
- Transformation from a neuroprotective to a neuroToxic microglial phenotype in a mouse model of ALS.
- Neuroprotective activities of regulatory T cells.
- Excess Circulating Alternatively Activated Myeloid (M2) Cells Accelerate ALS Progression While Inhibiting Experimental Autoimmune Encephalomyelitis.
- Neuroinflammation modulates distinct regional and temporal clinical responses in ALS mice.
- Trauma, TDP-43, and amyotrophic lateral Sclerosis.
- Extracellular mutant SOD1 induces microglial-mediated motoneuron injury.
- Microglia in ALS: the good, the bad, and the resting.
- Abnormal serum concentrations of proteins in Parkinson's Disease.
- CD4+ T cells mediate cytotoxicity in neurodegenerative Diseases.
- CD4+ T cells support glial neuroprotection, slow Disease progression, and modify glial morphology in an animal model of inherited ALS.
- Hematopoietic stem cell transplantation in patients with sporadic amyotrophic lateral Sclerosis.
- Novel therapeutic targets in neurodegenerative Diseases: lessons from amyotrophic lateral sclerosis.
- ALSFRS and appel ALS scores: discordance with Disease progression.
- Whole-genome analysis of sporadic amyotrophic lateral Sclerosis.
- Predictability of Disease progression in amyotrophic lateral sclerosis.
- Wild-type microglia extend survival in PU.1 knockout mice with familial amyotrophic lateral Sclerosis.
- Slower Disease progression and prolonged survival in contemporary patients with amyotrophic lateral sclerosis: is the natural history of amyotrophic lateral sclerosis ...
- Complement C3c and related protein biomarkers in amyotrophic lateral sclerosis and Parkinson's Disease.
- The chemokine MCP-1 and the dendritic and myeloid cells it attracts are increased in the mSOD1 mouse model of ALS.
- Is ALS a systemic disorder? Evidence from muscle mitochondria.
- Incidence and characteristics of spinal decompression surgery after the onset of symptoms of amyotrophic lateral Sclerosis.
- CalPain activation in apoptosis of ventral spinal cord 4.1 (VSC4.1) motoneurons exposed to glutamate: calPain inhibition provides functional neuroprotection.
- Euthanasia and physician-assisted suicide in ALS: a commentary.
- Activated microglia initiate motor neuron injury by a nitric oxide and glutamate-mediated mechanism.
- Presence of dendritic cells, MCP-1, and activated microglia/macrophages in amyotrophic lateral Sclerosis spinal cord tissue.
- Oxidative Stress: a common denominator in the pathogenesis of amyotrophic lateral Sclerosis.
- Mutations in NR4A2 associated with familial Parkinson Disease.
- PARP expression is increased in astrocytes but decreased in motor neurons in the spinal cord of sporadic ALS patients.
- Role of Fcgamma receptors in nigral cell injury induced by Parkinson Disease immunoglobulin injection into mouse substantia nigra.
- Immunoglobulin Fc gamma receptor promotes immunoglobulin uptake, immunoglobulin-mediated calcium increase, and neurotransmitter release in motor neurons.
- Mechanisms of Disease pathogenesis in amyotrophic lateral sclerosis. A central role for calcium.
Schools
Columbia University College Of Physicians and Surgeons
Massachusetts General Hospital
Hospital Of The University Of Pennsylvania
Mt Sinai Hospital
Procedures Preformed
- EEG (Electroencephalogram)
Conditions Treated
- Alzheimer's Disease
- Anterior Horn Disease
- Ataxia
- Autonomic Disorders
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Doctors Specialties
- Neurology
Accepted Insurances
Awards
- Investigator Alumnus
- Elected Member
- Top MD
- Super Doctor
- Whos Who in Medicine and Healthcare
Education
-
University of Pennsylvania Health System -
Mount Sinai School of Medicine -
Massachusetts General Hospital -
Columbia University College of Physicians & Surgeons
Drug Facts
| NPI NUMBER | 1124009113 | |
| NPPES Provider LastName | APPEL | |
| NPPES Provider FirstName | STANLEY | |
| NPPES Provider ZIPCode | 77030 | |
| NPPES Provider State | TX | |
| Specialty Description | Neurology | |
| Total Claim Count | 291.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1124009113 |
| Last Name Of The Provider | APPEL |
| First Name Of The Provider | STANLEY |
| View All |
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