Professor, Medicine, UCLA School of Medicine
Gastroenterology
Dr. Stephen J. Pandol, MD
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Dr. Stephen J. Pandol works in the field of Gastroenterology. He went to medical school at Saint Louis University School of Medicine. Saint Louis University School of Medicine is ranked 67/57 in Research/PrimaryCare. He received 2 awards: "CMS Meaningful Use Stage 1 Certification" and "Elected Member". Stephen Pandol is a published MD as well. He has 98 publications published. The latest was: Early NF-kappaB activation is associated with hormone-induced pancreatitis. Stephen J. Pandol accepts Medicare payments and is registered with Medicare.gov.
Publications
- Exosomal MIF: A Potential New Biomarker and Therapeutic Target for Pancreatic Cancer.
- Pancreatic adaptive responses in alcohol abuse: Role of the unfolded protein response.
- Genetic inhibition of protein kinase Cε attenuates necrosis in experimental Pancreatitis.
- Simvastatin is associated with reduced risk of acute pancreatitis: findings from a regional integrated healthcare System.
- Determination of Rottlerin, a Natural Protein Kinases C Inhibitor, in Pancreatic Cancer Cells and Mouse Xenografts by RP-HPLC Method.
- Akt Kinase Mediates the Prosurvival Effect of Smoking Compounds in Pancreatic Ductal Cells.
- Apigenin inhibits NNK-induced focal adhesion kinase activation in pancreatic cancer cells.
- The burning question: Why is smoking a risk factor for pancreatic cancer?
- Molecular basis for the interplay of apoptosis and proliferation mediated by Bcl-xL:Bim interactions in pancreatic cancer cells.
- Protein kinase d regulates cell death pathways in experimental Pancreatitis.
- Minutes of the business meeting of the american pancreatic association friday, november 5, 2010, chicago, illinois.
- The Emerging Role of Smoking in the Development of Pancreatitis.
- Organellar Dysfunction in the Pathogenesis of Pancreatitis.
- Drinking and driving Pancreatitis: Links between endoplasmic reticulum stress and autophagy.
- NADPH oxidase activation in pancreatic cancer cells is mediated through Akt-dependent up-regulation of p22phox.
- Adaptive unfolded protein response attenuates alcohol-induced pancreatic damage.
- A novel protein kinase D inhibitor attenuates early events of experimental Pancreatitis in isolated rat acini.
- Grp78 heterozygosity regulates chaperone balance in exocrine pancreas with differential response to cerulein-induced acute Pancreatitis.
- Mechanism and regulation of folate uptake by pancreatic acinar cells: effect of chronic alcohol consumption.
- Rottlerin stimulates apoptosis in pancreatic cancer cells through interactions with proteins of the Bcl-2 family.
- Inflammatory cells regulate p53 and caspases in acute Pancreatitis.
- Protein kinase C delta-mediated processes in cholecystokinin-8-stimulated pancreatic acini.
- Impaired autophagic flux mediates acinar cell vacuole formation and trypsinogen activation in rodent models of acute Pancreatitis.
- Prosurvival Bcl-2 proteins stabilize pancreatic mitochondria and protect against necrosis in experimental Pancreatitis.
- Ethanol diversely alters palmitate, stearate, and oleate metabolism in the liver and pancreas of rats using the deuterium oxide single tracer.
- Ethanol exposure depletes hepatic pigment epithelium-derived factor, a novel lipid regulator.
- Protein kinase D1 mediates NF-kappaB activation induced by cholecystokinin and cholinergic signaling in pancreatic acinar cells.
- Integrated response to a meal.
- A new severe acute necrotizing Pancreatitis model induced by L-ornithine in rats.
- The novel protein kinase C isoforms -delta and -epsilon modulate caerulein-induced zymogen activation in pancreatic acinar cells.
- Pancreatitis and calcium signalling: report of an international workshop.
- A rat model reproducing key pathological responses of alcoholic chronic Pancreatitis.
- NADPH oxidase promotes pancreatic cancer cell survival via inhibiting JAK2 dephosphorylation by tyrosine phosphatases.
- Insulin-like growth factor-I receptor mediates the prosurvival effect of fibronectin.
- Acute Pancreatitis: bench to the bedside.
- Substance P mediates cerulein-induced pancreatic microcirculatory dysfunction in mice.
- The pancreatic stellate cell: a star on the rise in pancreatic Diseases.
- Ethanol sensitizes NF-kappaB activation in pancreatic acinar cells through effects on protein kinase C-epsilon.
- Pancreas recovery following cerulein-induced Pancreatitis is impaired in plasminogen-deficient mice.
- Long-term ethanol consumption alters pancreatic gene expression in rats: a possible connection to pancreatic injury.
- Ethanol feeding alters death signaling in the pancreas.
- Cell death in Pancreatitis: caspases protect from necrotizing Pancreatitis.
- Extracellular matrix stimulates reactive oxygen species production and increases pancreatic cancer cell survival through 5-lipoxygenase and NADPH oxidase.
- Tumor necrosis factor-alpha mediates Pancreatitis responses in acinar cells via protein kinase C and proline-rich tyrosine kinase 2.
- Signal transduction in alcohol-related Diseases.
- Alcohol and pancreatic cancer.
- PKC-delta and -epsilon regulate NF-kappaB activation induced by cholecystokinin and TNF-alpha in pancreatic acinar cells.
- Neurohumoral control of exocrine pancreatic secretion.
- Reactive oxygen species produced by NAD(P)H oxidase inhibit apoptosis in pancreatic cancer cells.
- Phosphatidylinositide 3-kinase gamma regulates key pathologic responses to cholecystokinin in pancreatic acinar cells.
- Ethanol differentially regulates NF-kappaB activation in pancreatic acinar cells through calcium and protein kinase C pathways.
- Nonoxidative ethanol metabolites alter extracellular matrix protein content in rat pancreas.
- Animal and in vitro models of alcoholic Pancreatitis: role of cholecystokinin.
- Extracellular matrix proteins protect pancreatic cancer cells from death via mitochondrial and nonmitochondrial pathways.
- Prevention of metastatic pancreatic cancer growth in vivo by induction of apoptosis with genistein, a naturally occurring isoflavonoid.
- Role of S-adenosylmethionine in two experimental models of Pancreatitis.
- Curcumin ameliorates ethanol and nonethanol experimental Pancreatitis.
- Cholecystokinin induces caspase activation and mitochondrial dysfunction in pancreatic acinar cells. Roles in cell injury processes of Pancreatitis.
- Food-derived polyphenols inhibit pancreatic cancer growth through mitochondrial cytochrome C release and apoptosis.
- Neutrophils and NADPH oxidase mediate intrapancreatic trypsin activation in murine experimental acute Pancreatitis.
- Caspase 8-mediated cleavage of plectin precedes F-actin breakdown in acinar cells during Pancreatitis.
- Ethanol metabolism and transcription factor activation in pancreatic acinar cells in rats.
- Role of YAP and TAZ in pancreatic ductal adenocarcinoma and in stellate cells associated with cancer and chronic Pancreatitis.
- Diabetes, pancreatic cancer, and metformin therapy.
- Down-regulation of pancreatic and duodenal homeobox-1 by somatostatin receptor subtype 5: a novel mechanism for inhibition of cellular proliferation and insulin secret...
- Pancreatic cancer cachexia: a review of mechanisms and therapeutics.
- Src as the link between inflammation and cancer.
- Ellagic Acid and embelin affect key cellular components of pancreatic adenocarcinoma, cancer, and stellate cells.
- Phosphatidylinositol 3-kinase regulates Ca2+ signaling in pancreatic acinar cells through inhibition of sarco(endo)plasmic reticulum Ca2+-ATPase.
- Localized pancreatic NF-kappaB activation and inflammatory response in taurocholate-induced Pancreatitis.
- Activation of pancreatic acinar cells on isolation from tissue: cytokine upregulation via p38 MAP kinase.
- Cerulein upregulates ICAM-1 in pancreatic acinar cells, which mediates neutrophil adhesion to these cells.
- Multiple isoforms of the ryanodine receptor are expressed in rat pancreatic acinar cells.
- Endoplasmic reticulum Ca(2+)-ATPase inhibitors stimulate membrane guanylate cyclase in pancreatic acinar cells.
- Ethanol diet increases the sensitivity of rats to Pancreatitis induced by cholecystokinin octapeptide.
- Acyl-coenzyme A causes Ca2+ release in pancreatic acinar cells.
- nNOS and Ca2+ influx in rat pancreatic acinar and submandibular salivary gland cells.
- Pancreatic acinar cells produce, release, and respond to tumor necrosis factor-alpha. Role in regulating cell death and Pancreatitis.
- Myosin I is associated with zymogen granule membranes in the rat pancreatic acinar cell.
- The role of calcium in the regulation of protein synthesis in the exocrine pancreas.
- The role of neutrophils and platelet-activating factor in mediating experimental Pancreatitis.
- Mechanisms of cell death after pancreatic duct obstruction in the opossum and the rat.
- Isolation of subcellular agonist-sensitive calcium stores from the pancreatic acinar cell.
- Cellular mechanisms mediating agonist-stimulated calcium influx in the pancreatic acinar cell.
- Intracellular mechanisms involved in short-term regulation of net protein synthesis in pancreatic acini.
- Dual pathways for agonist-stimulated arachidonic acid release in pancreatic acini: roles in secretion.
- Effects of caerulein on the apical cytoskeleton of the pancreatic acinar cell.
- Differing effects of ethanol on in vitro stimulated pancreatic enzyme secretion in ethanol-fed and control rats.
- Cholecystokinin-induced changes in polysome structure regulate protein synthesis in pancreas.
- Quantal calcium release and calcium entry in the pancreatic acinar cell.
- The agonist-sensitive calcium pool in the pancreatic acinar cell. Activation of plasma membrane Ca2+ influx mechanism.
- Early NF-kappaB activation is associated with hormone-induced Pancreatitis.
- Phorbol myristate acetate induces IL-2 secretion by HUT 78 cells by a mechanism independent of protein kinase C translocation.
- Two components of hormone-evoked calcium release from intracellular stores of pancreatic acinar cells.
- Agonist-sensitive calcium pool in the pancreatic acinar cell. II. Characterization of reloading.
- Agonist-sensitive calcium pool in the pancreatic acinar cell. I. Permeability properties.
- Calcium mobilizing hormones activate the plasma membrane Ca2+ pump of pancreatic acinar cells.
Schools
St Louis Univ Sch Of Med
Vet Affairs Med Center W La
Procedures Preformed
- Colonoscopy, Proctosigmoidoscopy, and Sigmoidoscopy
- Endoscopy (Esophagus, Stomach, Small Intestine)
Conditions Treated
- Abdominal Pain
- Anemia
- Benign Neoplasm of the Digestive System
- Biliary Atresia
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Doctors Specialties
- Gastroenterology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- Elected Member
Education
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VA Greater Los Angeles Healthcare System -
Saint Louis University School of Medicine
Hospital
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Ronald Reagan UCLA Medical Center -
Cedars-Sinai Medical Center
Drug Facts
| NPI NUMBER | 1891892774 | |
| NPPES Provider LastName | PANDOL | |
| NPPES Provider FirstName | STEPHEN | |
| NPPES Provider ZIPCode | 90073 | |
| NPPES Provider State | CA | |
| Specialty Description | Gastroenterology | |
| Total Claim Count | 16.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1891892774 |
| Last Name Of The Provider | PANDOL |
| First Name Of The Provider | STEPHEN |
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